Adaptation of the nematode Caenorhabditis elegans to extreme osmotic stress. - Wikidata - awgldk - TriplyDB

Adaptation of the nematode Caenorhabditis elegans to extreme osmotic stress.

Adaptation of the nematode Caenorhabditis elegans to extreme osmotic stress.

http://www.wikidata.org/entity/Q44673762

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The Caenorhabditis elegans epidermis as a model skin. II: differentiation and physiological roles FLCN and AMPK Confer Resistance to Hyperosmotic Stress via Remodeling of Glycogen Stores Defects in the C. elegans acyl-CoA synthase, acs-3, and nuclear hormone receptor, nhr-25, cause sensitivity to distinct, but overlapping stresses A shift to organismal stress resistance in programmed cell death mutants The Caenorhabditis elegans mucin-like protein OSM-8 negatively regulates osmosensitive physiology via the transmembrane protein PTR-23 Neuronal responses to stress and injury in C. elegans Characterization of the proteostasis roles of glycerol accumulation, protein degradation and protein synthesis during osmotic stress in C. elegans Shifts in the distribution of mass densities is a signature of caloric restriction in Caenorhabditis elegans Analysis of nematode mechanics by piezoresistive displacement clamp.Adaptive sugar provisioning controls survival of C. elegans embryos in adverse environments Hydrogen sulfide increases hypoxia-inducible factor-1 activity independently of von Hippel-Lindau tumor suppressor-1 in C. elegans.Stress and aging induce distinct polyQ protein aggregation states Genetic and physiological activation of osmosensitive gene expression mimics transcriptional signatures of pathogen infection in C. elegans.Neuronal Chemosensation and Osmotic Stress Response Converge in the Regulation of aqp-8 in C. elegans skn-1-Dependent and -independent regulation of aip-1 expression following metabolic stress in Caenorhabditis elegans.Genetic analysis of lysosomal trafficking in Caenorhabditis elegans Experimental Evolution with Caenorhabditis Nematodes.Ku86 preserves chromatin integrity in cells adapted to high NaCl.Glucose shortens the life span of C. elegans by downregulating DAF-16/FOXO activity and aquaporin gene expression.An LEA group 3 family member is involved in survival of C. elegans during exposure to stress.Reproductive assurance drives transitions to self-fertilization in experimental Caenorhabditis elegans.Caenorhabditis elegans OSR-1 regulates behavioral and physiological responses to hyperosmotic environments.Genome-wide RNAi screening identifies protein damage as a regulator of osmoprotective gene expression.O-linked-N-acetylglucosamine cycling and insulin signaling are required for the glucose stress response in Caenorhabditis elegans Pollution breaks down the genetic architecture of life history traits in Caenorhabditis elegans.Identification of a novel gene family involved in osmotic stress response in Caenorhabditis elegans.Hypertonic stress induces rapid and widespread protein damage in C. elegans.DNA double-strand breaks induced by high NaCl occur predominantly in gene deserts Expression of aquaporin 3 in gills of the Atlantic killifish (Fundulus heteroclitus): Effects of seawater acclimation.Biochemistry of Anhydrobiosis in Beddingia siricidicola, a Biological Control Agent of Sirex noctilio.In the beginning, there was the cell: cellular homeostasis.Abnormal Osmotic Avoidance Behavior in C. elegans Is Associated with Increased Hypertonic Stress Resistance and Improved Proteostasis A quantitative genome-wide RNAi screen in C. elegans for antifungal innate immunity genes.Sorbitol treatment extends lifespan and induces the osmotic stress response in Caenorhabditis elegans.Formation and regulation of adaptive response in nematode Caenorhabditis elegans.Role of the Caenorhabditis elegans multidrug resistance gene, mrp-4, in gut granule differentiation The cystic-fibrosis-associated ΔF508 mutation confers post-transcriptional destabilization on the C. elegans ABC transporter PGP-3.GCN-2 dependent inhibition of protein synthesis activates osmosensitive gene transcription via WNK and Ste20 kinase signaling.C. elegans lifespan extension by osmotic stress requires FUdR, base excision repair, FOXO, and sirtuins.C. elegans flavin-containing monooxygenase-4 is essential for osmoregulation in hypotonic stress

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Adaptation of the nematode Caenorhabditis elegans to extreme osmotic stress.

http://www.wikidata.org/entity/Q44673762

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

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2003年學術文章

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2003年學術文章

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name

Adaptation of the nematode Caenorhabditis elegans to extreme osmotic stress.

@en

Adaptation of the nematode Caenorhabditis elegans to extreme osmotic stress.

@nl

type

label

Adaptation of the nematode Caenorhabditis elegans to extreme osmotic stress.

@en

Adaptation of the nematode Caenorhabditis elegans to extreme osmotic stress.

@nl

prefLabel

Adaptation of the nematode Caenorhabditis elegans to extreme osmotic stress.

@en

Adaptation of the nematode Caenorhabditis elegans to extreme osmotic stress.

@nl

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AJPCELL.00381.2003

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American Journal of Physiology - Cell Physiology

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Adaptation of the nematode Caenorhabditis elegans to extreme osmotic stress.

@en

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Gilbert W Moeckel

http://www.w3.org/2001/XMLSchema#string

Kevin Strange

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Rebecca Morrison

http://www.w3.org/2001/XMLSchema#string

S Todd Lamitina

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P2860

Yeast HOG1 MAP kinase cascade is regulated by a multistep phosphorelay mechanism in the SLN1-YPD1-SSK1 "two-component" osmosensor.

Yeast osmosensor Sln1 and plant cytokinin receptor Cre1 respond to changes in turgor pressure.

The FPS1 gene product functions as a glycerol facilitator in the yeast Saccharomyces cerevisiae.

A third osmosensing branch in Saccharomyces cerevisiae requires the Msb2 protein and functions in parallel with the Sho1 branch.

Glycerol export and glycerol-3-phosphate dehydrogenase, but not glycerol phosphatase, are rate limiting for glycerol production in Saccharomyces cerevisiae.

GPD1, which encodes glycerol-3-phosphate dehydrogenase, is essential for growth under osmotic stress in Saccharomyces cerevisiae, and its expression is regulated by the high-osmolarity glycerol response pathway.

A Caenorhabditis elegans MAP kinase kinase, MEK-1, is involved in stress responses

A transmembrane hybrid-type histidine kinase in Arabidopsis functions as an osmosensor

Living with water stress: evolution of osmolyte systems

Osmotic stress signaling and osmoadaptation in yeasts.

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C785-91

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scholarly article

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10.1152/AJPCELL.00381.2003

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4

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286

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14644776

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Caenorhabditis elegans