Regulation of oxidative phosphorylation during work transitions results from its kinetic properties.
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'Idealized' state 4 and state 3 in mitochondria vs. rest and work in skeletal muscleMechanisms of Attenuation of Pulmonary V'O2 Slow Component in Humans after Prolonged Endurance Training.Each-step activation of oxidative phosphorylation is necessary to explain muscle metabolic kinetic responses to exercise and recovery in humans.Faster and stronger manifestation of mitochondrial diseases in skeletal muscle than in heart related to cytosolic inorganic phosphate (Pi) accumulation.Contribution of proton leak to oxygen consumption in skeletal muscle during intense exercise is very low despite large contribution at rest.Possible mechanisms underlying slow component of V̇O2 on-kinetics in skeletal muscle.Mechanisms responsible for the acceleration of pulmonary V̇O2 on-kinetics in humans after prolonged endurance training.
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P2860
Regulation of oxidative phosphorylation during work transitions results from its kinetic properties.
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2013 nî lūn-bûn
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2013年の論文
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Regulation of oxidative phosph ...... s from its kinetic properties.
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Regulation of oxidative phosph ...... s from its kinetic properties.
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Regulation of oxidative phosph ...... s from its kinetic properties.
@en
Regulation of oxidative phosph ...... s from its kinetic properties.
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Regulation of oxidative phosph ...... s from its kinetic properties.
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Regulation of oxidative phosph ...... s from its kinetic properties.
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P2860
P1476
Regulation of oxidative phosph ...... s from its kinetic properties.
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P2860
P356
10.1152/JAPPLPHYSIOL.00759.2013
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P577
2013-10-24T00:00:00Z