Human lymphocytes exposed to low doses of ionizing radiations become refractory to high doses of radiation as well as to chemical mutagens that induce double-strand breaks in DNA.
about
Radioprotection by short-term oxidative preconditioning: role of manganese superoxide dismutaseOxidants, antioxidants, and the degenerative diseases of agingA perspective on the scientific, philosophical, and policy dimensions of hormesisThe adaptive response in radiobiology: evolving insights and implicationsThe risk linked to ionizing radiation: an alternative epidemiologic approach.Transcriptional regulation of human DNA repair genes following genotoxic stress: trigger mechanisms, inducible responses and genotoxic adaptationThe dose window for radiation-induced protective adaptive responsesCancer risk at low doses of ionizing radiation: artificial neural networks inference from atomic bomb survivors.Effects of acute low doses of gamma-radiation on erythrocytes membrane.Low-dose or low-dose-rate ionizing radiation-induced bioeffects in animal models.Perspectives on the adaptive response from studies on the response to low radiation doses (or to cisplatin) in mammalian cells.Chemical carcinogenesis: too many rodent carcinogensNature's chemicals and synthetic chemicals: comparative toxicologyLow-dose cancer risk modeling must recognize up-regulation of protection.Induced radioresistance: an overview and historical perspective.Evidence for beneficial low level radiation effects and radiation hormesis.Toxicological interactions between nickel and radiation on chromosome damage and repair.Granulocytes affect double-strand break repair assays in primary human lymphocytes.A single whole-body low dose X-irradiation does not affect L1, B1 and IAP repeat element DNA methylation longitudinallyRadiation-induced versus endogenous DNA damage: possible effect of inducible protective responses in mitigating endogenous damage.Pre-exposure to ionizing radiation stimulates DNA double strand break end resection, promoting the use of homologous recombination repairDifferential expression of protein kinase C epsilon protein in lung cancer cell lines by ionising radiation.Potassium-channel activation in response to low doses of gamma-irradiation involves reactive oxygen intermediates in nonexcitatory cells.What are the risks from medical X-rays and other low dose radiation?A manganese superoxide dismutase (SOD2)-mediated adaptive response.Irradiation with He--Ne laser can influence the cytotoxic response of HeLa cells to ionizing radiation.DNA-binding protein activated by gamma radiation in human cells.Sparsely ionizing diagnostic and natural background radiations are likely preventing cancer and other genomic-instability-associated diseases.Nucleotide sequence and transcriptional regulation of the yeast recombinational repair gene RAD51.Health risk assessment of radiation.It's time for a new low-dose-radiation risk assessment paradigm--one that acknowledges hormesis.A survivin-associated adaptive response in radiation therapy.Responses to low doses of ionizing radiation in biological systemsLow-dose radiation and genotoxic chemicals can protect against stochastic biological effects.Radiation-stimulated epigenetic reprogramming of adaptive-response genes in the lung: an evolutionary gift for mounting adaptive protection against lung cancerDose and Radioadaptive Response Analysis of Micronucleus Induction in Mouse Bone Marrow.Molecular mechanisms of low dose ionizing radiation-induced hormesis, adaptive responses, radioresistance, bystander effects, and genomic instability.Integrative genomic and proteomic analyses identifies glycerol-3-phosphate acyltransferase as a target of low-dose ionizing radiation in EBV infected-B cells.Low-dose radiation may be a novel approach to enhance the effectiveness of cancer therapeutics.Radiation-induced adaptive response is not seen in cell lines showing a bystander effect but is seen in lines showing HRS/IRR response.
P2860
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P2860
Human lymphocytes exposed to low doses of ionizing radiations become refractory to high doses of radiation as well as to chemical mutagens that induce double-strand breaks in DNA.
description
1988 nî lūn-bûn
@nan
1988年の論文
@ja
1988年学术文章
@wuu
1988年学术文章
@zh
1988年学术文章
@zh-cn
1988年学术文章
@zh-hans
1988年学术文章
@zh-my
1988年学术文章
@zh-sg
1988年學術文章
@yue
1988年學術文章
@zh-hant
name
Human lymphocytes exposed to l ...... e double-strand breaks in DNA.
@en
Human lymphocytes exposed to l ...... e double-strand breaks in DNA.
@nl
type
label
Human lymphocytes exposed to l ...... e double-strand breaks in DNA.
@en
Human lymphocytes exposed to l ...... e double-strand breaks in DNA.
@nl
prefLabel
Human lymphocytes exposed to l ...... e double-strand breaks in DNA.
@en
Human lymphocytes exposed to l ...... e double-strand breaks in DNA.
@nl
P2093
P2860
P1433
P1476
Human lymphocytes exposed to l ...... e double-strand breaks in DNA.
@en
P2093
P2860
P356
10.1080/09553008814550401
P577
1988-01-01T00:00:00Z