Hypercholesterolemia inhibits L-type calcium current in coronary macro-, not microcirculation.
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Poly(ethylene glycol)-cholesterol inhibits L-type Ca2+ channel currents and augments voltage-dependent inactivation in A7r5 cellsOligonucleotide microarray analysis of dietary-induced hyperlipidemia gene expression profiles in miniature pigsHeart of the matter: coronary dysfunction in metabolic syndrome.Contribution of electromechanical coupling between Kv and Ca v1.2 channels to coronary dysfunction in obesity.Rimonabant improves metabolic parameters partially attributed to restoration of high voltage-activated Ca2+ channels in skeletal muscle in HFD-fed miceEnantioselective protein-sterol interactions mediate regulation of both prokaryotic and eukaryotic inward rectifier K+ channels by cholesterol.Cholesterol influences voltage-gated calcium channels and BK-type potassium channels in auditory hair cells.Upregulation of intermediate-conductance Ca2+-activated K+ channels (KCNN4) in porcine coronary smooth muscle requires NADPH oxidase 5 (NOX5).Testosterone and cholesterol vasodilation of rat aorta involves L-type calcium channel inhibition.Caveolin-1 expression and membrane cholesterol content modulate N-type calcium channel activity in NG108-15 cellsCholesterol sensitivity of KIR2.1 is controlled by a belt of residues around the cytosolic pore.Identification of a cholesterol-binding pocket in inward rectifier K(+) (Kir) channels.Cholesterol down-regulates BK channels stably expressed in HEK 293 cells.Ca2+ regulatory mechanisms of exercise protection against coronary artery disease in metabolic syndrome and diabetes.Differential effects of androgens on coronary blood flow regulation and arteriolar diameter in intact and castrated swine.Distant cytosolic residues mediate a two-way molecular switch that controls the modulation of inwardly rectifying potassium (Kir) channels by cholesterol and phosphatidylinositol 4,5-bisphosphate (PI(4,5)P(2)).Physical activity-induced remodeling of vasculature in skeletal muscle: role in treatment of type 2 diabetesBiphasic alterations in coronary smooth muscle Ca(2+) regulation in a repeat cross-sectional study of coronary artery disease severity in metabolic syndrome.Effects of exercise training and hypercholesterolemia on adenosine activation of voltage-dependent K+ channels in coronary arterioles.The non-excitable smooth muscle: calcium signaling and phenotypic switching during vascular disease.Endogenous testosterone attenuates neointima formation after moderate coronary balloon injury in male swine.Impaired function of coronary BK(Ca) channels in metabolic syndromeCholesterol and Kir channels.Cholesterol and ion channels.Influence of cholesterol on cellular signaling and fusion pore kinetics.Regulation of ion channels by membrane lipids.Smooth Muscle Ion Channels and Regulation of Vascular Tone in Resistance Arteries and Arterioles.Comparative analysis of cholesterol sensitivity of Kir channels: role of the CD loop.Exercise training prevents Ca2+ dysregulation in coronary smooth muscle from diabetic dyslipidemic yucatan swine.Influence of sex, high-fat diet, and exercise training on potassium currents of swine coronary smooth muscle.Cholesterol depletion modulates basal L-type Ca2+ current and abolishes its -adrenergic enhancement in ventricular myocytes.Cholesterol depletion alters coronary artery myocyte Ca(2+) signalling in a stimulus-specific manner.Isoform dependent regulation of human HCN channels by cholesterol.Inhibition of CaV2.3 channels by NK1 receptors is sensitive to membrane cholesterol but insensitive to caveolin-1.Endogenous testosterone increases L-type Ca2+ channel expression in porcine coronary smooth muscle.Hypercholesterolemia abolishes voltage-dependent K+ channel contribution to adenosine-mediated relaxation in porcine coronary arterioles.Molecular dynamics simulations on the interaction of the transmembrane NavAb channel with cholesterol and lipids in the membrane.Arterial endothelial function in a porcine model of early stage atherosclerotic vascular disease.Cholesterol Enrichment Impairs Capacitative Calcium Entry, eNOS Phosphorylation & Shear Stress-Induced NO Production.Liver and intestinal protective effects of Castanea sativa Mill. bark extract in high-fat diet rats
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P2860
Hypercholesterolemia inhibits L-type calcium current in coronary macro-, not microcirculation.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh-hant
name
Hypercholesterolemia inhibits ...... macro-, not microcirculation.
@en
Hypercholesterolemia inhibits ...... macro-, not microcirculation.
@nl
type
label
Hypercholesterolemia inhibits ...... macro-, not microcirculation.
@en
Hypercholesterolemia inhibits ...... macro-, not microcirculation.
@nl
prefLabel
Hypercholesterolemia inhibits ...... macro-, not microcirculation.
@en
Hypercholesterolemia inhibits ...... macro-, not microcirculation.
@nl
P2093
P2860
P1476
Hypercholesterolemia inhibits ...... y macro-, not microcirculation
@en
P2093
P2860
P304
P356
10.1152/JAPPLPHYSIOL.01229.2003
P407
P50
P577
2004-01-29T00:00:00Z