The role of oxidant injury in the pathophysiology of human thalassemias.
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Suppression of Fas-FasL coexpression by erythropoietin mediates erythroblast expansion during the erythropoietic stress response in vivoIneffective erythropoiesis in β -thalassemiaRed Blood Cell Function and Dysfunction: Redox Regulation, Nitric Oxide Metabolism, Anemia.Phosphoproteomic analysis of apoptotic hematopoietic stem cells from hemoglobin E/β-thalassemia.α-Globin as a molecular target in the treatment of β-thalassemiaIron chelation in the treatment of cancer: a new role for deferasirox?Selenoproteins are involved in antioxidant defense systems in thalassemia.Oxidation status of β-thalassemia minor and Hb H disease, and its association with glycerol lysis time (GLT50).Low IL-2 Expressing T Cells in Thalassemia Major Patients: Is It Immune Aging
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P2860
The role of oxidant injury in the pathophysiology of human thalassemias.
description
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name
The role of oxidant injury in the pathophysiology of human thalassemias.
@en
The role of oxidant injury in the pathophysiology of human thalassemias.
@nl
type
label
The role of oxidant injury in the pathophysiology of human thalassemias.
@en
The role of oxidant injury in the pathophysiology of human thalassemias.
@nl
prefLabel
The role of oxidant injury in the pathophysiology of human thalassemias.
@en
The role of oxidant injury in the pathophysiology of human thalassemias.
@nl
P2093
P2860
P1433
P1476
The role of oxidant injury in the pathophysiology of human thalassemias.
@en
P2093
Filippo Centis
Michael Verneris
Stanley L Schrier
P2860
P304
P356
10.1179/135100003225002835
P577
2003-01-01T00:00:00Z