How does Parkinson's disease begin? The role of compensatory mechanisms.
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Are cyclooxygenase-2 and nitric oxide involved in the dyskinesia of Parkinson's disease induced by L-DOPA?How compensation breaks down in Parkinson's disease: Insights from modeling of denervated striatumPharmacogenetics of antipsychotic-induced movement disorders as a resource for better understanding Parkinson's disease modifier genesAbnormal Temporal Coupling of Tactile Perception and Motor Action in Parkinson's Disease.Pre-synaptic dopaminergic compensation after moderate nigrostriatal damage in non-human primates.Dopamine replacement therapy does not restore the ability of Parkinsonian patients to make rapid adjustments in motor strategies according to changing sensorimotor contextsTargeting the progression of Parkinson's disease.Operationalizing compensation over time in neurodegenerative diseasePerspective: Identification of genetic variants associated with dopaminergic compensatory mechanisms in early Parkinson's disease.Monoamine oxidase-B (MAO-B) inhibitors: implications for disease-modification in Parkinson's disease.Motor learning in animal models of Parkinson's disease: Aberrant synaptic plasticity in the motor cortex.Health benefits of methylxanthines in neurodegenerative diseases.The functional connectivity in the motor loop of human basal ganglia.Changes in brain functional activation during resting and locomotor states after unilateral nigrostriatal damage in rats.Inter-hemispheric asymmetry of nigrostriatal dopaminergic lesion: a possible compensatory mechanism in Parkinson's disease.Compensatory Vasodilator Mechanisms in the Ophthalmic Artery of Endothelial Nitric Oxide Synthase Gene Knockout Mice.Transcranial magnetic stimulation follow-up study in early Parkinson's disease: A decline in compensation with disease progression?Localized vs. Systematic Neurodegeneration: A Paradigm Shift in Understanding Neurodegenerative Diseases.Calbindin content and differential vulnerability of midbrain efferent dopaminergic neurons in macaques.Gray matter correlates of dopaminergic degeneration in Parkinson's disease: A hybrid PET/MR study using (18) F-FP-CIT.Counteraction by nitric oxide synthase inhibitor of neurochemical alterations of dopaminergic system in 6-OHDA-lesioned rats under L-DOPA treatment.Enhanced expression of glutamate decarboxylase 65 improves symptoms of rat parkinsonian models.Evidence for cross-hemispheric preconditioning in experimental Parkinson's disease.Consequences of manganese administration for striatal dopamine and motor behavior in 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-exposed C57BL/6 mice.A new hypothesis for Parkinson's disease pathogenesis: GTPase-p38 MAPK signaling and autophagy as convergence points of etiology and genomics
P2860
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P2860
How does Parkinson's disease begin? The role of compensatory mechanisms.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年學術文章
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2004年學術文章
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name
How does Parkinson's disease begin? The role of compensatory mechanisms.
@en
How does Parkinson's disease begin? The role of compensatory mechanisms.
@nl
type
label
How does Parkinson's disease begin? The role of compensatory mechanisms.
@en
How does Parkinson's disease begin? The role of compensatory mechanisms.
@nl
prefLabel
How does Parkinson's disease begin? The role of compensatory mechanisms.
@en
How does Parkinson's disease begin? The role of compensatory mechanisms.
@nl
P50
P1476
How does Parkinson's disease begin? The role of compensatory mechanisms
@en
P2093
Jose A Obeso
P304
125-7; author reply 127-8
P356
10.1016/J.TINS.2003.12.006
P577
2004-03-01T00:00:00Z