Nanog regulates self-renewal of cancer stem cells through the insulin-like growth factor pathway in human hepatocellular carcinoma. - Wikidata - awgldk - TriplyDB

Nanog regulates self-renewal of cancer stem cells through the insulin-like growth factor pathway in human hepatocellular carcinoma.

Nanog regulates self-renewal of cancer stem cells through the insulin-like growth factor pathway in human hepatocellular carcinoma.

http://www.wikidata.org/entity/Q44811513

about

Cancer stem cells (CSCs), cervical CSCs and targeted therapies Epigenetic modulators, modifiers and mediators in cancer aetiology and progression Understanding the Key to Targeting the IGF Axis in Cancer: A Biomarker Assessment The links between insulin resistance, diabetes, and cancer Recognition and killing of cancer stem-like cell population in hepatocellular carcinoma cells by cytokine-induced killer cells via NKG2d-ligands recognition Turning hepatic cancer stem cells inside out--a deeper understanding through multiple perspectives.The Progress and Prospects of Putative Biomarkers for Liver Cancer Stem Cells in Hepatocellular Carcinoma Insights into the Nanog gene: A propeller for stemness in primitive stem cells Cancer stem cells in basic science and in translational oncology: can we translate into clinical application?The role of NANOG transcriptional factor in the development of malignant phenotype of cancer cells Concise Review: NANOG in Cancer Stem Cells and Tumor Development: An Update and Outstanding Questions Sustained proliferation in cancer: Mechanisms and novel therapeutic targets Nanog1 in NTERA-2 and recombinant NanogP8 from somatic cancer cells adopt multiple protein conformations and migrate at multiple M.W species Inflammation Promotes Expression of Stemness-Related Properties in HBV-Related Hepatocellular Carcinoma Emerging role of nanog in tumorigenesis and cancer stem cells.HOXB7 accelerates the malignant progression of hepatocellular carcinoma by promoting stemness and epithelial-mesenchymal transition Decrease of 5-hydroxymethylcytosine is associated with progression of hepatocellular carcinoma through downregulation of TET1.Regulation of NANOG in cancer cells.Epigenetic regulation of pluripotent genes mediates stem cell features in human hepatocellular carcinoma and cancer cell lines.Identification of a unique hepatocellular carcinoma line, Li-7, with CD13(+) cancer stem cells hierarchy and population change upon its differentiation during culture and effects of sorafenib.Role of IGF1R in Breast Cancer Subtypes, Stemness, and Lineage Differentiation Insulin-like growth factor (IGF) signaling in tumorigenesis and the development of cancer drug resistance.The role of Nanog expression in tamoxifen-resistant breast cancer cells Lgr5 Methylation in Cancer Stem Cell Differentiation and Prognosis-Prediction in Colorectal Cancer Cytokine-induced killer cells efficiently kill stem-like cancer cells of nasopharyngeal carcinoma via the NKG2D-ligands recognition Nanog induces suppression of senescence through downregulation of p27KIP1 expression.CD24 cell surface expression in Mvt1 mammary cancer cells serves as a biomarker for sensitivity to anti-IGF1R therapy.Notch and Wnt/β-catenin signaling pathway play important roles in activating liver cancer stem cells.The Notch pathway promotes the cancer stem cell characteristics of CD90+ cells in hepatocellular carcinoma The expression and significance of insulin-like growth factor-1 receptor and its pathway on breast cancer stem/progenitors.Strategies for isolating and enriching cancer stem cells: well begun is half done Bile acids promote diethylnitrosamine-induced hepatocellular carcinoma via increased inflammatory signaling.Targeting cancer stem cells: emerging role of Nanog transcription factor Oncogenic NanogP8 expression regulates cell proliferation and migration through the Akt/mTOR signaling pathway in human gastric cancer - SGC-7901cell line CWP232228 targets liver cancer stem cells through Wnt/β-catenin signaling: a novel therapeutic approach for liver cancer treatment MEK1 signaling promotes self-renewal and tumorigenicity of liver cancer stem cells via maintaining SIRT1 protein stabilization GP73-regulated oncolytic adenoviruses possess potent killing effect on human liver cancer stem-like cells.miR-612 suppresses stem cell-like property of hepatocellular carcinoma cells by modulating Sp1/Nanog signaling.In vivo functional studies of tumor-specific retrogene NanogP8 in transgenic animals.Essential role of insulin-like growth factor 2 in resistance to histone deacetylase inhibitors.

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Nanog regulates self-renewal of cancer stem cells through the insulin-like growth factor pathway in human hepatocellular carcinoma.

http://www.wikidata.org/entity/Q44811513

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2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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name

Nanog regulates self-renewal o ...... uman hepatocellular carcinoma.

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Nanog regulates self-renewal o ...... uman hepatocellular carcinoma.

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Nanog regulates self-renewal o ...... uman hepatocellular carcinoma.

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Nanog regulates self-renewal o ...... uman hepatocellular carcinoma.

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Nanog regulates self-renewal o ...... uman hepatocellular carcinoma.

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Cheng Qian

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Chuan Xu

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Feng Xia

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Guangjie Duan

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Jia Liu

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Juanjuan Shan

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Junjie Shen

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Limei Liu

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Ping Bie

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Qianzhen Zhang

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P2860

The epithelial-mesenchymal transition generates cells with properties of stem cells

Transitions between epithelial and mesenchymal states: acquisition of malignant and stem cell traits

Significance of CD90+ cancer stem cells in human liver cancer

Cancer stem cells in solid tumours: accumulating evidence and unresolved questions

Nanog transforms NIH3T3 cells and targets cell-type restricted genes

A Myc network accounts for similarities between embryonic stem and cancer cell transcription programs

Hepatocellular carcinoma pathogenesis: from genes to environment

EMT, cancer stem cells and drug resistance: an emerging axis of evil in the war on cancer

Wnt activity defines colon cancer stem cells and is regulated by the microenvironment

Heterogeneity in cancer: cancer stem cells versus clonal evolution

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