Microvascular dysfunction after transient high glucose is caused by superoxide-dependent reduction in the bioavailability of NO and BH(4).
about
Twenty-five years since the discovery of endothelium-derived relaxing factor (EDRF): does a dysfunctional endothelium contribute to the development of type 2 diabetes?Paradoxical role of angiotensin II type 2 receptors in resistance arteries of old rats.Coronary microvascular dysfunction in diabetes mellitus: A reviewVascular TRP channels: performing under pressure and going with the flowIncreased availability of angiotensin AT 1 receptors leads to sustained arterial constriction to angiotensin II in diabetes - role for Rho-kinase activationMicrovascular responsiveness in obesity: implications for therapeutic intervention.Cerebral myogenic reactivity and blood flow in type 2 diabetic rats: role of peroxynitrite in hypoxia-mediated loss of myogenic tone.Hyperglycemia in acute ischemic stroke: a vascular perspective.Selective up-regulation of arginase-1 in coronary arteries of diabetic patients.Role of glycocalyx in flow-induced production of nitric oxide and reactive oxygen species.Mitochondrial-derived hydrogen peroxide inhibits relaxation of bovine coronary arterial smooth muscle to hypoxia through stimulation of ERK MAP kinase.Mechanisms of coronary microvascular adaptation to obesity.Effects of diabetes on myocardial capillary density and serum angiogenesis biomarkers in male ratsDevelopmental conditioning of the vasculature.Hyperglycemia adversely modulates endothelial nitric oxide synthase during anesthetic preconditioning through tetrahydrobiopterin- and heat shock protein 90-mediated mechanisms.Activation of hexosamine pathway impairs nitric oxide (NO)-dependent arteriolar dilations by increased protein O-GlcNAcylation.H2O2 increases production of constrictor prostaglandins in smooth muscle leading to enhanced arteriolar tone in Type 2 diabetic mice.Cyclooxygenase-2 preserves flow-mediated remodelling in old obese Zucker rat mesenteric arteries.Effects of ageing and exercise training on eNOS uncoupling in skeletal muscle resistance arterioles.Estradiol attenuates high glucose-induced endothelial nitrotyrosine: role for neuronal nitric oxide synthase.Ageing diminishes endothelium-dependent vasodilatation and tetrahydrobiopterin content in rat skeletal muscle arterioles.Effect of apocynin treatment on renal expression of COX-2, NOS1, and renin in Wistar-Kyoto and spontaneously hypertensive rats.Delineation of epicardial stenosis in patients with microvascular disease using pressure drop coefficient: A pilot outcome study.Glycemic effect of administration of epinephrine-containing local anaesthesia in patients undergoing dental extraction, a comparison between healthy and diabetic patients.Too much TRAFfic at the crossroads of diabetes and endothelial dysfunction.
P2860
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P2860
Microvascular dysfunction after transient high glucose is caused by superoxide-dependent reduction in the bioavailability of NO and BH(4).
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
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2004年學術文章
@zh-hant
name
Microvascular dysfunction afte ...... e bioavailability of NO and BH
@nl
Microvascular dysfunction afte ...... oavailability of NO and BH(4).
@en
type
label
Microvascular dysfunction afte ...... e bioavailability of NO and BH
@nl
Microvascular dysfunction afte ...... oavailability of NO and BH(4).
@en
prefLabel
Microvascular dysfunction afte ...... e bioavailability of NO and BH
@nl
Microvascular dysfunction afte ...... oavailability of NO and BH(4).
@en
P2093
P2860
P1476
Microvascular dysfunction afte ...... oavailability of NO and BH(4).
@en
P2093
Akos Koller
Erika Toth
Gabor Kaley
Zsolt Bagi
P2860
P304
P356
10.1152/AJPHEART.00074.2004
P577
2004-03-25T00:00:00Z