Long-term hypoxia alters endocrine and physiologic responses to umbilical cord occlusion in the ovine fetus.
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Fetal endocrine and metabolic adaptations to hypoxia: the role of the hypothalamic-pituitary-adrenal axisNitric oxide inhibits ACTH-induced cortisol production in near-term, long-term hypoxic ovine fetal adrenocortical cellsLong-term hypoxia enhances cortisol biosynthesis in near-term ovine fetal adrenal cortical cellsAdrenocortical and adipose responses to high-altitude-induced, long-term hypoxia in the ovine fetusMild chronic hypoxia modifies the fetal sheep neural and cardiovascular responses to repeated umbilical cord occlusion.Gestational hypoxia modulates expression of corticotropin-releasing hormone and arginine vasopressin in the paraventricular nucleus in the ovine fetus.Leptin receptor antagonist treatment ameliorates the effects of long-term maternal hypoxia on adrenal expression of key steroidogenic genes in the ovine fetus.Extracellular signal-regulated kinases (ERK1/2) signaling pathway plays a role in cortisol secretion in the long-term hypoxic ovine fetal adrenal near term.Long-term hypoxia increases endothelial nitric oxide synthase expression in the ovine fetal adrenal.Long-term hypoxia enhances ACTH response to arginine vasopressin but not corticotropin-releasing hormone in the near-term ovine fetus.Long-term hypoxia increases leptin receptors and plasma leptin concentrations in the late-gestation ovine fetus.Role of placental insufficiency and intrauterine growth restriction on the activation of fetal hepatic glucose production.Adrenocorticotropic Hormone and PI3K/Akt Inhibition Reduce eNOS Phosphorylation and Increase Cortisol Biosynthesis in Long-Term Hypoxic Ovine Fetal Adrenal Cortical Cells.Expression of StAR and Key Genes Regulating Cortisol Biosynthesis in Near Term Ovine Fetal Adrenocortical Cells: Effects of Long-Term Hypoxia.Long-term hypoxia alters ovine fetal endocrine and physiological responses to hypotension.Long-term hypoxia enhances proopiomelanocortin processing in the near-term ovine fetus.Long-term hypoxia modulates expression of key genes regulating adrenomedullary function in the late gestation ovine fetus.Expression and distribution of glucocorticoid receptors in the ovine fetal adrenal cortex: effect of long-term hypoxia.Long-term hypoxia represses the expression of key genes regulating cortisol biosynthesis in the near-term ovine fetus.Long-term hypoxia modulates expression of key genes regulating adipose function in the late-gestation ovine fetus.Prolonged mild hypoxia alters fetal sheep electrocorticogram activity.Hypoxaemia-induced catecholamine secretion from adrenal chromaffin cells inhibits glucose-stimulated hyperinsulinaemia in fetal sheep.Muscarinic-mediated vasoconstriction in human, rat and sheep umbilical cords and related vasoconstriction mechanisms.
P2860
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P2860
Long-term hypoxia alters endocrine and physiologic responses to umbilical cord occlusion in the ovine fetus.
description
2004 nî lūn-bûn
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name
Long-term hypoxia alters endoc ...... occlusion in the ovine fetus.
@en
Long-term hypoxia alters endoc ...... occlusion in the ovine fetus.
@nl
type
label
Long-term hypoxia alters endoc ...... occlusion in the ovine fetus.
@en
Long-term hypoxia alters endoc ...... occlusion in the ovine fetus.
@nl
prefLabel
Long-term hypoxia alters endoc ...... occlusion in the ovine fetus.
@en
Long-term hypoxia alters endoc ...... occlusion in the ovine fetus.
@nl
P2093
P2860
P1476
Long-term hypoxia alters endoc ...... occlusion in the ovine fetus.
@en
P2093
Charles A Ducsay
Hikaru Umezaki
Kanchan M Kaushal
Toshiko Imamura
P2860
P304
P356
10.1016/J.JSGI.2003.09.006
P577
2004-04-01T00:00:00Z