Opening of Ca2+-activated K+ channels triggers early and delayed preconditioning against I/R injury independent of NOS in mice.
about
Mitochondrial reactive oxygen species: a double edged sword in ischemia/reperfusion vs preconditioningRole of epoxyeicosatrienoic acids in protecting the myocardium following ischemia/reperfusion injuryMitoBK(Ca) is encoded by the Kcnma1 gene, and a splicing sequence defines its mitochondrial locationDelayed neuronal preconditioning by NS1619 is independent of calcium activated potassium channels.KCNMA1 encoded cardiac BK channels afford protection against ischemia-reperfusion injury.Morphine induces preconditioning via activation of mitochondrial K(Ca) channelsAntecedent ethanol attenuates cerebral ischemia/reperfusion-induced leukocyte-endothelial adhesive interactions and delayed neuronal death: role of large conductance, Ca2+-activated K+ channelsMitochondrial K+ channels are involved in ischemic postconditioning in rat hearts.BK channel activators and their therapeutic perspectivesAntecedent hydrogen sulfide elicits an anti-inflammatory phenotype in postischemic murine small intestine: role of BK channels.Mitochondrial ion channels: gatekeepers of life and death.Mitochondrial BKCa channels contribute to protection of cardiomyocytes isolated from chronically hypoxic rats.Hydrogen sulfide preconditioning or neutrophil depletion attenuates ischemia-reperfusion-induced mitochondrial dysfunction in rat small intestine.Cardiac sodium/calcium exchanger preconditioning promotes anti-arrhythmic and cardioprotective effects through mitochondrial calcium-activated potassium channel.Mitochondrial ion channels.Immediate neuronal preconditioning by NS1619.Cardioprotective signaling to mitochondriaPhysiology of potassium channels in the inner membrane of mitochondria.Intracellular BK(Ca) (iBK(Ca)) channels.Mitochondrial channels: ion fluxes and more.Mitochondrial potassium channels as pharmacological target for cardioprotective drugs.Preconditioning with the BKCa Channel Activator NS-1619 Prevents Ischemia/Reperfusion-Induced Inflammation and Mucosal Barrier Dysfunction: ROS and HO-1.The Slo(w) path to identifying the mitochondrial channels responsible for ischemic protection.Role of large-conductance Ca²+-activated K+ channels in adenosine A₁ receptor-mediated pharmacological postconditioning in H9c2 cells.A non-cardiomyocyte autonomous mechanism of cardioprotection involving the SLO1 BK channel.Cardiac mitochondrial preconditioning by Big Ca2+-sensitive K+ channel opening requires superoxide radical generation.Mitochondrial Ca2+-induced K+ influx increases respiration and enhances ROS production while maintaining membrane potential.Pharmacological and physiological stimuli do not promote Ca(2+)-sensitive K+ channel activity in isolated heart mitochondria.
P2860
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P2860
Opening of Ca2+-activated K+ channels triggers early and delayed preconditioning against I/R injury independent of NOS in mice.
description
2004 nî lūn-bûn
@nan
2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
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2004年學術文章
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name
Opening of Ca2+-activated K+ c ...... ry independent of NOS in mice.
@en
Opening of Ca2+-activated K+ c ...... ry independent of NOS in mice.
@nl
type
label
Opening of Ca2+-activated K+ c ...... ry independent of NOS in mice.
@en
Opening of Ca2+-activated K+ c ...... ry independent of NOS in mice.
@nl
prefLabel
Opening of Ca2+-activated K+ c ...... ry independent of NOS in mice.
@en
Opening of Ca2+-activated K+ c ...... ry independent of NOS in mice.
@nl
P2093
P2860
P1476
Opening of Ca2+-activated K+ c ...... ury independent of NOS in mice
@en
P2093
Rakesh C Kukreja
Xiaoyin Wang
P2860
P304
P356
10.1152/AJPHEART.00431.2004
P50
P577
2004-06-24T00:00:00Z