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ErbB2 and ErbB3 receptors mediate inhibition of calcium-dependent chloride secretion in colonic epithelial cellsApical leptin induces chloride secretion by intestinal epithelial cells and in a rat model of acute chemotherapy-induced colitisCarbachol-stimulated transactivation of epidermal growth factor receptor and mitogen-activated protein kinase in T(84) cells is mediated by intracellular Ca2+, PYK-2, and p60(src).Characterization of AQPs in Mouse, Rat, and Human Colon and Their Selective Regulation by Bile Acids.Bile acids in regulation of intestinal physiology.Epithelial acetylcholine--a new paradigm for cholinergic regulation of intestinal fluid and electrolyte transport.The Farnesoid X Receptor: Good for BAD.Ursodeoxycholic acid inhibits TNFα-induced IL-8 release from monocytes.JNK mitogen-activated protein kinase limits calcium-dependent chloride secretion across colonic epithelial cells.Bradykinin regulates human colonic ion transport in vitro.Induction of Na+/K+/2Cl- cotransporter expression mediates chronic potentiation of intestinal epithelial Cl- secretion by EGF.Bile acid-induced secretion in polarized monolayers of T84 colonic epithelial cells: Structure-activity relationships.Transactivation of the epidermal growth factor receptor mediates muscarinic stimulation of focal adhesion kinase in intestinal epithelial cells.Gs protein-coupled receptor agonists induce transactivation of the epidermal growth factor receptor in T84 cells: implications for epithelial secretory responses.Immune regulation of human colonic electrolyte transport in vitro.The bile acid receptor, TGR5, regulates basal and cholinergic-induced secretory responses in rat colon.Bile acids stimulate chloride secretion through CFTR and calcium-activated Cl- channels in Calu-3 airway epithelial cells.Insulin and IGF-I inhibit calcium-dependent chloride secretion by T84 human colonic epithelial cells.p38 mitogen-activated protein kinase inhibits calcium-dependent chloride secretion in T84 colonic epithelial cells.New highly toxic bile acids derived from deoxycholic acid, chenodeoxycholic acid and lithocholic acid.Carbachol stimulates transactivation of epidermal growth factor receptor and mitogen-activated protein kinase in T84 cells. Implications for carbachol-stimulated chloride secretion.Bile acids differentially impact on platelet activation.The hydroxylase inhibitor dimethyloxalylglycine is protective in a murine model of colitis.Epidermal growth factor chronically upregulates Ca(2+)-dependent Cl(-) conductance and TMEM16A expression in intestinal epithelial cells.Regulation of ion transport by histamine in human colonMissing link identified: GpBAR1 is a neuronal bile acid receptorHydroxylase inhibition attenuates colonic epithelial secretory function and ameliorates experimental diarrheaUrsodeoxycholic acid attenuates colonic epithelial secretory functionThe alpha2beta1 integrin is a necessary co-receptor for collagen-induced activation of Syk and the subsequent phosphorylation of phospholipase Cgamma2 in plateletsThe secondary bile acids, ursodeoxycholic acid and lithocholic acid, protect against intestinal inflammation by inhibition of epithelial apoptosis
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description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Stephen Keely
@ast
Stephen Keely
@en
Stephen Keely
@es
Stephen Keely
@nl
Stephen Keely
@sl
type
label
Stephen Keely
@ast
Stephen Keely
@en
Stephen Keely
@es
Stephen Keely
@nl
Stephen Keely
@sl
prefLabel
Stephen Keely
@ast
Stephen Keely
@en
Stephen Keely
@es
Stephen Keely
@nl
Stephen Keely
@sl
P106
P21
P31
P496
0000-0002-6315-3587