Smad3 initiates oxidative stress and proteolysis that underlies diaphragm dysfunction during mechanical ventilation.

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Smad3 initiates oxidative stress and proteolysis that underlies diaphragm dysfunction during mechanical ventilation.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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Smad3 initiates oxidative stre ...... during mechanical ventilation.

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Smad3 initiates oxidative stre ...... during mechanical ventilation.

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Smad3 initiates oxidative stre ...... during mechanical ventilation.

@en

Smad3 initiates oxidative stre ...... during mechanical ventilation.

@nl

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Smad3 initiates oxidative stre ...... during mechanical ventilation.

@en

Smad3 initiates oxidative stre ...... during mechanical ventilation.

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P1476

Smad3 initiates oxidative stre ...... during mechanical ventilation

@en

P2093

Amir Tehrani

http://www.w3.org/2001/XMLSchema#string

Catherine L Kennedy

http://www.w3.org/2001/XMLSchema#string

Danielle A Fraga

http://www.w3.org/2001/XMLSchema#string

Francesca Olguin

http://www.w3.org/2001/XMLSchema#string

Hui Xia

http://www.w3.org/2001/XMLSchema#string

Huibin Tang

http://www.w3.org/2001/XMLSchema#string

Joseph B Shrager

http://www.w3.org/2001/XMLSchema#string

Kelsey Ayers

http://www.w3.org/2001/XMLSchema#string

Michael Kim

http://www.w3.org/2001/XMLSchema#string

Myung Lee

http://www.w3.org/2001/XMLSchema#string

P2860

Dysfunction of respiratory muscles in critically ill patients on the intensive care unit

A histone deacetylase 4/myogenin positive feedback loop coordinates denervation-dependent gene induction and suppression

Transforming growth factor beta (TGF-beta) signaling is regulated by electrical activity in skeletal muscle cells. TGF-beta type I receptor is transcriptionally regulated by myotube excitability

The histone deacetylase HDAC4 connects neural activity to muscle transcriptional reprogramming

Effect of intermittent phrenic nerve stimulation during cardiothoracic surgery on mitochondrial respiration in the human diaphragm

Inhibition of Janus kinase signaling during controlled mechanical ventilation prevents ventilation-induced diaphragm dysfunction

Oxidative stress is required for mechanical ventilation-induced protease activation in the diaphragm.

Mechanical ventilation induces diaphragmatic mitochondrial dysfunction and increased oxidant production

Prolonged mechanical ventilation alters diaphragmatic structure and function.

Assist-control mechanical ventilation attenuates ventilator-induced diaphragmatic dysfunction.

P2888

s41598-017-11978-4

P304

14530

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P31

scholarly article

P356

10.1038/S41598-017-11978-4

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P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Thomas A. Rando

P577

2017-11-06T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29109401

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P921

proteolysis

mechanical ventilation

P932

5673963

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