Mechanoporation induced by diffuse traumatic brain injury: an irreversible or reversible response to injury?
about
A possible role for integrin signaling in diffuse axonal injuryThe young brain and concussion: imaging as a biomarker for diagnosis and prognosisTraumatic Axonal Injury: Mechanisms and Translational OpportunitiesAcute death of astrocytes in blast-exposed rat organotypic hippocampal slice culturesTrauma-induced plasmalemma disruptions in three-dimensional neural cultures are dependent on strain modality and rate.Acute plasmalemma permeability and protracted clearance of injured cells after controlled cortical impact in mice.Extensive mononuclear infiltration and myogenesis characterize recovery of dysferlin-null skeletal muscle from contraction-induced injuries.Novel model of frontal impact closed head injury in the rat.A plasma membrane wound proteome: reversible externalization of intracellular proteins following reparable mechanical damage.A modified controlled cortical impact technique to model mild traumatic brain injury mechanics in mice.A Mechanistic End-to-End Concussion Model That Translates Head Kinematics to Neurologic Injury.Pathology dynamics predict spinal cord injury therapeutic successPEG-PDLLA micelle treatment improves axonal function of the corpus callosum following traumatic brain injuryDiminished brain resilience syndrome: A modern day neurological pathology of increased susceptibility to mild brain trauma, concussion, and downstream neurodegeneration.Diffuse brain injury elevates tonic glutamate levels and potassium-evoked glutamate release in discrete brain regions at two days post-injury: an enzyme-based microelectrode array study.Diffuse axonal injury in brain trauma: insights from alterations in neurofilaments.Moderately elevated intracranial pressure after diffuse traumatic brain injury is associated with exacerbated neuronal pathology and behavioral morbidity in the rat.Rate of neurodegeneration in the mouse controlled cortical impact model is influenced by impactor tip shape: implications for mechanistic and therapeutic studies.Experimental subarachnoid haemorrhage results in multifocal axonal injury.Current status of fluid biomarkers in mild traumatic brain injuryTherapy development for diffuse axonal injury.Mechanical membrane injury induces axonal beading through localized activation of calpainBiochemical, structural, and biomarker evidence for calpain-mediated cytoskeletal change after diffuse brain injury uncomplicated by contusion.Addressing the needs of traumatic brain injury with clinical proteomics.Calpain as a therapeutic target in traumatic brain injury.The pathophysiology of concussions in youth.Understanding the neuroinflammatory response following concussion to develop treatment strategies.Mechanisms of neuronal membrane sealing following mechanical trauma.Neurochemical cascade of concussion.Treatment of persistent post-concussion syndrome due to mild traumatic brain injury: current status and future directions.Necrostatin-1 reduces histopathology and improves functional outcome after controlled cortical impact in mice.Traumatically injured astrocytes release a proteomic signature modulated by STAT3-dependent cell survival.Plasmalemma permeability and necrotic cell death phenotypes after intracerebral hemorrhage in mice.Rapid neuroinflammatory response localized to injured neurons after diffuse traumatic brain injury in swine.Impact of Neuronal Membrane Damage on the Local Field Potential in a Large-Scale Simulation of Cerebral CortexKollidon VA64, a membrane-resealing agent, reduces histopathology and improves functional outcome after controlled cortical impact in mice.New astroglial injury-defined biomarkers for neurotrauma assessment.Increased intracranial pressure after diffuse traumatic brain injury exacerbates neuronal somatic membrane poration but not axonal injury: evidence for primary intracranial pressure-induced neuronal perturbation.Dicyclomine, an M1 muscarinic antagonist, reduces biomarker levels, but not neuronal degeneration, in fluid percussion brain injury.Tumor necrosis factor alpha and Fas receptor contribute to cognitive deficits independent of cell death after concussive traumatic brain injury in mice.
P2860
Q21135274-FB44A8DB-6F0B-43AF-B67A-D34B6106A7C6Q24606139-214B9107-83D0-4DFC-80F6-70E59F163696Q26753059-629EFCD1-8C00-48E2-B2CD-2D1EEF46C342Q30361334-568A388D-E4EF-4109-A1E3-859434DC7CA0Q30461911-734A2142-5C1F-49DB-A7F6-087F2A9B6BAAQ33294878-854C314D-889D-48C8-9A3E-195A352F1870Q33655550-4B93BF03-C3E9-4E7E-9D79-CC884C777BE1Q33666550-F857A69F-C79F-46F9-9FD8-D5E3F1846FDAQ33681733-F15EC934-847A-4F45-8622-9224082054EEQ33771195-69F2A64E-09F5-4361-8B65-675875A92E02Q33799878-9963F189-EAE8-48D4-9330-0D05EA3F2AA5Q33805235-A85F0D5E-A606-4716-9FC6-5BE9F8864E74Q33847572-D72BA559-71EB-4D5F-8A93-5D49670B8916Q33887236-F2D3A0A1-C5E7-42D6-A28B-D857A2E19D89Q34147858-7B11D6D5-E88F-4635-9F52-F5E3616FC62FQ34722271-9FE1FD25-E82F-40E3-B916-EE219875DE78Q34727074-97996FD6-DAB1-4121-91DE-60589B591C11Q35556472-C9AF8A24-ECA3-4759-AF0B-BCF657ACD18DQ36041412-1C4ABD77-6AC7-4690-A89A-FEB93BB31711Q36424343-C3C86639-C4A0-46B6-95CD-0FA9746BCB97Q36768885-C6917BA4-061F-48E2-B66C-304C0F717DD9Q37354335-B53B9450-E986-4D9A-8CDC-D110D4937A74Q37425364-D6B82E0E-F55B-42DE-B99C-50349AE0B811Q37685775-8B10181F-3AB5-44A7-9851-BA08ECF0A4B0Q37686765-4D87EC08-350F-42F6-B026-28F476D642B5Q37952410-0FBDEEE4-AC59-406C-9614-10199E78FBFCQ38068175-7998CC90-40E5-477D-B85A-273E61369C99Q38226141-2232954B-B2AC-42C4-97B4-1601995997F3Q38318040-A157F44A-926A-473B-89F5-2478C4675188Q38849337-5257E837-BC73-477F-9D4B-6C196017981AQ39365993-5A9206DC-0617-4DBF-BF72-9ED329F7C9A0Q39421531-017D40EB-FB52-44C9-A3B4-AE7515EA1797Q39444673-AE4AD4A3-450C-47E4-BB5F-7D802AD9D1C9Q40383266-0A6271FF-46C8-4FEA-BCA6-2B07282B4E10Q41815737-8F53937A-5041-495A-8CEB-EA94F842992FQ41972006-BBB79184-3064-415B-8247-2B07E4117077Q42005136-9D46B75A-7B71-4205-97AA-C2A541736411Q42157107-2EA268C9-64AF-4A26-BF1E-9A90C717D995Q42182877-C0CE5664-9028-492C-B156-E1C9E8F3B0FBQ42653063-F44DDA2C-0A8B-42E5-9BF0-605C9B6F551B
P2860
Mechanoporation induced by diffuse traumatic brain injury: an irreversible or reversible response to injury?
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh
2006年學術文章
@zh-hant
name
Mechanoporation induced by dif ...... reversible response to injury?
@en
Mechanoporation induced by dif ...... reversible response to injury?
@nl
type
label
Mechanoporation induced by dif ...... reversible response to injury?
@en
Mechanoporation induced by dif ...... reversible response to injury?
@nl
prefLabel
Mechanoporation induced by dif ...... reversible response to injury?
@en
Mechanoporation induced by dif ...... reversible response to injury?
@nl
P2093
P1476
Mechanoporation induced by dif ...... reversible response to injury?
@en
P2093
John T Povlishock
Jonathan Lifshitz
Orsolya Farkas
P304
P356
10.1523/JNEUROSCI.5119-05.2006
P407
P577
2006-03-01T00:00:00Z