TNFR gene-modified mesenchymal stem cells attenuate inflammation and cardiac dysfunction following MI.
about
Genetic engineering of mesenchymal stem cells and its application in human disease therapy.Urban air pollution produces up-regulation of myocardial inflammatory genes and dark chocolate provides cardioprotection.In vitro augmentation of mesenchymal stem cells viability in stressful microenvironments : In vitro augmentation of mesenchymal stem cells viabilityStem cells for cardiac repair: status, mechanisms, and new strategies.AAV vectors for cardiac gene transfer: experimental tools and clinical opportunities.Mesenchymal stem cells for cardiac regeneration: translation to bedside realityIntra-city Differences in Cardiac Expression of Inflammatory Genes and Inflammasomes in Young Urbanites: A Pilot StudyRole of TNF-alpha in vascular dysfunction.Recent progress on tissue-resident adult stem cell biology and their therapeutic implications.Myocardial regeneration by transplantation of modified endothelial progenitor cells expressing SDF-1 in a rat model.Up-regulation of mRNA ventricular PRNP prion protein gene expression in air pollution highly exposed young urbanites: endoplasmic reticulum stress, glucose regulated protein 78, and nanosized particles.Comparative mRNA and MicroRNA Profiling during Acute Myocardial Infarction Induced by Coronary Occlusion and Ablation Radio-Frequency Currents.Modification of mesenchymal stem cells for cardiac regeneration.Optimizing stem cell function for the treatment of ischemic heart diseaseCardiac cell therapy: boosting mesenchymal stem cells effects.Perspective and challenges of mesenchymal stem cells for cardiovascular regeneration.Nanofiber for cardiovascular tissue engineering.Adeno-Associated Virus Vectors and Stem Cells: Friends or Foes?Rebuilding the Damaged Heart: Mesenchymal Stem Cells, Cell-Based Therapy, and Engineered Heart Tissue.Promise of adeno-associated virus as a gene therapy vector for cardiovascular diseases.Tumor necrosis factor-α signaling via TNFR1/p55 is deleterious whereas TNFR2/p75 signaling is protective in adult infarct myocardium.Tumor necrosis factor-α and its role as a mediator in myocardial infarction: A brief review.Critical Role of Tumor Necrosis Factor Signaling in Mesenchymal Stem Cell-Based Therapy for Autoimmune and Inflammatory Diseases
P2860
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P2860
TNFR gene-modified mesenchymal stem cells attenuate inflammation and cardiac dysfunction following MI.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
TNFR gene-modified mesenchymal ...... diac dysfunction following MI.
@en
TNFR gene-modified mesenchymal ...... diac dysfunction following MI.
@nl
type
label
TNFR gene-modified mesenchymal ...... diac dysfunction following MI.
@en
TNFR gene-modified mesenchymal ...... diac dysfunction following MI.
@nl
prefLabel
TNFR gene-modified mesenchymal ...... diac dysfunction following MI.
@en
TNFR gene-modified mesenchymal ...... diac dysfunction following MI.
@nl
P2093
P2860
P1476
TNFR gene-modified mesenchymal ...... diac dysfunction following MI.
@en
P2093
P2860
P356
10.1080/14017430701543556
P577
2008-02-01T00:00:00Z