Rectal taurocholate increases L cell and insulin secretion, and decreases blood glucose and food intake in obese type 2 diabetic volunteers.
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How do patients' clinical phenotype and the physiological mechanisms of the operations impact the choice of bariatric procedure?Stimulation of incretin secreting cellsPharmacology and physiology of gastrointestinal enteroendocrine cellsImproved glucose metabolism following bariatric surgery is associated with increased circulating bile acid concentrations and remodeling of the gut microbiomeGender Differences in Bile Acids and Microbiota in Relationship with Gender Dissimilarity in Steatosis Induced by Diet and FXR Inactivation.Hypertrophy dependent doubling of L-cells in Roux-en-Y gastric bypass operated rats.An integrated epigenomic analysis for type 2 diabetes susceptibility loci in monozygotic twins.The physiology underlying Roux-en-Y gastric bypass: a status report.Gut chemosensing mechanisms.Bariatric surgery: prevalence, predictors, and mechanisms of diabetes remission.Temporal changes in bile acid levels and 12α-hydroxylation after Roux-en-Y gastric bypass surgery in type 2 diabetes.Determinants of post-prandial plasma bile acid kinetics in human volunteers.Systemic bile acid sensing by G protein-coupled bile acid receptor 1 (GPBAR1) promotes PYY and GLP-1 release.Relationships between gastric emptying, postprandial glycemia, and incretin hormonesMolecular mechanisms of incretin hormone secretion.Neuroendocrinology of obesity.Bile acid sequestrants: glucose-lowering mechanisms and efficacy in type 2 diabetes.The effect of a tertiary bile acid, taurocholic acid, on the morphology and physical characteristics of microencapsulated probucol: potential applications in diabetes: a characterization study.The Contributing Role of Bile Acids to Metabolic Improvements After Obesity and Metabolic Surgery.Inter-regulation of gastric emptying and incretin hormone secretion: implications for postprandial glycemic control.Intestinal bile acid receptors are key regulators of glucose homeostasis.New insights into the anti-diabetic actions of metformin: from the liver to the gut.Ghrelin, CCK, GLP-1, and PYY(3-36): Secretory Controls and Physiological Roles in Eating and Glycemia in Health, Obesity, and After RYGB.Mechanisms of Action of Surgical Interventions on Weight-Related Diseases: the Potential Role of Bile Acids.Evidence connecting old, new and neglected glucose-lowering drugs to bile acid-induced GLP-1 secretion: A review.Effect of chenodeoxycholic acid and the bile acid sequestrant colesevelam on glucagon-like peptide-1 secretion.Difference in postprandial GLP-1 response despite similar glucose kinetics after consumption of wheat breads with different particle size in healthy men.Chenodeoxycholic acid stimulates glucagon-like peptide-1 secretion in patients after Roux-en-Y gastric bypass.Enhanced fasting and post-prandial plasma bile acid responses after Roux-en-Y gastric bypass surgery.Superior reductions in hepatic steatosis and fibrosis with co-administration of a glucagon-like peptide-1 receptor agonist and obeticholic acid in mice.The bile acid-sequestering resin sevelamer eliminates the acute GLP-1 stimulatory effect of endogenously released bile acids in patients with type 2 diabetes.Targeted intestinal delivery of incretin secretagogues-towards new diabetes and obesity therapies.Bile acids are important direct and indirect regulators of the secretion of appetite- and metabolism-regulating hormones from the gut and pancreas.A combination of Lactobacillus mali APS1 and dieting improved the efficacy of obesity treatment via manipulating gut microbiome in mice.Obesity diabetes and the role of bile acids in metabolismTargeting the intestinal L-cell for obesity and type 2 diabetes treatmentDissecting the Physiology and Pathophysiology of Glucagon-Like Peptide-1Do colorectal cancer resections improve diabetes in long-term survivors? A case–control study
P2860
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P2860
Rectal taurocholate increases L cell and insulin secretion, and decreases blood glucose and food intake in obese type 2 diabetic volunteers.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh-hant
name
Rectal taurocholate increases ...... se type 2 diabetic volunteers.
@en
Rectal taurocholate increases ...... se type 2 diabetic volunteers.
@nl
type
label
Rectal taurocholate increases ...... se type 2 diabetic volunteers.
@en
Rectal taurocholate increases ...... se type 2 diabetic volunteers.
@nl
prefLabel
Rectal taurocholate increases ...... se type 2 diabetic volunteers.
@en
Rectal taurocholate increases ...... se type 2 diabetic volunteers.
@nl
P2093
P2860
P1433
P1476
Rectal taurocholate increases ...... se type 2 diabetic volunteers.
@en
P2093
J Al Kaabi
K A Parekh
N Nagelkerke
S A Thomas
S Gariballa
T E Adrian
P2860
P2888
P304
P356
10.1007/S00125-012-2593-2
P577
2012-06-14T00:00:00Z