Decrease of Hsp25 protein expression precedes degeneration of motoneurons in ALS-SOD1 mice.
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Activation of the heat shock response in a primary cellular model of motoneuron neurodegeneration-evidence for neuroprotective and neurotoxic effectsA comprehensive assessment of the SOD1G93A low-copy transgenic mouse, which models human amyotrophic lateral sclerosis.Gamma-synucleinopathy: neurodegeneration associated with overexpression of the mouse protein.Aluminum hydroxide injections lead to motor deficits and motor neuron degenerationArimoclomol: a potential therapy under development for ALS.Early-stage treatment with Withaferin A reduces levels of misfolded superoxide dismutase 1 and extends lifespan in a mouse model of amyotrophic lateral sclerosis.Modeling pain in vitro using nociceptor neurons reprogrammed from fibroblasts.Seeking homeostasis: temporal trends in respiration, oxidation, and calcium in SOD1 G93A Amyotrophic Lateral Sclerosis mice.Mutant HSPB1 overexpression in neurons is sufficient to cause age-related motor neuronopathy in miceSmall heat shock proteins in inherited peripheral neuropathies.Detergent-insoluble aggregates associated with amyotrophic lateral sclerosis in transgenic mice contain primarily full-length, unmodified superoxide dismutase-1.Differential regulation of small heat shock proteins in transgenic mouse models of neurodegenerative diseases.The small heat shock proteins αB-crystallin and Hsp27 suppress SOD1 aggregation in vitro.The Expression of NMDA Receptor 1 Correlates with Clinicopathological Parameters in Cutaneous Squamous Cell Carcinoma.Mechanisms of compartmental purkinje cell death and survival in the lurcher mutant mouse.The protective and therapeutic function of small heat shock proteins in neurological diseases.Different anti-aggregation and pro-degradative functions of the members of the mammalian sHSP family in neurological disorders.A novel p.T139M mutation in HSPB1 highlighting the phenotypic spectrum in a family.HSPB1 mutations causing hereditary neuropathy in humans disrupt non-cell autonomous protection of motor neurons.Age-related changes in HSP25 expression in basal ganglia and cortex of F344/BN rats.Heat-shock protein 105 interacts with and suppresses aggregation of mutant Cu/Zn superoxide dismutase: clues to a possible strategy for treating ALS.The heat shock response in neurons and astroglia and its role in neurodegenerative diseases.Heat shock proteins in neurodegenerative diseases: pathogenic roles and therapeutic implications.Spinal inhibitory interneuron pathology follows motor neuron degeneration independent of glial mutant superoxide dismutase 1 expression in SOD1-ALS mice.Novel insights in the disease biology of mutant small heat shock proteins in neuromuscular diseases.Inhibition of Chaperone Activity Is a Shared Property of Several Cu,Zn-Superoxide Dismutase Mutants That Cause Amyotrophic Lateral Sclerosis
P2860
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P2860
Decrease of Hsp25 protein expression precedes degeneration of motoneurons in ALS-SOD1 mice.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年學術文章
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name
Decrease of Hsp25 protein expr ...... motoneurons in ALS-SOD1 mice.
@en
Decrease of Hsp25 protein expr ...... motoneurons in ALS-SOD1 mice.
@nl
type
label
Decrease of Hsp25 protein expr ...... motoneurons in ALS-SOD1 mice.
@en
Decrease of Hsp25 protein expr ...... motoneurons in ALS-SOD1 mice.
@nl
prefLabel
Decrease of Hsp25 protein expr ...... motoneurons in ALS-SOD1 mice.
@en
Decrease of Hsp25 protein expr ...... motoneurons in ALS-SOD1 mice.
@nl
P2093
P2860
P1476
Decrease of Hsp25 protein expr ...... motoneurons in ALS-SOD1 mice.
@en
P2093
Angela Vlug
Arjen Maatkamp
Dick Jaarsma
Dirk Troost
Elize Haasdijk
Pim J French
P2860
P356
10.1111/J.1460-9568.2004.03430.X
P407
P577
2004-07-01T00:00:00Z