The role of vasopressin V1A receptors in cytotoxic brain edema formation following brain injury.
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Real-time monitoring of changes in brain extracellular sodium and potassium concentrations and intracranial pressure after selective vasopressin-1a receptor inhibition following focal traumatic brain injury in rats.Antagonists of the Vasopressin V1 Receptor and of the β(1)-Adrenoceptor Inhibit Cytotoxic Brain Edema in Stroke by Effects on Astrocytes - but the Mechanisms Differ.Selective vasopressin-1a receptor antagonist prevents brain edema, reduces astrocytic cell swelling and GFAP, V1aR and AQP4 expression after focal traumatic brain injury.Synergistic interactions between cytokines and AVP at the blood-CSF barrier result in increased chemokine production and augmented influx of leukocytes after brain injuryArginine-Vasopressin Receptor Blocker Conivaptan Reduces Brain Edema and Blood-Brain Barrier Disruption after Experimental Stroke in MiceRole of vasopressin and its antagonism in stroke related edema.Recent advances and future directions in preclinical research of arginine-vasopressin (AVP) receptor blocker conivaptan in the context of stroke.Exploring the Virchow-Robin spaces function: A unified theory of brain diseases.Intranasal Administration of the Antisecretory Peptide AF-16 Reduces Edema and Improves Cognitive Function Following Diffuse Traumatic Brain Injury in the Rat.Alpha-1 antichymotrypsin is involved in astrocyte injury in concert with arginine-vasopressin during the development of acute hepatic encephalopathy.Evaluating the involvement of cerebral microvascular endothelial Na(+)/K(+)-ATPase and Na(+)-K(+)-2Cl(-) co-transporter in electrolyte fluxes in an in vitro blood-brain barrier model of dehydration.Conivaptan, a Selective Arginine Vasopressin V1a and V2 Receptor Antagonist Attenuates Global Cerebral Edema Following Experimental Cardiac Arrest via Perivascular Pool of Aquaporin-4.Effect of estrogen and/or progesterone administration on traumatic brain injury-caused brain edema: the changes of aquaporin-4 and interleukin-6.Increased brain volume among good grade patients with intracerebral hemorrhage. Results from the Antihypertensive Treatment of Acute Cerebral Hemorrhage (ATACH) study.
P2860
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P2860
The role of vasopressin V1A receptors in cytotoxic brain edema formation following brain injury.
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2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年學術文章
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name
The role of vasopressin V1A re ...... mation following brain injury.
@en
The role of vasopressin V1A re ...... mation following brain injury.
@nl
type
label
The role of vasopressin V1A re ...... mation following brain injury.
@en
The role of vasopressin V1A re ...... mation following brain injury.
@nl
prefLabel
The role of vasopressin V1A re ...... mation following brain injury.
@en
The role of vasopressin V1A re ...... mation following brain injury.
@nl
P2093
P2860
P1433
P1476
The role of vasopressin V1A re ...... mation following brain injury.
@en
P2093
Andrea Kleindienst
Anthony Marmarou
Jana G Dunbar
Renee Glisson
P2860
P2888
P304
P356
10.1007/S00701-012-1558-Z
P577
2012-11-28T00:00:00Z