Babesia and plasmodia increase host erythrocyte permeability through distinct mechanisms.
about
Fosmidomycin uptake into Plasmodium and Babesia-infected erythrocytes is facilitated by parasite-induced new permeability pathwaysSynergistic Malaria Parasite Killing by Two Types of Plasmodial Surface Anion Channel InhibitorsExtensive Shared Chemosensitivity between Malaria and Babesiosis Blood-Stage Parasites.Hemoparasites in a wild primate: Infection patterns suggest interaction of Plasmodium and Babesia in a lemur speciesProteolysis at a specific extracellular residue implicates integral membrane CLAG3 in malaria parasite nutrient channelsMalaria parasite mutants with altered erythrocyte permeability: a new drug resistance mechanism and important molecular tool.Suppression of Plasmodium cynomolgi in rhesus macaques by coinfection with Babesia microti.Altered plasmodial surface anion channel activity and in vitro resistance to permeating antimalarial compounds.Malaria parasite clag3 genes determine channel-mediated nutrient uptake by infected red blood cells.The conserved clag multigene family of malaria parasites: essential roles in host-pathogen interactionApicoplast isoprenoid precursor synthesis and the molecular basis of fosmidomycin resistance in Toxoplasma gondii.A CLAG3 mutation in an amphipathic transmembrane domain alters malaria parasite nutrient channels and confers leupeptin resistance.A blasticidin S-resistant Plasmodium falciparum mutant with a defective plasmodial surface anion channelDetection and quantification of subtle changes in red blood cell density using a cell phone.Solute restriction reveals an essential role for clag3-associated channels in malaria parasite nutrient acquisition.Complex inheritance of the plasmodial surface anion channel in a Plasmodium falciparum genetic cross.Role of gap junctions and hemichannels in parasitic infections.Natural Babesia bovis Infection in Water Buffaloes (Bubalus bubalis) and Crossbred Cattle under Field Conditions in Egypt: a Preliminary Study.Continuous separation of cells by balanced dielectrophoretic forces at multiple frequencies.Status of lipid peroxidation and antioxidant enzymes in goats naturally infected with Babesia ovis.Insights gained from P. falciparum cultivation in modified media.Malaria parasite proteins involved in nutrient channels at the host erythrocyte membrane: advances and questions for future research
P2860
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P2860
Babesia and plasmodia increase host erythrocyte permeability through distinct mechanisms.
description
2006 nî lūn-bûn
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2006年の論文
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年学术文章
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2006年學術文章
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name
Babesia and plasmodia increase host erythrocyte permeability through distinct mechanisms.
@en
Babesia and plasmodia increase host erythrocyte permeability through distinct mechanisms.
@nl
type
label
Babesia and plasmodia increase host erythrocyte permeability through distinct mechanisms.
@en
Babesia and plasmodia increase host erythrocyte permeability through distinct mechanisms.
@nl
prefLabel
Babesia and plasmodia increase host erythrocyte permeability through distinct mechanisms.
@en
Babesia and plasmodia increase host erythrocyte permeability through distinct mechanisms.
@nl
P2860
P1476
Babesia and plasmodia increase host erythrocyte permeability through distinct mechanisms.
@en
P2093
Abdulnaser Alkhalil
Sanjay A Desai
P2860
P304
P356
10.1111/J.1462-5822.2006.00834.X
P577
2006-11-03T00:00:00Z