Increased formation of pyridinoline cross-links due to higher telopeptide lysyl hydroxylase levels is a general fibrotic phenomenon. - Wikidata - awgldk - TriplyDB

Increased formation of pyridinoline cross-links due to higher telopeptide lysyl hydroxylase levels is a general fibrotic phenomenon.

Increased formation of pyridinoline cross-links due to higher telopeptide lysyl hydroxylase levels is a general fibrotic phenomenon.

http://www.wikidata.org/entity/Q45008820

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Signaling in Fibrosis: TGF-β, WNT, and YAP/TAZ Converge Unravelling osteoarthritis-related synovial fibrosis: a step closer to solving joint stiffness Recent developments in myofibroblast biology: paradigms for connective tissue remodeling Interleukin-4 Receptor α Signaling in Myeloid Cells Controls Collagen Fibril Assembly in Skin Repair.NADPH oxidase enzymes in skin fibrosis: molecular targets and therapeutic agents Fox-2 protein regulates the alternative splicing of scleroderma-associated lysyl hydroxylase 2 messenger RNA Ochratoxin A-induced renal cortex fibrosis and epithelial-to-mesenchymal transition: molecular mechanisms of ochratoxin A-injury and potential effects of red wine.Extracellular matrix genes as hypoxia-inducible targets.Hypoxia and the extracellular matrix: drivers of tumour metastasis Interleukin-1β attenuates myofibroblast formation and extracellular matrix production in dermal and lung fibroblasts exposed to transforming growth factor-β1 Genomic structure and embryonic expression of zebrafish lysyl hydroxylase 1 and lysyl hydroxylase 2.A scalable lysyl hydroxylase 2 expression system and luciferase-based enzymatic activity assay.Effect of strain magnitude on the tissue properties of engineered cardiovascular constructs.Lysine post-translational modifications of collagen Procollagen lysyl hydroxylase 2 is essential for hypoxia-induced breast cancer metastasis.Transcriptional profiling of rapamycin-treated fibroblasts from hypertrophic and keloid scars.Osteitis fibrosa is mediated by Platelet-Derived Growth Factor-A via a phosphoinositide 3-kinase-dependent signaling pathway in a rat model for chronic hyperparathyroidism.Loss of BMP signaling through BMPR1A in osteoblasts leads to greater collagen cross-link maturation and material-level mechanical properties in mouse femoral trabecular compartments.The roles of Tenascin C and Fibronectin 1 in adhesive capsulitis: a pilot gene expression study.Disentangling mechanisms involved in collagen pyridinoline cross-linking: The immunophilin FKBP65 is critical for dimerization of lysyl hydroxylase 2 Deregulation of the lysyl hydroxylase matrix cross-linking system in experimental and clinical bronchopulmonary dysplasia.Understanding fibrosis in systemic sclerosis: shifting paradigms, emerging opportunities.Current concepts in scar evolution and control.Cellular/extracellular matrix cross-talk in scar evolution and control.Matrix mechanics and regulation of the fibroblast phenotype.The pro-fibrotic properties of transforming growth factor on human fibroblasts are counteracted by caffeic acid by inhibiting myofibroblast formation and collagen synthesis.Scar management in burn injuries using drug delivery and molecular signaling: Current treatments and future directions.The myofibroblast, a key cell in normal and pathological tissue repair.Regulation of the collagen cross-linking enzymes LOXL2 and PLOD2 by tumor-suppressive microRNA-26a/b in renal cell carcinoma.Missense mutations that cause Bruck syndrome affect enzymatic activity, folding, and oligomerization of lysyl hydroxylase 2 Reduction of lysyl hydroxylase 3 causes deleterious changes in the deposition and organization of extracellular matrix.Post-translational modifications of collagen upon BMP-induced osteoblast differentiation.Pathophysiological Mechanisms in Sclerosing Skin Diseases.Effects of aging and cyclosporin a on collagen turnover in human gingiva.The IκB kinase inhibitor ACHP strongly attenuates TGFβ1-induced myofibroblast formation and collagen synthesis.Procollagen Lysyl Hydroxylase 2 Expression Is Regulated by an Alternative Downstream Transforming Growth Factor β-1 Activation Mechanism.Loss of Type I Collagen Telopeptide Lysyl Hydroxylation Causes Musculoskeletal Abnormalities in a Zebrafish Model of Bruck Syndrome.ECHO parameters of diastolic dysfunction.A Chaperone Complex Formed by HSP47, FKBP65, and BiP Modulates Telopeptide Lysyl Hydroxylation of Type I Procollagen.Stimulation of fibrotic processes by the infrapatellar fat pad in cultured synoviocytes from patients with osteoarthritis: a possible role for prostaglandin f2α.

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P2860

Increased formation of pyridinoline cross-links due to higher telopeptide lysyl hydroxylase levels is a general fibrotic phenomenon.

http://www.wikidata.org/entity/Q45008820

description

2004 nî lūn-bûn

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Increased formation of pyridin ...... a general fibrotic phenomenon.

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Increased formation of pyridin ...... a general fibrotic phenomenon.

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Increased formation of pyridin ...... a general fibrotic phenomenon.

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Increased formation of pyridin ...... a general fibrotic phenomenon.

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Increased formation of pyridin ...... a general fibrotic phenomenon.

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Increased formation of pyridin ...... a general fibrotic phenomenon.

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Anne-Marie Zuurmond

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Annemarie J van der Slot

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Antoon J van den Bogaerdt

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Esther Middelkoop

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H Karel Ronday

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Jeroen DeGroot

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Ruud A Bank

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10.1016/J.MATBIO.2004.06.001

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