Sevoflurane-induced preconditioning of rat brain in vitro and the role of KATP channels.
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Neuronal preconditioning by inhalational anesthetics: evidence for the role of plasmalemmal adenosine triphosphate-sensitive potassium channelsmiR-15b suppression of Bcl-2 contributes to cerebral ischemic injury and is reversed by sevoflurane preconditioningIsoflurane preconditioning increases endothelial cell tolerance to in-vitro simulated ischaemia.Isoflurane preconditioning improves short-term and long-term neurological outcome after focal brain ischemia in adult rats.Pretreatment with volatile anesthetics, but not with the nonimmobilizer 1,2-dichlorohexafluorocyclobutane, reduced cell injury in rat cerebellar slices after an in vitro simulated ischemiaNeuroprotective effect of volatile anesthetic agents: molecular mechanisms.Prolonged exposure to isoflurane ameliorates infarction severity in the rat pup model of neonatal hypoxia-ischemiaUnexpected prolonged coma after general anesthesia in a patient with history of type II diabetes mellitusInhalational anesthetics as preconditioning agents in ischemic brain.Inhalational anesthetics as neuroprotectants or chemical preconditioning agents in ischemic brainPatents related to therapeutic activation of K(ATP) and K(2P) potassium channels for neuroprotection: ischemic/hypoxic/anoxic injury and general anesthetics.Paradigms and mechanisms of inhalational anesthetics mediated neuroprotection against cerebral ischemic stroke.A review of the practice of sedation with inhalational anaesthetics in the intensive care unit with the AnaConDa(®) device.Cellular signaling pathways and molecular mechanisms involving inhalational anesthetics-induced organoprotection.Duplicate preconditioning with sevoflurane in vitro improves neuroprotection in rat brain via activating the extracellular signal-regulated protein kinase.Isoflurane-induced depolarization of neural mitochondria increases with age.Effects of halothane, isoflurane, and sevoflurane on lipid peroxidation following experimental closed head trauma in rats.Postconditioning with sevoflurane protects against focal cerebral ischemia and reperfusion injury involving mitochondrial ATP-dependent potassium channel and mitochondrial permeability transition pore.Sevoflurane-induced delayed neuroprotection involves mitoK(ATP) channel opening and PKC ε activation.Delayed neuroprotection induced by sevoflurane via opening mitochondrial ATP-sensitive potassium channels and p38 MAPK phosphorylation.Volatile anaesthetics depolarize neural mitochondria by inhibiton of the electron transport chain.Comparison of volatile anesthetic-induced preconditioning in cardiac and cerebral system: molecular mechanisms and clinical aspects.
P2860
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P2860
Sevoflurane-induced preconditioning of rat brain in vitro and the role of KATP channels.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年学术文章
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2004年學術文章
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name
Sevoflurane-induced preconditioning of rat brain in vitro and the role of KATP channels.
@en
Sevoflurane-induced preconditioning of rat brain in vitro and the role of KATP channels.
@nl
type
label
Sevoflurane-induced preconditioning of rat brain in vitro and the role of KATP channels.
@en
Sevoflurane-induced preconditioning of rat brain in vitro and the role of KATP channels.
@nl
prefLabel
Sevoflurane-induced preconditioning of rat brain in vitro and the role of KATP channels.
@en
Sevoflurane-induced preconditioning of rat brain in vitro and the role of KATP channels.
@nl
P2093
P1433
P1476
Sevoflurane-induced preconditioning of rat brain in vitro and the role of KATP channels.
@en
P2093
Avital Schurr
Franz Kehl
Norbert Roewer
Ralphiel S Payne
P356
10.1016/J.BRAINRES.2004.06.038
P407
P577
2004-09-01T00:00:00Z