Celecoxib induces functional recovery after intracerebral hemorrhage with reduction of brain edema and perihematomal cell death. - Wikidata - awgldk - TriplyDB

Celecoxib induces functional recovery after intracerebral hemorrhage with reduction of brain edema and perihematomal cell death.

Celecoxib induces functional recovery after intracerebral hemorrhage with reduction of brain edema and perihematomal cell death.

http://www.wikidata.org/entity/Q45055104

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Preclinical and clinical research on inflammation after intracerebral hemorrhage Immune interventions in stroke Intracerebral Hemorrhage: Perihemorrhagic Edema and Secondary Hematoma Expansion: From Bench Work to Ongoing Controversies Parecoxib is neuroprotective in spontaneously hypertensive rats after transient middle cerebral artery occlusion: a divided treatment response?PGE2 receptor agonist misoprostol protects brain against intracerebral hemorrhage in mice Toxic role of prostaglandin E2 receptor EP1 after intracerebral hemorrhage in mice Cerebroprotection by the neuronal PGE2 receptor EP2 after intracerebral hemorrhage in middle-aged mice Research in people with psychosis risk syndrome: a review of the current evidence and future directions Clinical trials for neuroprotective therapies in intracerebral hemorrhage: a new roadmap from bench to bedside.Effects of genetic deficiency of cyclooxygenase-1 or cyclooxygenase-2 on functional and histological outcomes following traumatic brain injury in mice.New Concept of Neural Stem Cell Transplantation: Anti-inflammatory Role.Mouse models of intracerebral hemorrhage in ventricle, cortex, and hippocampus by injections of autologous blood or collagenase Preclinical Studies and Translational Applications of Intracerebral Hemorrhage.Systemic administration of urocortin after intracerebral hemorrhage reduces neurological deficits and neuroinflammation in rats.Neuroprotection in a rabbit model of intraventricular haemorrhage by cyclooxygenase-2, prostanoid receptor-1 or tumour necrosis factor-alpha inhibition.Experimental intracerebral hemorrhage: avoiding pitfalls in translational research.Treatment targets in intracerebral hemorrhage.Intracerebral hemorrhage outcomes following selective blockade or stimulation of the PGE2 EP1 receptor.The Effect of Human Umbilical Cord Blood-Derived Mesenchymal Stem Cells in a Collagenase-Induced Intracerebral Hemorrhage Rat Model.P2X7R blockade prevents NLRP3 inflammasome activation and brain injury in a rat model of intracerebral hemorrhage: involvement of peroxynitrite Neuroprotective agents in schizophrenia and affective disorders.The effects of cyclooxygenase inhibitors on the brain inflammatory response following traumatic brain injury in rats.Detrimental effects of leptin on intracerebral hemorrhage via the STAT3 signal pathway.Microglial Polarization and Inflammatory Mediators After Intracerebral Hemorrhage.Role of PGE₂ EP1 receptor in intracerebral hemorrhage-induced brain injury Genetic disruption of cyclooxygenase-2 does not improve histological or behavioral outcome after traumatic brain injury in mice Corticorelin acetate injections for the treatment of peritumoral brain edema.Baicalin attenuates brain edema in a rat model of intracerebral hemorrhage.Neuroprotective and neurotrophic curcuminoids to treat stroke: a translational perspective.Advances in neuroprotective strategies: potential therapies for intracerebral hemorrhage.Chemokines and their receptors in intracerebral hemorrhage.Neuroinflammation after intracerebral hemorrhage.Inhibition of neuronal ferroptosis protects hemorrhagic brain Stages of the Inflammatory Response in Pathology and Tissue Repair after Intracerebral Hemorrhage.Elevated blood pressure aggravates intracerebral hemorrhage-induced brain injury Age-dependent effect of treadmill exercise on hemorrhage-induced neuronal cell death in rats.Anti-high mobility group box-1 (HMGB1) antibody inhibits hemorrhage-induced brain injury and improved neurological deficits in rats.MicroRNA-27b inhibition promotes Nrf2/ARE pathway activation and alleviates intracerebral hemorrhage-induced brain injury.Memantine reduces hematoma expansion in experimental intracerebral hemorrhage, resulting in functional improvement.Lithium pretreatment reduces brain injury after intracerebral hemorrhage in rats.

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P2860

Celecoxib induces functional recovery after intracerebral hemorrhage with reduction of brain edema and perihematomal cell death.

http://www.wikidata.org/entity/Q45055104

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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Celecoxib induces functional r ...... and perihematomal cell death.

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Celecoxib induces functional r ...... and perihematomal cell death.

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Celecoxib induces functional r ...... and perihematomal cell death.

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Celecoxib induces functional r ...... and perihematomal cell death.

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Celecoxib induces functional r ...... and perihematomal cell death.

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Celecoxib induces functional r ...... and perihematomal cell death.

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Journal of Cerebral Blood Flow & Metabolism

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Celecoxib induces functional r ...... and perihematomal cell death.

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P2093

Jae-Kyu Roh

http://www.w3.org/2001/XMLSchema#string

Keun-Hwa Jung

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Kon Chu

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Manho Kim

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Sang-Wuk Jeong

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So-Young Han

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Soon-Tae Lee

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P2860

Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation

Spreading depression and focal brain ischemia induce cyclooxygenase-2 in cortical neurons through N-methyl-D-aspartic acid-receptors and phospholipase A2

Evidence for apoptosis after intercerebral hemorrhage in rat striatum

Cyclooxygenases 1 and 2

Effect of FK-506 on inflammation and behavioral outcome following intracerebral hemorrhage in rat.

Interaction between inducible nitric oxide synthase and cyclooxygenase-2 after cerebral ischemia

The coxibs, selective inhibitors of cyclooxygenase-2.

Reduced susceptibility to ischemic brain injury and N-methyl-D-aspartate-mediated neurotoxicity in cyclooxygenase-2-deficient mice.

Cyclooxygenases in central nervous system diseases: a special role for cyclooxygenase 2 in neuronal cell death.

Potentiation of excitotoxicity in transgenic mice overexpressing neuronal cyclooxygenase-2

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