FK506 independently upregulates transforming growth factor beta and downregulates inducible nitric oxide synthase in cultured human keratinocytes: possible mechanisms of how tacrolimus ointment interacts with atopic skin.
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Loss of expression of TGF-βs and their receptors in chronic skin lesions induced by sulfur mustard as compared with chronic contact dermatitis patientsArginase I levels are decreased in the plasma of pediatric patients with atopic dermatitis.A pilot study of 1% pimecrolimus cream for the treatment of childhood segmental vitiligo.Impact on Autophagy and Ultraviolet B Induced Responses of Treatment with the MTOR Inhibitors Rapamycin, Everolimus, Torin 1, and pp242 in Human Keratinocytes.Cilomilast, tacrolimus and rapamycin modulate dendritic cell function in the elicitation phase of allergic contact dermatitis.Pimecrolimus 1% cream for the treatment of steroid-induced rosacea: an 8-week split-face clinical trial.FK506 inhibits tumour necrosis factor-alpha secretion in human keratinocytes via regulation of nuclear factor-kappaB.Treatment Strategies for Hypopigmentation in the Context of Burn Hypertrophic Scars.
P2860
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P2860
FK506 independently upregulates transforming growth factor beta and downregulates inducible nitric oxide synthase in cultured human keratinocytes: possible mechanisms of how tacrolimus ointment interacts with atopic skin.
description
2004 nî lūn-bûn
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2004年の論文
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name
FK506 independently upregulate ...... nt interacts with atopic skin.
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FK506 independently upregulate ...... nt interacts with atopic skin.
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type
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FK506 independently upregulate ...... nt interacts with atopic skin.
@en
FK506 independently upregulate ...... nt interacts with atopic skin.
@nl
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FK506 independently upregulate ...... nt interacts with atopic skin.
@en
FK506 independently upregulate ...... nt interacts with atopic skin.
@nl
P2093
P1476
FK506 independently upregulate ...... nt interacts with atopic skin.
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P304
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10.1111/J.1365-2133.2004.06109.X
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2004-09-01T00:00:00Z