In the absence of IL-12, CD4(+) T cell responses to intracellular pathogens fail to default to a Th2 pattern and are host protective in an IL-10(-/-) setting.
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UNC93B1 is essential for TLR11 activation and IL-12-dependent host resistance to Toxoplasma gondiiReview series on helminths, immune modulation and the hygiene hypothesis: mechanisms underlying helminth modulation of dendritic cell functionThe Differentiation of CD4(+) T-Helper Cell Subsets in the Context of Helminth Parasite InfectionDifferential regulation of effector- and central-memory responses to Toxoplasma gondii Infection by IL-12 revealed by tracking of Tgd057-specific CD8+ T cells.Communication between Toxoplasma gondii and its host: impact on parasite growth, development, immune evasion, and virulence.MyD88-dependent induction of allergic Th2 responses to intranasal antigen.Immunization with Toxoplasma gondii GRA17 Deletion Mutant Induces Partial Protection and Survival in Challenged Mice.IL-10 production by CD4+ effector T cells: a mechanism for self-regulation.IL-10 production differentially influences the magnitude, quality, and protective capacity of Th1 responses depending on the vaccine platform.Innate control of adaptive immunity via remodeling of lymph node feed arterioleLipopolysaccharide potentiates effector T cell accumulation into nonlymphoid tissues through TRIFCurrent status of interleukin-10 and regulatory T-cells in cancerSignals from OX40 regulate nuclear factor of activated T cells c1 and T cell helper 2 lineage commitment.Interleukin-10 production by Th1 cells requires interleukin-12-induced STAT4 transcription factor and ERK MAP kinase activation by high antigen dose.Innate responses to Toxoplasma gondii in mice and humansUnique Roles of TLR9- and MyD88-Dependent and -Independent Pathways in Adaptive Immune Responses to AAV-Mediated Gene TransferDietary prevention of allergic diseases in infants and small children. Part I: immunologic background and criteria for hypoallergenicity.Turning it on and off: regulation of dendritic cell function in Toxoplasma gondii infection.Th2 response polarization during infection with the helminth parasite Schistosoma mansoni.T helper 2 (Th2) cell differentiation, type 2 innate lymphoid cell (ILC2) development and regulation of interleukin-4 (IL-4) and IL-13 production.The role of IL-27 in the development of T-cell responses during parasitic infections.Notch system in the linkage of innate and adaptive immunity.TAP-1 indirectly regulates CD4+ T cell priming in Toxoplasma gondii infection by controlling NK cell IFN-gamma production.Notch ligand Delta-like 4 regulates disease pathogenesis during respiratory viral infections by modulating Th2 cytokines.A subset of dendritic cells induces CD4+ T cells to produce IFN-gamma by an IL-12-independent but CD70-dependent mechanism in vivo.CD8- DCs induce IL-12-independent Th1 differentiation through Delta 4 Notch-like ligand in response to bacterial LPSSignaling through Fc gamma RIII is required for optimal T helper type (Th)2 responses and Th2-mediated airway inflammation.CD4(+)CD25(-)Foxp3(-) Th1 cells are the source of IL-10-mediated immune suppression in chronic cutaneous leishmaniasis.Conventional T-bet(+)Foxp3(-) Th1 cells are the major source of host-protective regulatory IL-10 during intracellular protozoan infection.Schistosome glycans and innate immunity.Priming of the immune response by schistosome eggs.Establishment of memory for IL-10 expression in developing T helper 2 cells requires repetitive IL-4 costimulation and does not impair proliferation.Interleukin 1alpha promotes Th1 differentiation and inhibits disease progression in Leishmania major-susceptible BALB/c miceNotch regulates IL-10 production by T helper 1 cells.T cell expression of MyD88 is required for resistance to Toxoplasma gondii.Parasite-induced TH1 cells and intestinal dysbiosis cooperate in IFN-γ-dependent elimination of Paneth cellsImmune privilege in sites of chronic infection: Leishmania and regulatory T cells.Cytokine regulation of immunopathology in toxoplasmosis.The contribution of toll-like receptors to the pathogenesis of asthma.Schistosoma mansoni egg antigen-mediated modulation of Toll-like receptor (TLR)-induced activation occurs independently of TLR2, TLR4, and MyD88
P2860
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P2860
In the absence of IL-12, CD4(+) T cell responses to intracellular pathogens fail to default to a Th2 pattern and are host protective in an IL-10(-/-) setting.
description
2002 nî lūn-bûn
@nan
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
@yue
2002年學術文章
@zh-hant
name
In the absence of IL-12, CD4(+ ...... tive in an IL-10(-/-) setting.
@en
In the absence of IL-12, CD4(+ ...... tive in an IL-10(-/-) setting.
@nl
type
label
In the absence of IL-12, CD4(+ ...... tive in an IL-10(-/-) setting.
@en
In the absence of IL-12, CD4(+ ...... tive in an IL-10(-/-) setting.
@nl
prefLabel
In the absence of IL-12, CD4(+ ...... tive in an IL-10(-/-) setting.
@en
In the absence of IL-12, CD4(+ ...... tive in an IL-10(-/-) setting.
@nl
P2093
P1433
P1476
In the absence of IL-12, CD4(+ ...... tive in an IL-10(-/-) setting.
@en
P2093
Carmen M Collazo
Dragana Jankovic
Marika C Kullberg
Patricia Caspar
Sara Hieny
P304
P356
10.1016/S1074-7613(02)00278-9
P407
P577
2002-03-01T00:00:00Z