Differences in the onset of the inflammatory response to cutaneous leishmaniasis in resistant and susceptible mice.
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In vivo involvement of polymorphonuclear neutrophils in Leishmania infantum infectionThe site of the bite: Leishmania interaction with macrophages, neutrophils and the extracellular matrix in the dermisThe influence of early neutrophil-Leishmania interactions on the host immune response to infectionRapid Sequestration of Leishmania mexicana by Neutrophils Contributes to the Development of Chronic LesionCrosstalk between purinergic receptors and lipid mediators in leishmaniasisGene-specific sex effects on eosinophil infiltration in leishmaniasisCC chemokine receptor (CCR)2 is required for langerhans cell migration and localization of T helper cell type 1 (Th1)-inducing dendritic cells. Absence of CCR2 shifts the Leishmania major-resistant phenotype to a susceptible state dominated by Th2 cNeutrophil-derived CCL3 is essential for the rapid recruitment of dendritic cells to the site of Leishmania major inoculation in resistant mice.A secondary wave of neutrophil infiltration causes necrosis and ulceration in lesions of experimental American cutaneous leishmaniasisInnate immunity to Leishmania infection: within phagocytes.Control of Leishmania infantum infection is associated with CD8(+) and gamma interferon- and interleukin-5-producing CD4(+) antigen-specific T cells.Efficient capture of infected neutrophils by dendritic cells in the skin inhibits the early anti-leishmania response.Insulin-like growth factor-I induces arginase activity in Leishmania amazonensis amastigote-infected macrophages through a cytokine-independent mechanism.Protective role of 5-lipoxigenase during Leishmania infantum infection is associated with Th17 subsetDeception and manipulation: the arms of leishmania, a successful parasiteTransgenic expression of CXCR3 on T cells enhances susceptibility to cutaneous Leishmania major infection by inhibiting monocyte maturation and promoting a Th2 response.The levels and patterns of cytokines produced by CD4 T lymphocytes of BALB/c mice infected with Leishmania major by inoculation into the ear dermis depend on the infectiousness and size of the inoculumNeutrophils have a protective role during early stages of Leishmania amazonensis infection in BALB/c mice.Comparative analysis of resistant and susceptible macrophage gene expression response to Leishmania major parasite.IRF-5-Mediated Inflammation Limits CD8+ T Cell Expansion by Inducing HIF-1α and Impairing Dendritic Cell Functions during Leishmania InfectionReactive oxygen species and nitric oxide in cutaneous leishmaniasis.Does T Helper Differentiation Correlate with Resistance or Susceptibility to Infection with L. major? Some Insights From the Murine Model.Characterization of Neutrophil Function in Human Cutaneous Leishmaniasis Caused by Leishmania braziliensisCholesterol Corrects Altered Conformation of MHC-II Protein in Leishmania donovani Infected Macrophages: Implication in Therapy.Metabolic characterization of Leishmania major infection in activated and nonactivated macrophages.The impact of vector-mediated neutrophil recruitment on cutaneous leishmaniasis.The Nlrp3 inflammasome, IL-1β, and neutrophil recruitment are required for susceptibility to a nonhealing strain of Leishmania major in C57BL/6 mice.Modulation of dendritic cell function by Leishmania parasitesIL-17 promotes progression of cutaneous leishmaniasis in susceptible miceMerTK cleavage limits proresolving mediator biosynthesis and exacerbates tissue inflammation.The prominent role of neutrophils during the initial phase of infection by Leishmania parasites.Complement receptor 3 deficiency influences lesion progression during Leishmania major infection in BALB/c miceAccelerated healing of cutaneous leishmaniasis in non-healing BALB/c mice using water soluble amphotericin B-polymethacrylic acid.Permissive and protective roles for neutrophils in leishmaniasis.Arginase I induction during Leishmania major infection mediates the development of diseaseImmune adherence-mediated opsonophagocytosis: the mechanism of Leishmania infection.All-Trans Retinoic Acid Promotes an M1- to M2-Phenotype Shift and Inhibits Macrophage-Mediated Immunity to Leishmania major.
P2860
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P2860
Differences in the onset of the inflammatory response to cutaneous leishmaniasis in resistant and susceptible mice.
description
1992 nî lūn-bûn
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1992年の論文
@ja
1992年学术文章
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1992年学术文章
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1992年学术文章
@zh-cn
1992年学术文章
@zh-hans
1992年学术文章
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1992年学术文章
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1992年學術文章
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1992年學術文章
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name
Differences in the onset of th ...... esistant and susceptible mice.
@en
Differences in the onset of th ...... esistant and susceptible mice.
@nl
type
label
Differences in the onset of th ...... esistant and susceptible mice.
@en
Differences in the onset of th ...... esistant and susceptible mice.
@nl
prefLabel
Differences in the onset of th ...... esistant and susceptible mice.
@en
Differences in the onset of th ...... esistant and susceptible mice.
@nl
P2093
P1476
Differences in the onset of th ...... esistant and susceptible mice.
@en
P2093
Meinardus-Hager G
Neugebauer DC
P304
P577
1992-08-01T00:00:00Z