Dissociation of NEPH1 from nephrin is involved in development of a rat model of focal segmental glomerulosclerosis.
about
Motor protein Myo1c is a podocyte protein that facilitates the transport of slit diaphragm protein Neph1 to the podocyte membraneSolution structure analysis of cytoplasmic domain of podocyte protein Neph1 using small/wide angle x-ray scattering (SWAXS)Dysregulated nephrin in diabetic nephropathy of type 2 diabetes: a cross sectional studyStructural Analysis of the Myo1c and Neph1 Complex Provides Insight into the Intracellular Movement of Neph1Oxidative/Nitrative Stress and Inflammation Drive Progression of Doxorubicin-Induced Renal Fibrosis in Rats as Revealed by Comparing a Normal and a Fibrosis-Resistant Rat Strain.Systematic implantation of dedifferentiated fat cells ameliorated monoclonal antibody 1-22-3-induced glomerulonephritis by immunosuppression with increases in TNF-stimulated gene 6.Role of calcineurin (CN) in kidney glomerular podocyte: CN inhibitor ameliorated proteinuria by inhibiting the redistribution of CN at the slit diaphragm.Inverted formin 2 regulates actin dynamics by antagonizing Rho/diaphanous-related formin signaling.Neph1 regulates steady-state surface expression of Slo1 Ca(2+)-activated K(+) channels: different effects in embryonic neurons and podocytesMyo1c is an unconventional myosin required for zebrafish glomerular development.Slit diaphragm protein Neph1 and its signaling: a novel therapeutic target for protection of podocytes against glomerular injuryFunctional roles for myosin 1c in cellular signaling pathways.Therapeutic target for nephrotic syndrome: Identification of novel slit diaphragm associated molecules.An update: the role of Nephrin inside and outside the kidney.Neurexin-1, a presynaptic adhesion molecule, localizes at the slit diaphragm of the glomerular podocytes in kidneys.Neph1 is reduced in primary focal segmental glomerulosclerosis, minimal change nephrotic syndrome, and corresponding experimental animal models of adriamycin-induced nephropathy and puromycin aminonucleoside nephrosis.Proteinuria in mice expressing PKB/SGK-resistant GSK3.Proinflammatory role of angiotensin II in a rat nephrosis model induced by adriamycin.Nephrin-Binding Ephrin-B1 at the Slit Diaphragm Controls Podocyte Function through the JNK Pathway.Alteration in the podoplanin-ezrin-cytoskeleton linkage is an important initiation event of the podocyte injury in puromycin aminonucleoside nephropathy, a mimic of minimal change nephrotic syndrome.
P2860
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P2860
Dissociation of NEPH1 from nephrin is involved in development of a rat model of focal segmental glomerulosclerosis.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh-hant
name
Dissociation of NEPH1 from nep ...... segmental glomerulosclerosis.
@en
Dissociation of NEPH1 from nep ...... segmental glomerulosclerosis.
@nl
type
label
Dissociation of NEPH1 from nep ...... segmental glomerulosclerosis.
@en
Dissociation of NEPH1 from nep ...... segmental glomerulosclerosis.
@nl
prefLabel
Dissociation of NEPH1 from nep ...... segmental glomerulosclerosis.
@en
Dissociation of NEPH1 from nep ...... segmental glomerulosclerosis.
@nl
P2093
P2860
P1476
Dissociation of NEPH1 from nep ...... segmental glomerulosclerosis.
@en
P2093
Akira Takada
Fujio Shimizu
Fumitake Gejyo
Hiroshi Kawachi
Mutsumi Higa
Naoko Miyauchi
Takeshi Kuroda
Yasuhiro Otaki
P2860
P304
P356
10.1152/AJPRENAL.00075.2008
P577
2008-08-20T00:00:00Z