Activation of p38 MAP kinase enhances sensitivity of muscle glucose transport to insulin. - Wikidata - awgldk - TriplyDB

Activation of p38 MAP kinase enhances sensitivity of muscle glucose transport to insulin.

Activation of p38 MAP kinase enhances sensitivity of muscle glucose transport to insulin.

http://www.wikidata.org/entity/Q45178407

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Exercise-induced cognitive plasticity, implications for mild cognitive impairment and Alzheimer's disease Insulin in central nervous system: more than just a peripheral hormone Regulation of glucose and glycogen metabolism during and after exercise p38 MAPK links oxidative stress to autophagy-related gene expression in cachectic muscle wasting.Lean and obese Zucker rats exhibit different patterns of p70s6 kinase regulation in the tibialis anterior muscle in response to high-force muscle contraction.Exercise and Glycemic Control: Focus on Redox Homeostasis and Redox-Sensitive Protein Signaling.Quercetin, a Lead Compound against Type 2 Diabetes Ameliorates Glucose Uptake via AMPK Pathway in Skeletal Muscle Cell Line.Variants in ASK1 are associated with skeletal muscle ASK1 expression, in vivo insulin resistance, and type 2 diabetes in Pima Indians.Cyclic AMP stimulates Mrp2 translocation by activating p38{alpha} MAPK in hepatic cells.Lipocalin 2 regulates brown fat activation via a nonadrenergic activation mechanism.A role for AMPK in increased insulin action after serum starvation Exercise alters mRNA expression of telomere-repeat binding factor 1 in skeletal muscle via p38 MAPK Activity- and calcineurin-independent nuclear shuttling of NFATc1, but not NFATc3, in adult skeletal muscle fibers.R1467H Variants of Rho Guanine Nucleotide Exchange Factor 11 (ARHGEF11) are Associated with Type 2 Diabetes Mellitus in Koreans.Acute heat treatment improves insulin-stimulated glucose uptake in aged skeletal muscle Improved insulin sensitivity with calorie restriction does not require reduced JNK1/2, p38, or ERK1/2 phosphorylation in skeletal muscle of 9-month-old rats.Exercise-induced increase in muscle insulin sensitivity.TNF-α induces CXCL1 chemokine expression and release in human vascular endothelial cells in vitro via two distinct signaling pathways.Intracellular signalling pathways associated with the glucose-lowering effect of ST36 electroacupuncture in streptozotocin-induced diabetic rats Metabolic reprogramming in dietary restriction.Diabetes alters contraction-induced mitogen activated protein kinase activation in the rat soleus and plantaris Zinc Chelation Mediates the Lysosomal Disruption without Intracellular ROS Generation.Skeletal muscle insulin resistance: roles of fatty acid metabolism and exercise Potential Role of the AMP-activated Protein Kinase in Regulation of Insulin Action.Acute High-Intensity Interval Exercise-Induced Redox Signaling Is Associated with Enhanced Insulin Sensitivity in Obese Middle-Aged Men.HSF1 critically attunes proteotoxic stress sensing by mTORC1 to combat stress and promote growth.Differential effects of p38 MAP kinase inhibitors SB203580 and SB202190 on growth and migration of human MDA-MB-231 cancer cell line.Role of AMP-Activated Protein Kinase for Regulating Post-exercise Insulin Sensitivity.Protein kinases: mechanisms and downstream targets in inflammation-mediated obesity and insulin resistance.Impaired dendritic cell differentiation of CD16-positive monocytes in tuberculosis: role of p38 MAPK.SRC3 expressed in BMSCs promotes growth and migration of multiple myeloma cells by regulating the expression of Cx43.Regulation of the nuclear export of the transcription factor NFATc1 by protein kinases after slow fibre type electrical stimulation of adult mouse skeletal muscle fibres.Association of ARHGEF11 R1467H polymorphism with risk for type 2 diabetes mellitus and insulin resistance in Chinese population.

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P2860

Activation of p38 MAP kinase enhances sensitivity of muscle glucose transport to insulin.

http://www.wikidata.org/entity/Q45178407

description

2004 nî lūn-bûn

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2004年の論文

@ja

2004年学术文章

@wuu

2004年学术文章

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2004年学术文章

@zh-cn

2004年学术文章

@zh-hans

2004年学术文章

@zh-my

2004年学术文章

@zh-sg

2004年學術文章

@yue

2004年學術文章

@zh-hant

name

Activation of p38 MAP kinase enhances sensitivity of muscle glucose transport to insulin.

@en

Activation of p38 MAP kinase enhances sensitivity of muscle glucose transport to insulin.

@nl

type

label

Activation of p38 MAP kinase enhances sensitivity of muscle glucose transport to insulin.

@en

Activation of p38 MAP kinase enhances sensitivity of muscle glucose transport to insulin.

@nl

prefLabel

Activation of p38 MAP kinase enhances sensitivity of muscle glucose transport to insulin.

@en

Activation of p38 MAP kinase enhances sensitivity of muscle glucose transport to insulin.

@nl

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P356

AJPENDO.00477.2004

P1433

American Journal of Physiology - Endocrinology and Metabolism

P1476

Activation of p38 MAP kinase enhances sensitivity of muscle glucose transport to insulin.

@en

P2093

David C Wright

http://www.w3.org/2001/XMLSchema#string

Dong-Ho Han

http://www.w3.org/2001/XMLSchema#string

John O Holloszy

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Paige C Geiger

http://www.w3.org/2001/XMLSchema#string

P2860

The p38 and MK2 kinase cascade phosphorylates tuberin, the tuberous sclerosis 2 gene product, and enhances its interaction with 14-3-3

Characterization of the structure and function of a new mitogen-activated protein kinase (p38beta)

A MAP kinase targeted by endotoxin and hyperosmolarity in mammalian cells

Distribution of the tight junction proteins ZO-1, occludin, and claudin-4, -8, and -12 in bladder epithelium

Effect of contraction on mitogen-activated protein kinase signal transduction in skeletal muscle. Involvement Of the mitogen- and stress-activated protein kinase 1

Stress-induced stimulation of early growth response gene-1 by p38/stress-activated protein kinase 2 is mediated by a cAMP-responsive promoter element in a MAPKAP kinase 2-independent manner

Characterization of the structure and function of the fourth member of p38 group mitogen-activated protein kinases, p38delta

SB 203580 is a specific inhibitor of a MAP kinase homologue which is stimulated by cellular stresses and interleukin-1

A dominant-negative p38 MAPK mutant and novel selective inhibitors of p38 MAPK reduce insulin-stimulated glucose uptake in 3T3-L1 adipocytes without affecting GLUT4 translocation.

A forty-year memoir of research on the regulation of glucose transport into muscle.

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E782-8

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10.1152/AJPENDO.00477.2004

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15585585

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