Prostacyclin attenuates oxidative damage of myocytes by opening mitochondrial ATP-sensitive K+ channels via the EP3 receptor.
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Cardiomyocyte cyclooxygenase-2 influences cardiac rhythm and functionResearch progress of cardioprotective agents for prevention of anthracycline cardiotoxicity.Sphingosine-1-phosphate as a mediator of high-density lipoprotein effects in cardiovascular protectionLY294002 inhibits glucocorticoid-induced COX-2 gene expression in cardiomyocytes through a phosphatidylinositol 3 kinase-independent mechanismArray data extractor (ADE): a LabVIEW program to extract and merge gene array data.Compensatory hypertrophy induced by ventricular cardiomyocyte-specific COX-2 expression in miceProstanoid receptors involved in regulation of the beating rate of neonatal rat cardiomyocytes.Molecular mechanisms in exercise-induced cardioprotectionCardiovascular biology of microsomal prostaglandin E synthase-1.Aging-shifted prostaglandin profile in endothelium as a factor in cardiovascular disordersExercise and the cardiovascular system.Cardiac-generated prostanoids mediate cardiac myocyte apoptosis after myocardial ischaemiaInactivation of the E-prostanoid 3 receptor attenuates the angiotensin II pressor response via decreasing arterial contractility.Prostaglandin E2/cyclooxygenase pathway in human skeletal muscle: influence of muscle fiber type and ageCorticosteroids induce COX-2 expression in cardiomyocytes: role of glucocorticoid receptor and C/EBP-betaNitric oxide, oxidative stress and inflammation in pulmonary arterial hypertension.The action of prostaglandins on ion channels.Protective effect of beraprost sodium, a stable prostacyclin analog, in the development of cigarette smoke extract-induced emphysema.Heart failure-induced activation of phospholipase iPLA2γ generates hydroxyeicosatetraenoic acids opening the mitochondrial permeability transition pore.Roles of cyclooxygenase 2 in sevoflurane- and olprinone-induced early phase of preconditioning and postconditioning against myocardial infarction in rat hearts.
P2860
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P2860
Prostacyclin attenuates oxidative damage of myocytes by opening mitochondrial ATP-sensitive K+ channels via the EP3 receptor.
description
2004 nî lūn-bûn
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2004年の論文
@ja
2004年学术文章
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2004年学术文章
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2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
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2004年學術文章
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2004年學術文章
@zh-hant
name
Prostacyclin attenuates oxidat ...... channels via the EP3 receptor.
@en
Prostacyclin attenuates oxidat ...... channels via the EP3 receptor.
@nl
type
label
Prostacyclin attenuates oxidat ...... channels via the EP3 receptor.
@en
Prostacyclin attenuates oxidat ...... channels via the EP3 receptor.
@nl
prefLabel
Prostacyclin attenuates oxidat ...... channels via the EP3 receptor.
@en
Prostacyclin attenuates oxidat ...... channels via the EP3 receptor.
@nl
P2093
P2860
P1476
Prostacyclin attenuates oxidat ...... channels via the EP3 receptor.
@en
P2093
Hideyuki Ishida
Kayoko Tamaki
Ken Shinmura
Roberto Bolli
Toshiaki Sato
P2860
P304
P356
10.1152/AJPHEART.01003.2004
P577
2004-12-16T00:00:00Z