Chronic ethanol treatment enhances inflammatory mediators and cell death in the brain and in astrocytes.
about
Neuropsychological functioning in opiate-dependent subjects receiving and following methadone maintenance treatmentEthanol-Induced Neurodegeneration and Glial Activation in the Developing BrainNeuroimmune Function and the Consequences of Alcohol ExposureReduced Contextual Discrimination following Alcohol Consumption or MDMA Administration in MiceMechanisms of neuroimmune gene induction in alcoholismAlcoholism and inflammation: neuroimmunology of behavioral and mood disordersJNK pathway activation is controlled by Tao/TAOK3 to modulate ethanol sensitivityChronic ethanol exposure during adolescence in rats induces motor impairments and cerebral cortex damage associated with oxidative stressCell death is associated with reduced base excision repair during chronic alcohol administration in adult rat brainExposure of neonatal rats to alcohol has differential effects on neuroinflammation and neuronal survival in the cerebellum and hippocampus.Prefrontal cortex volumes in adolescents with alcohol use disorders: unique gender effects.The effect of alcohol on the differential expression of cluster of differentiation 14 gene, associated pathways, and genetic networkElevated fatty acid ethyl esters in meconium of sheep fetuses exposed in utero to ethanol--a new animal model.Innate immune factors modulate ethanol interaction with GABAergic transmission in mouse central amygdala.Alcohol and adult hippocampal neurogenesis: promiscuous drug, wanton effectsMolecular and behavioral aspects of the actions of alcohol on the adult and developing brain.Toll-like receptor 4 participates in the myelin disruptions associated with chronic alcohol abuse.Temporal changes in innate immune signals in a rat model of alcohol withdrawal in emotional and cardiorespiratory homeostatic nuclei.Ethanol impairs microtubule formation via interactions at a microtubule associated protein-sensitive siteEffects of ethanol exposure during adolescence or in adulthood on Pavlovian conditioned approach in Sprague-Dawley ratsHIV-1 infection and alcohol abuse: neurocognitive impairment, mechanisms of neurodegeneration and therapeutic interventions.HIV-1 neuropathogenesis: glial mechanisms revealed through substance abuseAlcohol abuse promotes changes in non-synaptic epileptiform activity with concomitant expression changes in cotransporters and glial cells.IL-1 interacts with ethanol effects on GABAergic transmission in the mouse central amygdalaEnhancement of the hypothalamic-pituitary-adrenal axis but not cytokine responses to stress challenges imposed during withdrawal from acute alcohol exposure in Sprague-Dawley ratsMeasurement of serum, liver, and brain cytokine induction, thiamine levels, and hepatopathology in rats exposed to a 4-day alcohol binge protocol.Pre- and postnatal exposure to moderate levels of ethanol can have long-lasting effects on hippocampal glutamate uptake in adolescent offspring.Understanding Alcoholism Through microRNA Signatures in Brains of Human Alcoholics.Inflammasome-IL-1β Signaling Mediates Ethanol Inhibition of Hippocampal NeurogenesisMale adolescent rats display blunted cytokine responses in the CNS after acute ethanol or lipopolysaccharide exposure.Inhibiting the TLR4-MyD88 signalling cascade by genetic or pharmacological strategies reduces acute alcohol-induced sedation and motor impairment in miceChronic Intracerebroventricular Infusion of Monocyte Chemoattractant Protein-1 Leads to a Persistent Increase in Sweetened Ethanol Consumption During Operant Self-Administration But Does Not Influence Sucrose Consumption in Long-Evans Rats.Transgenic mice with increased astrocyte expression of IL-6 show altered effects of acute ethanol on synaptic function.Transcriptional control of maladaptive and protective responses in alcoholics: a role of the NF-κB system.Intoxication- and withdrawal-dependent expression of central and peripheral cytokines following initial ethanol exposure.Brief alcohol exposure alters transcription in astrocytes via the heat shock pathway.Energy Drink Administration in Combination with Alcohol Causes an Inflammatory Response and Oxidative Stress in the Hippocampus and Temporal Cortex of Rats.Recent binge drinking predicts smaller cerebellar volumes in adolescents.Alcohol-induced IL-1β in the brain is mediated by NLRP3/ASC inflammasome activation that amplifies neuroinflammationVolumetric brain abnormalities in polysubstance use disorder patients.
P2860
Q24681779-BA5CF00E-8634-4632-8BB2-94CFA83F86FAQ26740387-33CCCBC8-EC8A-4A4A-B4E3-910EC22AFBB6Q26773207-5148C090-5D94-4FA4-A560-FE6A10456EF1Q27342762-6B8972BF-7584-4441-8566-42DE6F8E03FDQ28085043-1D62FE86-F96C-4B77-824C-B593AA9B8679Q28302013-76421AB3-5951-4746-8C7E-EB01224D606DQ28485496-D1F713FF-AE0C-4F81-86DE-5B629C150900Q28540083-22CBB60E-775F-4635-9CF9-0926FF52916CQ28574437-E84A0256-5936-47EC-8FE1-F336A5C35F1DQ30663525-71E6B76A-A606-482A-8439-A6994D8DC5CDQ33671823-0D71C28D-B19C-4719-9474-27B0C52A7B51Q33757810-B734A585-7D4C-46EA-BBB0-D1BCEBA8E29AQ33922871-B5FA5CC5-B0F9-4922-92A3-D62EC8FC8693Q34020259-DD83EB90-9A64-4963-9E4C-D6D3A25572B8Q34053689-DD063ECD-C982-4876-BE69-1505E1FDE733Q34191211-8FA2DC3B-3E91-4F9B-B58E-6323B0C82426Q34262386-539199C3-38A4-4E55-ACBF-7F70FCF70066Q34280763-3F2DCA3C-09A1-4A0D-8AD9-5B7B39254D2CQ34454047-FA529149-5908-4E12-B9B6-3ADA2DDE1A4AQ34625520-17F7701D-EA4A-4E58-9D99-EBAE0FBA37BBQ34993393-28C5D670-D4D3-49AF-BACD-C7DB63729D5DQ35009465-851EC692-B45B-412F-8150-2D998A6B0A00Q35043611-CFD84DCA-5BD5-4EF9-B864-80974048B3ABQ35194512-46DC1534-7774-44D3-BDBA-4A1CBAADBD0FQ35332116-7C35EC96-B310-4C77-8449-E1E200A194C3Q35361640-28769D36-31E1-4A37-95AA-6A4C0C07FBEEQ35611116-538A3AFA-5FFE-4931-84BC-CA80B8AC306DQ35878186-45469A6A-91F0-40D1-881D-15BCB2CBE22BQ35996491-B3267550-15E5-4005-A4E2-61D077129C48Q36003803-12A29CD4-8F73-4D9B-B400-B6C48CF20177Q36024856-332FFC90-8389-4FCD-93E1-9D5239D01B4AQ36430894-DDA983A7-5E7F-47D5-A066-B8CE70C535F5Q36586181-A0FEDCD6-4530-427F-9459-FA007FDDC87FQ36656436-08C5B5BD-79B8-47AD-AA1B-458C4A84A732Q36692981-069CEE2B-2CF1-483E-8B9B-FA6661891368Q36713124-1D29F9F4-1747-4359-AD1D-8C2F4E6F6BD2Q36741990-4D196974-5719-433D-94A3-F8C4E089C9D3Q36897070-F186F788-E09D-4E11-AF5B-EB7ADC1D0931Q36935742-254B11B2-6517-462F-A94F-F54D00E67ABAQ37016940-827C1FE6-B5FB-418A-ACB0-DF800EA3095D
P2860
Chronic ethanol treatment enhances inflammatory mediators and cell death in the brain and in astrocytes.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh-hant
name
Chronic ethanol treatment enha ...... n the brain and in astrocytes.
@en
Chronic ethanol treatment enha ...... n the brain and in astrocytes.
@nl
type
label
Chronic ethanol treatment enha ...... n the brain and in astrocytes.
@en
Chronic ethanol treatment enha ...... n the brain and in astrocytes.
@nl
prefLabel
Chronic ethanol treatment enha ...... n the brain and in astrocytes.
@en
Chronic ethanol treatment enha ...... n the brain and in astrocytes.
@nl
P2093
P1433
P1476
Chronic ethanol treatment enha ...... n the brain and in astrocytes.
@en
P2093
Ana M Blanco
Consuelo Guerri
María Pascual
Soraya L Vallés
P304
P356
10.1111/J.1750-3639.2004.TB00079.X
P577
2004-10-01T00:00:00Z