Mobilization of sarcoplasmic reticulum stores by hypoxia leads to consequent activation of capacitative Ca2+ entry in isolated canine pulmonary arterial smooth muscle cells.
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Heterogeneous gene expression and functional activity of ryanodine receptors in resistance and conduit pulmonary as well as mesenteric artery smooth muscle cells.Acute oxygen-sensing mechanisms.Prenatal programming of pulmonary hypertension induced by chronic hypoxia or ductal ligation in sheepROS-dependent signaling mechanisms for hypoxic Ca(2+) responses in pulmonary artery myocytes.High altitude pulmonary hypertension: role of K+ and Ca2+ channels.Effect of Yifei Huoxue Granule on the proliferation of rat pulmonary artery smooth muscle cells upon exposure to chronic hypoxic conditions in vitro.Interactions between calcium and reactive oxygen species in pulmonary arterial smooth muscle responses to hypoxia.Hypoxia. 4. Hypoxia and ion channel functionLong-term maternal hypoxia: the role of extracellular Ca2+ entry during serotonin-mediated contractility in fetal ovine pulmonary arteries.Ca2+ responses of pulmonary arterial myocytes to acute hypoxia require release from ryanodine and inositol trisphosphate receptors in sarcoplasmic reticulum.Hypoxic pulmonary vasoconstriction: mechanisms and controversies.Type-3 ryanodine receptors mediate hypoxia-, but not neurotransmitter-induced calcium release and contraction in pulmonary artery smooth muscle cells.AMP-activated protein kinase and the regulation of Ca2+ signalling in O2-sensing cells.AMP-activated protein kinase underpins hypoxic pulmonary vasoconstriction and carotid body excitation by hypoxia in mammals.Effect of chronic perinatal hypoxia on the role of rho-kinase in pulmonary artery contraction in newborn lambs.Differences in STIM1 and TRPC expression in proximal and distal pulmonary arterial smooth muscle are associated with differences in Ca2+ responses to hypoxiaEnhanced capacitative calcium entry and sarcoplasmic-reticulum calcium storage capacity with advanced age in murine mesenteric arterial smooth muscle cells.Caffeine inhibits InsP3 responses and capacitative calcium entry in canine pulmonary arterial smooth muscle cellsMechanisms of oxygen sensing: a key to therapy of pulmonary hypertension and patent ductus arteriosus.Knockdown of stromal interaction molecule 1 attenuates store-operated Ca2+ entry and Ca2+ responses to acute hypoxia in pulmonary arterial smooth muscle.Acute hypoxia activates store-operated Ca(2+) entry and increases intracellular Ca(2+) concentration in rat distal pulmonary venous smooth muscle cellsThe role of protein O-linked beta-N-acetylglucosamine in mediating cardiac stress responses.Role of ASIC1 in the development of chronic hypoxia-induced pulmonary hypertension.Lung Circulation.Protein O-GlcNAcylation: A critical regulator of the cellular response to stress.Hypoxia induces intracellular Ca2+ release by causing reactive oxygen species-mediated dissociation of FK506-binding protein 12.6 from ryanodine receptor 2 in pulmonary artery myocytesRole of ROS signaling in differential hypoxic Ca2+ and contractile responses in pulmonary and systemic vascular smooth muscle cells.Mitochondrial ROS-PKCepsilon signaling axis is uniquely involved in hypoxic increase in [Ca2+]i in pulmonary artery smooth muscle cells.Role of InsP3 and ryanodine receptors in the activation of capacitative Ca2+ entry by store depletion or hypoxia in canine pulmonary arterial smooth muscle cells.A role for 5,6-epoxyeicosatrienoic acid in calcium entry by de novo conformational coupling in human platelets.Hypoxic pulmonary vasoconstriction in the absence of pretone: essential role for intracellular Ca2+ release.TRPC1 and Orai1 interact with STIM1 and mediate capacitative Ca(2+) entry caused by acute hypoxia in mouse pulmonary arterial smooth muscle cells.Inhibition of hypoxic pulmonary vasoconstriction by antagonists of store-operated Ca2+ and nonselective cation channels.Hypoxia stimulates via separate pathways ERK phosphorylation and NF-kappaB activation in skeletal muscle cells in primary culture.Hypoxia divergently regulates production of reactive oxygen species in human pulmonary and coronary artery smooth muscle cells.Superoxide constricts rat pulmonary arteries via Rho-kinase-mediated Ca(2+) sensitization.Hydrogen peroxide is a critical regulator of the hypoxia-induced alterations of store-operated Ca2+ entry into rat pulmonary arterial smooth muscle cells.Heterogeneity of hypoxia-mediated decrease in I(K(V)) and increase in [Ca2+](cyt) in pulmonary artery smooth muscle cells.Cell culture alters Ca2+ entry pathways activated by store-depletion or hypoxia in canine pulmonary arterial smooth muscle cells.Ca2+ signaling in hypoxic pulmonary vasoconstriction: effects of myosin light chain and Rho kinase antagonists.
P2860
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P2860
Mobilization of sarcoplasmic reticulum stores by hypoxia leads to consequent activation of capacitative Ca2+ entry in isolated canine pulmonary arterial smooth muscle cells.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh-hant
name
Mobilization of sarcoplasmic r ...... arterial smooth muscle cells.
@en
Mobilization of sarcoplasmic r ...... arterial smooth muscle cells.
@nl
type
label
Mobilization of sarcoplasmic r ...... arterial smooth muscle cells.
@en
Mobilization of sarcoplasmic r ...... arterial smooth muscle cells.
@nl
prefLabel
Mobilization of sarcoplasmic r ...... arterial smooth muscle cells.
@en
Mobilization of sarcoplasmic r ...... arterial smooth muscle cells.
@nl
P2860
P1476
Mobilization of sarcoplasmic r ...... arterial smooth muscle cells.
@en
P2093
Lih Chyuan Ng
Sean M Wilson
P2860
P304
P356
10.1113/JPHYSIOL.2004.078311
P407
P577
2004-12-21T00:00:00Z