Construction of a rhamnose mutation in Bacillus anthracis affects adherence to macrophages but not virulence in guinea pigs.
about
A structural basis for the allosteric regulation of non-hydrolysing UDP-GlcNAc 2-epimerasesMorphogenesis of the Bacillus anthracis sporeRole of purine biosynthesis in Bacillus anthracis pathogenesis and virulence.Localization and assembly of proteins comprising the outer structures of the Bacillus anthracis spore.Exposure to Bacillus anthracis capsule results in suppression of human monocyte-derived dendritic cellsRoles of macrophages and neutrophils in the early host response to Bacillus anthracis spores in a mouse model of infection.Integrative genomic analysis identifies isoleucine and CodY as regulators of Listeria monocytogenes virulenceA recombinant horseshoe crab plasma lectin recognizes specific pathogen-associated molecular patterns of bacteria through rhamnose.Role played by exosporium glycoproteins in the surface properties of Bacillus cereus spores and in their adhesion to stainless steel.Fully virulent Bacillus anthracis does not require the immunodominant protein BclA for pathogenesis.Microarray analysis of transposon insertion mutations in Bacillus anthracis: global identification of genes required for sporulation and germination.The BclB glycoprotein of Bacillus anthracis is involved in exosporium integrityThe Exosporium Layer of Bacterial Spores: a Connection to the Environment and the Infected HostRecombinant exosporium protein BclA of Bacillus anthracis is effective as a booster for mice primed with suboptimal amounts of protective antigen.Disrupting the luxS quorum sensing gene does not significantly affect Bacillus anthracis virulence in mice or guinea pigs.Anthrose biosynthetic operon of Bacillus anthracisContribution of toxins to the pathogenesis of inhalational anthrax.Glycosylation of BclA Glycoprotein from Bacillus cereus and Bacillus anthracis Exosporium Is Domain-specific.Localization and assembly of the novel exosporium protein BetA of Bacillus anthracis.Bacillus anthracis spores of the bclA mutant exhibit increased adherence to epithelial cells, fibroblasts, and endothelial cells but not to macrophages.Inactivation of Bacillus anthracis spores in murine primary macrophages.Anthrax toxin receptor 2 mediates Bacillus anthracis killing of macrophages following spore challenge.A genetic approach for the identification of exosporium assembly determinants of Bacillus anthracis.Structure of the Bacillus anthracis dTDP-L-rhamnose-biosynthetic enzyme dTDP-4-dehydrorhamnose 3,5-epimerase (RfbC).
P2860
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P2860
Construction of a rhamnose mutation in Bacillus anthracis affects adherence to macrophages but not virulence in guinea pigs.
description
2005 nî lūn-bûn
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2005年の論文
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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name
Construction of a rhamnose mut ...... not virulence in guinea pigs.
@en
Construction of a rhamnose mut ...... not virulence in guinea pigs.
@nl
type
label
Construction of a rhamnose mut ...... not virulence in guinea pigs.
@en
Construction of a rhamnose mut ...... not virulence in guinea pigs.
@nl
prefLabel
Construction of a rhamnose mut ...... not virulence in guinea pigs.
@en
Construction of a rhamnose mut ...... not virulence in guinea pigs.
@nl
P2093
P1476
Construction of a rhamnose mut ...... not virulence in guinea pigs.
@en
P2093
Arthur M Friedlander
Avigdor Shafferman
Christopher K Cote
Itai Mendelson
Joel A Bozue
Lawrence R Phillips
Narayanan Parthasarathy
Patricia F Fellows
P356
10.1016/J.MICPATH.2004.10.001
P577
2005-01-06T00:00:00Z