CD4+ subset expression in murine candidiasis. Th responses correlate directly with genetically determined susceptibility or vaccine-induced resistance.
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How Chemotherapy Increases the Risk of Systemic Candidiasis in Cancer Patients: Current Paradigm and Future DirectionsSpecific Human and Candida Cellular Interactions Lead to Controlled or Persistent Infection Outcomes during Granuloma-Like Formation.Antibody complementarity-determining regions (CDRs): a bridge between adaptive and innate immunity.Candida albicans is phagocytosed, killed, and processed for antigen presentation by human dendritic cells.Defective induction of interleukin-12 in human monocytes by germ-tube forms of Candida albicansCellular and cytokine correlates of mucosal protection in murine model of oral candidiasis.Interleukin 18 restores defective Th1 immunity to Candida albicans in caspase 1-deficient miceNatural killer cells do not play a dominant role in CD4+ subset differentiation in Candida albicans-infected mice.Candida-specific Th1-type responsiveness in mice with experimental vaginal candidiasisRecombinant vaccinia virus coexpressing the F protein of respiratory syncytial virus (RSV) and interleukin-4 (IL-4) does not inhibit the development of RSV-specific memory cytotoxic T lymphocytes, whereas priming is diminished in the presence of higCD4(+)-T-Cell-mediated resistance to systemic murine candidiasis induced by a membrane fraction of Candida albicans.Early resistance of interleukin-10 knockout mice to acute systemic candidiasis.Analysis of vaginal cell populations during experimental vaginal candidiasis.Candida albicans and Candida krusei differentially induce human blood mononuclear cell interleukin-12 and gamma interferon production.Cell adhesion molecule and lymphocyte activation marker expression during experimental vaginal candidiasis.Candida-specific antibodies during experimental vaginal candidiasis in mice.IL-12 and related cytokines: function and regulatory implications in Candida albicans infectionImmunopathogenesis of recurrent vulvovaginal candidiasis.Tolerance to staphylococcal enterotoxin B initiated Th1 cell differentiation in mice infected with Candida albicansA mannoprotein constituent of Candida albicans that elicits different levels of delayed-type hypersensitivity, cytokine production, and anticandidal protection in mice.Role for dendritic cells in immunoregulation during experimental vaginal candidiasisChronic vulvovaginal Candida hypersensitivity: An underrecognized and undertreated disorder by allergistsMice immunized by primary vaginal Candida albicans infection develop acquired vaginal mucosal immunity.Differential susceptibility of yeast and hyphal forms of Candida albicans to proteolytic activity of macrophagesPurification and biochemical characterization of a 65-kilodalton mannoprotein (MP65), a main target of anti-Candida cell-mediated immune responses in humans.CD4+ T-helper-cell responses in mice with low-level Candida albicans infection.Gamma interferon is not essential in host defense against disseminated candidiasis in miceUse of cellular depletion analysis to examine circulation of immune effector function between the vagina and the peripheryCD8+ T cells are activated during the early Th1 and Th2 immune responses in a murine Lyme disease model.Th1 and Th2 cytokines in mice with invasive aspergillosis.Reduced virulence of Candida albicans MKC1 mutants: a role for mitogen-activated protein kinase in pathogenesis.Secretory Aspartyl Proteinases Cause Vaginitis and Can Mediate Vaginitis Caused by Candida albicans in Mice.Concanavalin A protects mice from a lethal inoculation of intragastric Klebsiella pneumoniae and reduces the induced liver damage.Adaptive immune responses to Candida albicans infection.Leishmania promastigotes evade interleukin 12 (IL-12) induction by macrophages and stimulate a broad range of cytokines from CD4+ T cells during initiation of infection.T cell genetic background determines default T helper phenotype development in vitro.T helper phenotype and genetic susceptibility in experimental Lyme diseaseImpaired neutrophil response and CD4+ T helper cell 1 development in interleukin 6-deficient mice infected with Candida albicansEndogenous interleukin 4 is required for development of protective CD4+ T helper type 1 cell responses to Candida albicans.Macrophages in resistance to candidiasis
P2860
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P2860
CD4+ subset expression in murine candidiasis. Th responses correlate directly with genetically determined susceptibility or vaccine-induced resistance.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh-hant
name
CD4+ subset expression in muri ...... or vaccine-induced resistance.
@en
CD4+ subset expression in muri ...... or vaccine-induced resistance.
@nl
type
label
CD4+ subset expression in muri ...... or vaccine-induced resistance.
@en
CD4+ subset expression in muri ...... or vaccine-induced resistance.
@nl
prefLabel
CD4+ subset expression in muri ...... or vaccine-induced resistance.
@en
CD4+ subset expression in muri ...... or vaccine-induced resistance.
@nl
P2093
P1476
CD4+ subset expression in muri ...... or vaccine-induced resistance.
@en
P2093
P304
P407
P577
1993-02-01T00:00:00Z