Bilateral subthalamotomy in Parkinson's disease: initial and long-term response.
about
Human pallidothalamic and cerebellothalamic tracts: anatomical basis for functional stereotactic neurosurgeryThe subthalamic nucleus, oscillations, and conflictDrug-induced dyskinesia in Parkinson's disease. Should success in clinical management be a function of improvement of motor repertoire rather than amplitude of dyskinesia?Basal ganglia activity patterns in parkinsonism and computational modeling of their downstream effectsSubthalamic, not striatal, activity correlates with basal ganglia downstream activity in normal and parkinsonian monkeysWhat happened to posteroventral pallidotomy for Parkinson's disease and dystonia?Towards model-based control of Parkinson's disease.Review of the therapeutic management of Parkinson's disease. Report of a joint task force of the European Federation of Neurological Societies (EFNS) and the Movement Disorder Society-European Section (MDS-ES). Part II: late (complicated) Parkinson'Autonomous initiation and propagation of action potentials in neurons of the subthalamic nucleus.Limiting glutamate transmission in a Vglut2-expressing subpopulation of the subthalamic nucleus is sufficient to cause hyperlocomotionMPTP treatment increases expression of pre-pro-nociceptin/orphanin FQ mRNA in a subset of substantia nigra reticulata neurons.Deep brain stimulation: eye movements reveal anomalous effects of electrode placement and stimulation.On the origin of tremor in Parkinson's diseaseThe subthalamic microlesion story in Parkinson's disease: electrode insertion-related motor improvement with relative cortico-subcortical hypoactivation in fMRIMilestones in research on the pathophysiology of Parkinson's diseasePathophysiology of parkinsonism.Alterations in neuronal activity in basal ganglia-thalamocortical circuits in the parkinsonian state.Deep-Brain Stimulation for Basal Ganglia Disorders.Movement-related discharge in the macaque globus pallidus during high-frequency stimulation of the subthalamic nucleusLesion of the centromedian thalamic nucleus in MPTP-treated monkeysParkinson's disease therapeutics: new developments and challenges since the introduction of levodopa.Resting-state functional magnetic resonance imaging of the subthalamic microlesion and stimulation effects in Parkinson's disease: Indications of a principal role of the brainstemInhibition of 5-HT neuron activity and induction of depressive-like behavior by high-frequency stimulation of the subthalamic nucleusDyskinesias induced by subthalamotomy in Parkinson's disease are unresponsive to amantadine.Allosteric modulation of the group III mGlu4 receptor provides functional neuroprotection in the 6-hydroxydopamine rat model of Parkinson's diseasePallidal burst activity during therapeutic deep brain stimulation.Deep brain stimulation: indications and evidence.The history and future of deep brain stimulationFoxa1 is essential for development and functional integrity of the subthalamic nucleus.The posterior subthalamic area in the treatment of movement disorders: past, present, and future.The role of deep brain stimulation in Parkinson's disease: an overview and update on new developments.Current applications and limitations of surgical treatments for movement disorders.Impact of parameters of radiofrequency coagulation on volume of stereotactic lesion in pallidotomy and thalamotomy.Antiparkinsonian potential of targeting group III metabotropic glutamate receptor subtypes in the rodent substantia nigra pars reticulata.Symptomatic and neuroprotective effects following activation of nigral group III metabotropic glutamate receptors in rodent models of Parkinson's diseaseBasal ganglia, movement disorders and deep brain stimulation: advances made through non-human primate research.Neuronal discharge patterns in the globus pallidus pars interna in a patient with Parkinson's disease and hemiballismus secondary to subthalamotomy.Sub-synaptic localization of Cav3.1 T-type calcium channels in the thalamus of normal and parkinsonian monkeys.Microglia may compensate for dopaminergic neuron loss in experimental Parkinsonism through selective elimination of glutamatergic synapses from the subthalamic nucleus.Involvement of the subthalamic nucleus in impulse control disorders associated with Parkinson's disease.
P2860
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P2860
Bilateral subthalamotomy in Parkinson's disease: initial and long-term response.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Bilateral subthalamotomy in Parkinson's disease: initial and long-term response.
@en
Bilateral subthalamotomy in Parkinson's disease: initial and long-term response.
@nl
type
label
Bilateral subthalamotomy in Parkinson's disease: initial and long-term response.
@en
Bilateral subthalamotomy in Parkinson's disease: initial and long-term response.
@nl
prefLabel
Bilateral subthalamotomy in Parkinson's disease: initial and long-term response.
@en
Bilateral subthalamotomy in Parkinson's disease: initial and long-term response.
@nl
P2093
P50
P356
P1433
P1476
Bilateral subthalamotomy in Parkinson's disease: initial and long-term response
@en
P2093
Maragoto C
P304
P356
10.1093/BRAIN/AWH397
P407
P577
2005-02-02T00:00:00Z