Enhanced oxidative stress in iNOS-deficient mice after traumatic brain injury: support for a neuroprotective role of iNOS. - Wikidata - awgldk - TriplyDB

Enhanced oxidative stress in iNOS-deficient mice after traumatic brain injury: support for a neuroprotective role of iNOS.

Enhanced oxidative stress in iNOS-deficient mice after traumatic brain injury: support for a neuroprotective role of iNOS.

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Cathepsin B is a New Drug Target for Traumatic Brain Injury Therapeutics: Evidence for E64d as a Promising Lead Drug Candidate Tissue damage and oxidant/antioxidant balance Combination therapies for neurobehavioral and cognitive recovery after experimental traumatic brain injury: Is more better?Oxidative stress and compartment of gene expression determine proatherosclerotic effects of inducible nitric oxide synthase.A new chiral pyrrolyl α-nitronyl nitroxide radical attenuates β-amyloid deposition and rescues memory deficits in a mouse model of Alzheimer disease Magnetic resonance imaging assessment of macrophage accumulation in mouse brain after experimental traumatic brain injury Antioxidant therapies in traumatic brain and spinal cord injury Hypothermia decreases cerebrospinal fluid asymmetric dimethylarginine levels in children with traumatic brain injury.Adenosine A1 receptor activation as a brake on the microglial response after experimental traumatic brain injury in mice Protective vascular and cardiac effects of inducible nitric oxide synthase in mice with hyperhomocysteinemia.Attenuation of brain nitrostative and oxidative damage by brain cooling during experimental traumatic brain injury.Cannabinoid receptor type-2 stimulation, blockade, and deletion alter the vascular inflammatory responses to traumatic brain injury.Design and Synthesis of a Mitochondria-Targeted Mimic of Glutathione Peroxidase, MitoEbselen-2, as a Radiation Mitigator.Scutellarin regulates microglia-mediated TNC1 astrocytic reaction and astrogliosis in cerebral ischemia in the adult rats Relationship of nitric oxide synthase induction to peroxynitrite-mediated oxidative damage during the first week after experimental traumatic brain injury Electromagnetic controlled cortical impact device for precise, graded experimental traumatic brain injury Postischemic hyperoxia reduces hippocampal pyruvate dehydrogenase activity Drug targets for traumatic brain injury from poly(ADP-ribose)polymerase pathway modulation.PARP-1 inhibition attenuates neuronal loss, microglia activation and neurological deficits after traumatic brain injury.Omega-3 Fatty acids could alleviate the risks of traumatic brain injury - a mini review.Free radicals and antioxidants at a glance using EPR spectroscopy.Intraoperative Targeted Temperature Management in Acute Brain and Spinal Cord Injury.Perspectives on molecular biomarkers of oxidative stress and antioxidant strategies in traumatic brain injury.Role of endothelial nitric oxide synthase-derived nitric oxide in activation and dysfunction of cerebrovascular endothelial cells during early onsets of sepsis.The NADPH oxidase NOX2 mediates loss of parvalbumin interneurons in traumatic brain injury: human autoptic immunohistochemical evidence Intracarotid Sodium Nitroprusside on Fifth Post Ischemic Stroke Day in Middle Cerebral Artery Occlusion Rat Model.Progression of amyloid pathology to Alzheimer's disease pathology in an amyloid precursor protein transgenic mouse model by removal of nitric oxide synthase 2 Correction to "Design and Synthesis of a Mitochondria-Targeted Mimic of Glutathione Peroxidase, MitoEbselen-2, as a Radiation Mitigator"L-Arginine decreases fluid-percussion injury-induced neuronal nitrotyrosine immunoreactivity in rats.Adenosine A1 receptor knockout mice develop lethal status epilepticus after experimental traumatic brain injury.Reduced neuronal injury after treatment with NG-nitro-L-arginine methyl ester (L-NAME) or 2-sulfo-phenyl-N-tert-butyl nitrone (S-PBN) following experimental brain contusion.The effect of thiamine pyrophosphate on ethambutol-induced ocular toxicity.Thyroid Hormone and the Brain: Mechanisms of Action in Development and Role in Protection and Promotion of Recovery after Brain Injury.Characterization of traumatic brain injury in human brains reveals distinct cellular and molecular changes in contusion and pericontusion.Inflammatory cell infiltration after endothelin-1-induced cerebral ischemia: histochemical and myeloperoxidase correlation with temporal changes in brain injury.iNOS-mediated secondary inflammatory response differs between rat strains following experimental brain contusion.L-N-iminoethyl-lysine after experimental brain trauma attenuates cellular proliferation and astrocyte differentiation.Pioglitazone Therapy and Fractures: Systematic Review and Meta-Analysis.Neutrophils in traumatic brain injury (TBI): friend or foe?The Role of Gaseous Molecules in Traumatic Brain Injury: An Updated Review.

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Enhanced oxidative stress in iNOS-deficient mice after traumatic brain injury: support for a neuroprotective role of iNOS.

http://www.wikidata.org/entity/Q45270337

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Journal of Cerebral Blood Flow & Metabolism

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Enhanced oxidative stress in i ...... neuroprotective role of iNOS.

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Deborah L Williams

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Grigory G Borisenko

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Hülya Bayir

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Keri L Janesko

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Timothy R Billiar

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Valerian E Kagan

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Vincent A Vagni

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Yulia Y Tyurina

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P2860

Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide

Accelerated s-nitrosothiol breakdown by amyotrophic lateral sclerosis mutant copper,zinc-superoxide dismutase.

S-Nitrosylation of mitochondrial caspases

Proteomic identification of nitrated proteins in Alzheimer's disease brain.

Oxidative post-translational modifications of alpha-synuclein in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) mouse model of Parkinson's disease.

Role of inducible nitric oxide synthase and cyclooxygenase-2 in endotoxin-induced cerebral hyperemia.

Inducible nitric oxide synthase is an endogenous neuroprotectant after traumatic brain injury in rats and mice

Ascorbate is an outstanding antioxidant in human blood plasma.

Tyrosine nitration: localisation, quantification, consequences for protein function and signal transduction.

Assessment of antioxidant reserves and oxidative stress in cerebrospinal fluid after severe traumatic brain injury in infants and children.

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6

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Patrick M. Kochanek

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8016106

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Traumatic stress

traumatic brain injury