Calcium transients in developing mouse skeletal muscle fibres.
about
Novel role of calpain-3 in the triad-associated protein complex regulating calcium release in skeletal muscleOrai1-dependent calcium entry promotes skeletal muscle growth and limits fatiguePrimary Murine Myotubes as a Model for Investigating Muscular DystrophyPhysiological and ultrastructural features of human induced pluripotent and embryonic stem cell-derived skeletal myocytes in vitroGreen tea catechins are potent sensitizers of ryanodine receptor type 1 (RyR1).IP(3)-dependent, post-tetanic calcium transients induced by electrostimulation of adult skeletal muscle fibersCalcium indicators and calcium signalling in skeletal muscle fibres during excitation-contraction coupling.Muscle weakness in Ryr1I4895T/WT knock-in mice as a result of reduced ryanodine receptor Ca2+ ion permeation and release from the sarcoplasmic reticulum.Non-proteolytic functions of calpain-3 in sarcoplasmic reticulum in skeletal muscles.Cholesterol removal from adult skeletal muscle impairs excitation-contraction coupling and aging reduces caveolin-3 and alters the expression of other triadic proteins.Intracellular calcium movements during excitation-contraction coupling in mammalian slow-twitch and fast-twitch muscle fibersGenetic deletion of trkB.T1 increases neuromuscular function.Structural and functional evaluation of branched myofibers lacking intermediate filamentsAltered contractility of skeletal muscle in mice deficient in titin's M-band region.Simulation of Ca2+ movements within the sarcomere of fast-twitch mouse fibers stimulated by action potentials.Sarcoplasmic reticulum calcium uptake and speed of relaxation are depressed in nebulin-free skeletal muscleMalformed mdx myofibers have normal cytoskeletal architecture yet altered EC coupling and stress-induced Ca2+ signaling.Low-affinity Ca2+ indicators compared in measurements of skeletal muscle Ca2+ transients.Analysis of embryonic and larval zebrafish skeletal myofibers from dissociated preparations.Muscle weakness in myotonic dystrophy associated with misregulated splicing and altered gating of Ca(V)1.1 calcium channel.Disruption of action potential and calcium signaling properties in malformed myofibers from dystrophin-deficient mice.Amyloid-β protein impairs Ca2+ release and contractility in skeletal muscle.Doublet stimulation increases Ca2+ binding to troponin C to ensure rapid force development in skeletal muscle.Myosin heavy chain isoform composition and Ca(2+) transients in fibres from enzymatically dissociated murine soleus and extensor digitorum longus muscles.
P2860
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P2860
Calcium transients in developing mouse skeletal muscle fibres.
description
2005 nî lūn-bûn
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name
Calcium transients in developing mouse skeletal muscle fibres.
@en
Calcium transients in developing mouse skeletal muscle fibres.
@nl
type
label
Calcium transients in developing mouse skeletal muscle fibres.
@en
Calcium transients in developing mouse skeletal muscle fibres.
@nl
prefLabel
Calcium transients in developing mouse skeletal muscle fibres.
@en
Calcium transients in developing mouse skeletal muscle fibres.
@nl
P2093
P2860
P1476
Calcium transients in developing mouse skeletal muscle fibres.
@en
P2093
Carlo Caputo
Joana Capote
Pura Bolaños
Ralph Peter Schuhmeier
Werner Melzer
P2860
P304
P356
10.1113/JPHYSIOL.2004.081034
P407
P577
2005-02-24T00:00:00Z