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Metabolism of vitamin K
Metabolism of vitamin K
Item
http://www.wikidata.org/entity/Q45317420
Q45317420
about
UBIA1D prenylates menadione to form MK4 (vitamin K hydroquinone)
VKORC1 reduces vitamin K epoxide to MK4 (vitamin K hydroquinone)
VKORC1L1 reduces vitamin K epoxide to MK4 (vitamin K hydroquinone)
VKORC1 inhibitors binds VKORC1 dimer
P361
Metabolism of fat-soluble vitamins
P527
Q50294867-3C6E0878-F6C7-4136-BCBB-225ADDD27778
Q50294868-51416569-B2C6-4B11-A51F-AEBC2A57FBA2
Q50294870-30F13FCA-58BB-4852-A906-9AB097578859
Q56860028-AFF335BC-5A85-446A-9F14-BC1C48ED0DAE
P361
Q45317419-0FF71A32-9995-4AE5-8E30-0484B1AFBB4A
P527
Metabolism of vitamin K
Item
http://www.wikidata.org/entity/Q45317420
description
An instance of the biological pathway Metabolism of vitamin K in Homo sapiens
@en
name
Metabolism of vitamin K
@en
type
Item
label
Metabolism of vitamin K
@en
prefLabel
Metabolism of vitamin K
@en
P2860
Q45317420-117204DB-E359-403E-8C8A-AA0B811BDF47
Q45317420-35898C14-44E7-43DC-89FB-EEC9F7044335
Q45317420-4AC8B95E-D39A-404A-B846-487077B5E721
Q45317420-BE5368DF-05BF-4744-90CB-AA3502DFE148
Q45317420-BFB34075-4568-4A5A-B127-53890CAF2DB2
Q45317420-D8119DC2-804D-41BC-8F2F-42750DBD4565
Q45317420-E5D1CED2-4B93-4221-B279-32BB565BE3DD
P2888
Q45317420-35B17AF7-9B6E-4560-9B7C-892C99AD007B
P31
Q45317420-232F265B-197C-459F-AE38-2DB6ADB748ED
Q45317420-BD805FAA-0845-4EAF-8FDE-CBBB4FA3B533
P361
Q45317420-32934F28-B9A0-4724-8D7B-6003335BE89F
P3937
Q45317420-5ACA41BB-F12A-4DC7-A0E6-C5332A0B400F
P527
Q45317420-278E0D97-7DD2-4FCA-81B9-553BFC062BEE
Q45317420-441BE7F6-A8D8-4FC5-A66B-28B2D99BFD36
Q45317420-5B23F493-5FE4-4EFB-A4B0-8EFC57744158
Q45317420-7C98F08F-A7EA-41B8-9C31-C3DDF8AC1FD5
P703
Q45317420-E58E7DC8-A9D6-4640-B2D3-8DC7400F3671
P2860
A missense mutation in gamma-glutamyl carboxylase gene causes combined deficiency of all vitamin K-dependent blood coagulation factors
Conserved loop cysteines of vitamin K epoxide reductase complex subunit 1-like 1 (VKORC1L1) are involved in its active site regeneration
VKORC1L1, an enzyme rescuing the vitamin K 2,3-epoxide reductase activity in some extrahepatic tissues during anticoagulation therapy
GGCX and VKORC1 inhibit osteocalcin endocrine functions.
Vitamin K nutrition, metabolism, and requirements: current concepts and future research.
Menadione (vitamin K3) is a catabolic product of oral phylloquinone (vitamin K1) in the intestine and a circulating precursor of tissue menaquinone-4 (vitamin K2) in rats.
Recent trends in the metabolism and cell biology of vitamin K with special reference to vitamin K cycling and MK-4 biosynthesis.
P2888
reactome:R-HSA-6806664
P31
vitamin K metabolic process
biological pathway
P361
Metabolism of fat-soluble vitamins
P3937
R-HSA-6806664
http://www.w3.org/2001/XMLSchema#string
P527
UBIA1D prenylates menadione to form MK4 (vitamin K hydroquinone)
VKORC1 reduces vitamin K epoxide to MK4 (vitamin K hydroquinone)
VKORC1L1 reduces vitamin K epoxide to MK4 (vitamin K hydroquinone)
VKORC1 inhibitors binds VKORC1 dimer
P703
Homo sapiens