Developmental exposure to the potent aryl hydrocarbon receptor agonist 2,3,7,8-tetrachlorodibenzo-p-dioxin Impairs the cell-mediated immune response to infection with influenza a virus, but enhances elements of innate immunity.
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Fetal Hematopoietic Stem Cells Are the Canaries in the Coal Mine That Portend Later Life Immune DeficiencyPerinatal immunotoxicity: why adult exposure assessment fails to predict riskAryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosisA Birth Cohort Study of Maternal and Infant Serum PCB-153 and DDE Concentrations and Responses to Infant Tuberculosis VaccinationArylhydrocarbon receptor activation in NCI-H441 cells and C57BL/6 mice: possible mechanisms for lung dysfunctionA cohort study of developmental polychlorinated biphenyl (PCB) exposure in relation to post-vaccination antibody response at 6-months of ageDevelopmental exposure to 2,3,7,8 tetrachlorodibenzo-p-dioxin attenuates capacity of hematopoietic stem cells to undergo lymphocyte differentiation.Aryl hydrocarbon receptor targets pathways extrinsic to bone marrow cells to enhance neutrophil recruitment during influenza virus infectionThe aryl hydrocarbon receptor affects distinct tissue compartments during ontogeny of the immune system.Environmental toxicants and the developing immune system: a missing link in the global battle against infectious disease?Effects of developmental activation of the AhR on CD4+ T-cell responses to influenza virus infection in adult mice.Developmental exposure to 2,3,7,8 tetrachlorodibenzo-p-dioxin attenuates later-life Notch1-mediated T cell development and leukemogenesis.Linking the aryl hydrocarbon receptor with altered DNA methylation patterns and developmentally induced aberrant antiviral CD8+ T cell responses.Activation of the aryl hydrocarbon receptor during development enhances the pulmonary CD4+ T-cell response to viral infection.Developmental Activation of the AHR Increases Effector CD4+ T Cells and Exacerbates Symptoms in Autoimmune Disease-Prone Gnaq+/- Mice.Immunomodulation By Subchronic Low Dose 2,3,7,8-Tetrachlorodibenzo-p-Dioxin in Experimental Autoimmune Encephalomyelitis in the Absence of Pertussis Toxin.Immune cell counts and risks of respiratory infections among infants exposed pre- and postnatally to organochlorine compounds: a prospective study.New developments in the assessment of developmental immunotoxicology.Relative effect potency estimates of dioxin-like activity for dioxins, furans, and dioxin-like PCBs in adults based on cytochrome P450 1A1 and 1B1 gene expression in blood.A dose-response study of the effects of prenatal and lactational exposure to TCDD on the immune response to influenza a virus.
P2860
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P2860
Developmental exposure to the potent aryl hydrocarbon receptor agonist 2,3,7,8-tetrachlorodibenzo-p-dioxin Impairs the cell-mediated immune response to infection with influenza a virus, but enhances elements of innate immunity.
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2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
@zh
2004年学术文章
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2004年学术文章
@zh-hans
2004年学术文章
@zh-my
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2004年學術文章
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name
Developmental exposure to the ...... s elements of innate immunity.
@en
Developmental exposure to the ...... s elements of innate immunity.
@nl
type
label
Developmental exposure to the ...... s elements of innate immunity.
@en
Developmental exposure to the ...... s elements of innate immunity.
@nl
prefLabel
Developmental exposure to the ...... s elements of innate immunity.
@en
Developmental exposure to the ...... s elements of innate immunity.
@nl
P2093
P2860
P1476
Developmental exposure to the ...... s elements of innate immunity.
@en
P2093
B Paige Lawrence
Beth A Vorderstrasse
Jennifer A Cundiff
P2860
P304
P356
10.1080/15476910490509244
P577
2004-04-01T00:00:00Z