Hypoxia can contribute to the induction of the Epstein-Barr virus (EBV) lytic cycle.
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The roles of viruses in brain tumor initiation and oncomodulationRegulation of the latent-lytic switch in Epstein-Barr virusEpstein-Barr Virus: Diseases Linked to Infection and TransformationEpstein-Barr virus lytic reactivation regulation and its pathogenic role in carcinogenesisHypoxia inducible factor-1 alpha activation of the JCV promoter: role in the pathogenesis of progressive multifocal leukoencephalopathyArsenic trioxide inhibits EBV reactivation and promotes cell death in EBV-positive lymphoma cells.Hypoxia-inducible factor-1α plays roles in Epstein-Barr virus's natural life cycle and tumorigenesis by inducing lytic infection through direct binding to the immediate-early BZLF1 gene promoter.Hypoxia-Inducible Factor (HIF) as a Pharmacological Target for Prevention and Treatment of Infectious DiseasesEpstein-Barr Virus MicroRNA miR-BART20-5p Suppresses Lytic Induction by Inhibiting BAD-Mediated caspase-3-Dependent Apoptosis.Role of cellular iron and oxygen in the regulation of HIV-1 infection.Contribution of myocyte enhancer factor 2 family transcription factors to BZLF1 expression in Epstein-Barr virus reactivation from latency.Regulation of HIV-1 transcription at 3% versus 21% oxygen concentrationEpstein-Barr virus utilizes Ikaros in regulating its latent-lytic switch in B cells.Switching of EBV cycles between latent and lytic states.Regulation of Epstein-Barr virus reactivation from latency.Chlorpyrifos Induces the Expression of the Epstein-Barr Virus Lytic Cycle Activator BZLF-1 via Reactive Oxygen Species.Epigallocatechin-3-gallate inhibits IGF-I-stimulated lung cancer angiogenesis through downregulation of HIF-1α and VEGF expression.An epidemiological and molecular study of the relationship between smoking, risk of nasopharyngeal carcinoma, and Epstein-Barr virus activation.Hypoxia induces the gene expression and extracellular transmission of persistent lymphocytic choriomeningitis virus.Hypoxia-targeting by tirapazamine (TPZ) induces preferential growth inhibition of nasopharyngeal carcinoma cells with Chk1/2 activation.X-box binding protein 1 contributes to induction of the Kaposi's sarcoma-associated herpesvirus lytic cycle under hypoxic conditions.Lactic Acid Downregulates Viral MicroRNA To Promote Epstein-Barr Virus-Immortalized B Lymphoblastic Cell Adhesion and Growth.
P2860
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P2860
Hypoxia can contribute to the induction of the Epstein-Barr virus (EBV) lytic cycle.
description
2006 nî lūn-bûn
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2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
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2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
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2006年學術文章
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name
Hypoxia can contribute to the induction of the Epstein-Barr virus (EBV) lytic cycle.
@en
Hypoxia can contribute to the induction of the Epstein-Barr virus
@nl
type
label
Hypoxia can contribute to the induction of the Epstein-Barr virus (EBV) lytic cycle.
@en
Hypoxia can contribute to the induction of the Epstein-Barr virus
@nl
prefLabel
Hypoxia can contribute to the induction of the Epstein-Barr virus (EBV) lytic cycle.
@en
Hypoxia can contribute to the induction of the Epstein-Barr virus
@nl
P2093
P1476
Hypoxia can contribute to the induction of the Epstein-Barr virus (EBV) lytic cycle.
@en
P2093
Da-Jiang Li
Jian-Ming Wen
Ju-Hong Jiang
Mu-Sheng Zeng
Shi-Juan Mai
Wen-Ting Liao
Yi-Xin Zeng
Zhi-Gang Pan
P304
P356
10.1016/J.JCV.2006.06.013
P577
2006-08-22T00:00:00Z