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Involvement of NADPH oxidase in A2A adenosine receptor-mediated increase in coronary flow in isolated mouse heartsMechanisms underlying uridine adenosine tetraphosphate-induced vascular contraction in mouse aorta: Role of thromboxane and purinergic receptorsEnhanced A2A adenosine receptor-mediated increase in coronary flow in type I diabetic mice.Phosphodiesterase-5 activity exerts a coronary vasoconstrictor influence in awake swine that is mediated in part via an increase in endothelin production.Pulmonary vasoconstrictor influence of endothelin in exercising swine depends critically on phosphodiesterase 5 activity.Altered purinergic signaling in uridine adenosine tetraphosphate-induced coronary relaxation in swine with metabolic derangement.Disruption of CD38 gene enhances cardiac functions by elevating serum testosterone in the male null mice.Uridine adenosine tetraphosphate is a novel vasodilator in the coronary microcirculation which acts through purinergic P1 but not P2 receptors.Impaired Aortic Contractility to Uridine Adenosine Tetraphosphate in Angiotensin II-Induced Hypertensive Mice: Receptor Desensitization?Divergent coronary flow responses to uridine adenosine tetraphosphate in atherosclerotic ApoE knockout mice.Phosphodiesterase 5 inhibition-induced coronary vasodilation is reduced after myocardial infarction.Uridine adenosine tetraphosphate acts as a proangiogenic factor in vitro through purinergic P2Y receptors.Coronary microvascular dysfunction after long-term diabetes and hypercholesterolemia.Uridine Adenosine Tetraphosphate-Induced Coronary Relaxation Is Blunted in Swine With Pressure Overload: A Role for Vasoconstrictor Prostanoids.Blunted coronary vasodilator response to uridine adenosine tetraphosphate in post-infarct remodeled myocardium is due to reduced P1 receptor activationCytochrome P-450 2C9 exerts a vasoconstrictor influence on coronary resistance vessels in swine at rest and during exerciseSunitinib-Induced Systemic Vasoconstriction in Swine Is Endothelin Mediated and Does Not Involve Nitric Oxide or Oxidative StressAltered Purinergic Receptor Sensitivity in Type 2 Diabetes-Associated Endothelial Dysfunction and Up₄A-Mediated Vascular ContractionUridine adenosine tetraphosphate and purinergic signaling in cardiovascular system: An updateErythrocytes From Patients With Type 2 Diabetes Induce Endothelial Dysfunction Via Arginase IRed Blood Cells in Type 2 Diabetes Impair Cardiac Post-Ischemic Recovery Through an Arginase-Dependent Modulation of Nitric Oxide Synthase and Reactive Oxygen SpeciesRed blood cell dysfunction: a new player in cardiovascular disease
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Q35587846-CDE54218-E978-4602-B219-3E0560278B2BQ36083437-37855CCB-4073-4A66-80EC-1C45849ACFE7Q36510139-B95388B9-CEA9-414B-956E-EB9E52B9FDEBQ37629227-7D4F8C95-2E23-454B-ACB6-7A9194993E68Q37629243-E1D15988-6F5E-438D-B402-8F65D0105127Q38679517-A4F64203-358A-46AD-9DA3-A42C7A9C0809Q42727603-E4B1EF5C-E4DE-4EC6-876C-F86F9D0D7216Q43500185-E2BC5382-48AF-47A1-A7C4-748BD85E8D5DQ45049857-61F25CDC-3970-4627-8308-016296833069Q45067797-80A0C26D-468E-4A24-AA90-32D701DC6FBCQ45423900-F3A61D95-CE64-4431-9265-C9C8F2FA300FQ48220205-6655EDED-BDD4-4819-87EA-9EA9ADFF707FQ48371916-E9E32490-EC5E-4C3A-9A17-B3320951AEC8Q55084123-3A56160E-4B59-47D5-857D-3461F1182D0EQ57816379-ED1EC498-6064-4C74-BFEF-5181BF6C871CQ57816396-23CE1E98-941A-44B8-923D-54ED8F7D7270Q57816403-6D9CD9E6-24E1-442F-BA6D-83A1E083B239Q60909629-8F68F6D6-6B7E-4612-A9D1-84F87222A130Q90556980-32D1933D-DE21-4282-B3E7-85286A6B5C7CQ90857259-BBD71B30-DDFE-4E58-9450-AA58B38D10DBQ91268229-8DEABE2F-DA83-4743-A100-244CCC696133Q92743885-D32BEA65-0EEC-4358-B1DA-4C99CF40A9BC
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description
researcher
@en
հետազոտող
@hy
name
Zhichao Zhou
@ast
Zhichao Zhou
@en
Zhichao Zhou
@es
Zhichao Zhou
@nl
Zhichao Zhou
@sl
type
label
Zhichao Zhou
@ast
Zhichao Zhou
@en
Zhichao Zhou
@es
Zhichao Zhou
@nl
Zhichao Zhou
@sl
prefLabel
Zhichao Zhou
@ast
Zhichao Zhou
@en
Zhichao Zhou
@es
Zhichao Zhou
@nl
Zhichao Zhou
@sl
P106
P31
P496
0000-0002-5107-6529