about
Midkine-a protein localization in the developing and adult retina of the zebrafish and its function during photoreceptor regenerationDifferential phosphoproteome of the striatum from pleiotrophin knockout and midkine knockout mice treated with amphetamine: correlations with amphetamine-induced neurotoxicity.Phosphoproteomic analysis of the striatum from pleiotrophin knockout and midkine knockout mice treated with cocaine reveals regulation of oxidative stress-related proteins potentially underlying cocaine-induced neurotoxicity and neurodegeneration.Targeting the pleiotrophin/receptor protein tyrosine phosphatase beta/zeta signaling pathway to limit neurotoxicity induced by drug abuse.The expression and function of midkine in the vertebrate retina.Connecting Parkinson's disease and drug addiction: common players reveal unexpected disease connections and novel therapeutic approaches.Pleiotrophin modulates morphine withdrawal but has no effects on morphine-conditioned place preference.Functional neuroimaging of amphetamine-induced striatal neurotoxicity in the pleiotrophin knockout mouse model.Genetic inactivation of pleiotrophin triggers amphetamine-induced cell loss in the substantia nigra and enhances amphetamine neurotoxicity in the striatum.Genetic deletion of pleiotrophin leads to disruption of spinal nociceptive transmission: evidence for pleiotrophin modulation of morphine-induced analgesia.Pleiotrophin inhibits hippocampal long-term potentiation: a role of pleiotrophin in learning and memory.Regulation of extinction of cocaine-induced place preference by midkine is related to a differential phosphorylation of peroxiredoxin 6 in dorsal striatum.Pleiotrophin prevents cocaine-induced toxicity in vitro.The heparin binding growth factors midkine and pleiotrophin regulate the antinociceptive effects of morphine through α(2)-adrenergic independent mechanisms.Comparative study of alpha2-adrenoceptors in Fischer 344 and Lewis rats. Evidence for clonidine-induced place aversion.Development of inhibitors of receptor protein tyrosine phosphatase β/ζ (PTPRZ1) as candidates for CNS disorders.Genetic inactivation of pleiotrophin but not midkine potentiates clonidine-induced alpha-2 adrenergic-mediated analgesia.Midkine regulates amphetamine-induced astrocytosis in striatum but has no effects on amphetamine-induced striatal dopaminergic denervation and addictive effects: functional differences between pleiotrophin and midkine.
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description
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հետազոտող
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name
Esther Gramage
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Esther Gramage
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Esther Gramage
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Esther Gramage
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type
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Esther Gramage
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Esther Gramage
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Esther Gramage
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Esther Gramage
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P1053
K-6441-2014
P106
P1153
24172719300
P21
P31
P3829
P3835
esther-gramage
P496
0000-0002-5134-1273