OM-101 Decreases the Fibrotic Response Associated with Proliferative Vitreoretinopathy.

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OM-101 Decreases the Fibrotic Response Associated with Proliferative Vitreoretinopathy.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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OM-101 Decreases the Fibrotic Response Associated with Proliferative Vitreoretinopathy.

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OM-101 Decreases the Fibrotic Response Associated with Proliferative Vitreoretinopathy.

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OM-101 Decreases the Fibrotic Response Associated with Proliferative Vitreoretinopathy.

@en

OM-101 Decreases the Fibrotic Response Associated with Proliferative Vitreoretinopathy.

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OM-101 Decreases the Fibrotic Response Associated with Proliferative Vitreoretinopathy.

@en

OM-101 Decreases the Fibrotic Response Associated with Proliferative Vitreoretinopathy.

@nl

P1476

OM-101 Decreases the Fibrotic Response Associated with Proliferative Vitreoretinopathy

@en

P2093

Keren Ben-Yaakov

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Tamir Weinberg

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Yoel Greenwald

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Zeev Dvashi

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P2860

The basics of epithelial-mesenchymal transition

Mechanism and in vitro pharmacology of TAK1 inhibition by (5Z)-7-Oxozeaenol

Non-Smad pathways in TGF-beta signaling

Cell size and invasion in TGF-beta-induced epithelial to mesenchymal transition is regulated by activation of the mTOR pathway

Tumor necrosis factor-alpha regulates transforming growth factor-beta-dependent epithelial-mesenchymal transition by promoting hyaluronan-CD44-moesin interaction.

Current management of proliferative vitreoretinopathy.

A mouse model of proliferative vitreoretinopathy induced by dispase.

TGF-β1 induced transdifferentiation of rpe cells is mediated by TAK1.

Ocular pathogenesis and immune reaction after intravitreal dispase injection in mice.

Non-Smad signaling pathways.

P304

1606854

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P31

scholarly article

P356

10.1155/2017/1606854

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P478

2017

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P50

Ayala Pollack

P577

2017-10-04T00:00:00Z

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P698

29109865

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P932

5646338

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