Attachment of human immunodeficiency virus-1 (HIV-1) particles bearing host-encoded B7-2 proteins leads to nuclear factor-kappa B- and nuclear factor of activated T cells-dependent activation of HIV-1 long terminal repeat transcription.
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Crystal Structure of NFAT Bound to the HIV-1 LTR Tandem κB Enhancer ElementHIV-1 induces DCIR expression in CD4+ T cells.Distinct mechanisms of CD4+ and CD8+ T-cell activation and bystander apoptosis induced by human immunodeficiency virus type 1 virions.Distinct host cell proteins incorporated by SIV replicating in CD4+ T cells from natural disease resistant versus non-natural disease susceptible hostsInhibition of HIV Env binding to cellular receptors by monoclonal antibody 2G12 as probed by Fc-tagged gp120.Reciprocal regulation of the nuclear factor of activated T cells and HIV-1.Does mitochondrial dysfunction during antiretroviral therapy in human immunodeficiency virus infection suggest antioxidant supplementation as a beneficial option?Characterization and role of lentivirus-associated host proteins.Molecular determinants of the ratio of inert to infectious virus particles.Polyclonal immunoglobulin G from patients neutralizes human immunodeficiency virus type 1 primary isolates by binding free virions, but without interfering with an initial CD4-independent attachment of the virus to primary blood mononuclear cells.BCL11B is a general transcriptional repressor of the HIV-1 long terminal repeat in T lymphocytes through recruitment of the NuRD complexInsertion of host-derived costimulatory molecules CD80 (B7.1) and CD86 (B7.2) into human immunodeficiency virus type 1 affects the virus life cycle.Presence of host ICAM-1 in laboratory and clinical strains of human immunodeficiency virus type 1 increases virus infectivity and CD4(+)-T-cell depletion in human lymphoid tissue, a major site of replication in vivo.Microarray study reveals that HIV-1 induces rapid type-I interferon-dependent p53 mRNA up-regulation in human primary CD4+ T cells.The nucleoside triphosphate diphosphohydrolase-1/CD39 is incorporated into human immunodeficiency type 1 particles, where it remains biologically active.Interaction between the cytoplasmic domain of ICAM-1 and Pr55Gag leads to acquisition of host ICAM-1 by human immunodeficiency virus type 1.Retrovirus infection strongly enhances scrapie infectivity release in cell culture.LFA-1 is a key determinant for preferential infection of memory CD4+ T cells by human immunodeficiency virus type 1.The C-type lectin surface receptor DCIR acts as a new attachment factor for HIV-1 in dendritic cells and contributes to trans- and cis-infection pathways.
P2860
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P2860
Attachment of human immunodeficiency virus-1 (HIV-1) particles bearing host-encoded B7-2 proteins leads to nuclear factor-kappa B- and nuclear factor of activated T cells-dependent activation of HIV-1 long terminal repeat transcription.
description
2000 nî lūn-bûn
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2000年の論文
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2000年学术文章
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2000年学术文章
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2000年学术文章
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2000年学术文章
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2000年学术文章
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name
Attachment of human immunodefi ...... terminal repeat transcription.
@en
Attachment of human immunodeficiency virus-1
@nl
type
label
Attachment of human immunodefi ...... terminal repeat transcription.
@en
Attachment of human immunodeficiency virus-1
@nl
prefLabel
Attachment of human immunodefi ...... terminal repeat transcription.
@en
Attachment of human immunodeficiency virus-1
@nl
P2093
P2860
P356
P1476
Attachment of human immunodefi ...... terminal repeat transcription.
@en
P2093
P2860
P304
P356
10.1074/JBC.M002198200
P407
P577
2000-11-28T00:00:00Z