Subcellular site of expression and route of vaccination influence pulmonary eosinophilia following respiratory syncytial virus challenge in BALB/c mice sensitized to the attachment G protein.
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Respiratory Syncytial Virus: Infection, Detection, and New Options for Prevention and Treatment.Induction of Th-1 and Th-2 responses by respiratory syncytial virus attachment glycoprotein is epitope and major histocompatibility complex independent.Differential role of gamma interferon in inhibiting pulmonary eosinophilia and exacerbating systemic disease in fusion protein-immunized mice undergoing challenge infection with respiratory syncytial virusVbeta14(+) T cells mediate the vaccine-enhanced disease induced by immunization with respiratory syncytial virus (RSV) G glycoprotein but not with formalin-inactivated RSVRespiratory syncytial virus G and/or SH protein alters Th1 cytokines, natural killer cells, and neutrophils responding to pulmonary infection in BALB/c mice.Respiratory syncytial virus infection and G and/or SH protein expression contribute to substance P, which mediates inflammation and enhanced pulmonary disease in BALB/c mice.Secreted respiratory syncytial virus G glycoprotein induces interleukin-5 (IL-5), IL-13, and eosinophilia by an IL-4-independent mechanism.Priming with a secreted form of the fusion protein of respiratory syncytial virus (RSV) promotes interleukin-4 (IL-4) and IL-5 production but not pulmonary eosinophilia following RSV challengeImmune responses and disease enhancement during respiratory syncytial virus infectionRespiratory syncytial virus (RSV) G glycoprotein is not necessary for vaccine-enhanced disease induced by immunization with formalin-inactivated RSV.Development and function of invariant natural killer T cells producing T(h)2- and T(h)17-cytokinesNonglycosylated G-Protein Vaccine Protects against Homologous and Heterologous Respiratory Syncytial Virus (RSV) Challenge, while Glycosylated G Enhances RSV Lung Pathology and Cytokine Levels.Single intranasal immunization with recombinant adenovirus-based vaccine induces protective immunity against respiratory syncytial virus infectionPulmonary eosinophilia requires interleukin-5, eotaxin-1, and CD4+ T cells in mice immunized with respiratory syncytial virus G glycoprotein.A respiratory syncytial virus (RSV) anti-G protein F(ab')2 monoclonal antibody suppresses mucous production and breathing effort in RSV rA2-line19F-infected BALB/c mice.The soluble form of human respiratory syncytial virus attachment protein differs from the membrane-bound form in its oligomeric state but is still capable of binding to cell surface proteoglycans.Respiratory syncytial virus G protein CX3C motif impairs human airway epithelial and immune cell responses.Nasal vaccination induces protective immunity without immunopathology.
P2860
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P2860
Subcellular site of expression and route of vaccination influence pulmonary eosinophilia following respiratory syncytial virus challenge in BALB/c mice sensitized to the attachment G protein.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh-hant
name
Subcellular site of expression ...... d to the attachment G protein.
@en
Subcellular site of expression ...... d to the attachment G protein.
@nl
type
label
Subcellular site of expression ...... d to the attachment G protein.
@en
Subcellular site of expression ...... d to the attachment G protein.
@nl
prefLabel
Subcellular site of expression ...... d to the attachment G protein.
@en
Subcellular site of expression ...... d to the attachment G protein.
@nl
P2093
P1476
Subcellular site of expression ...... d to the attachment G protein.
@en
P2093
Bembridge GP
Garcia-Beato R
P304
P407
P577
1998-09-01T00:00:00Z