The pathogenicity of a US3 protein kinase-deficient mutant of herpes simplex virus type 2 in mice.

The pathogenicity of a US3 protein kinase-deficient mutant of herpes simplex virus type 2 in mice.

Item
http://www.wikidata.org/entity/Q45777347
Inhibition of Epstein-Barr virus replication by a benzimidazole L-riboside: novel antiviral mechanism of 5, 6-dichloro-2-(isopropylamino)-1-beta-L-ribofuranosyl-1H-benzimidazole.Phosphorylation of cytokeratin 17 by herpes simplex virus type 2 US3 protein kinase.Herpes simplex virus protein kinases US3 and UL13 modulate VP11/12 phosphorylation, virion packaging, and phosphatidylinositol 3-kinase/Akt signaling activityNuclear accumulation of IE62, the varicella-zoster virus (VZV) major transcriptional regulatory protein, is inhibited by phosphorylation mediated by the VZV open reading frame 66 protein kinaseAmino acid differences in glycoproteins B (gB), C (gC), H (gH) and L (gL) are associated with enhanced herpes simplex virus type-1 (McKrae) entry via the paired immunoglobulin-like type-2 receptor α.A novel oncolytic herpes simplex virus that synergizes with phosphoinositide 3-kinase/Akt pathway inhibitors to target glioblastoma stem cells.Herpes simplex virus induces the marked up-regulation of the zinc finger transcriptional factor INSM1, which modulates the expression and localization of the immediate early protein ICP0.The varicella-zoster virus ORF66 protein induces kinase activity and is dispensable for viral replication.The ORF47 and ORF66 putative protein kinases of varicella-zoster virus determine tropism for human T cells and skin in the SCID-hu mouse.Subcellular localization of the alphaherpesvirus serine/threonine kinase Us3 as a determinant of Us3 function.Alphaherpesviruses and the cytoskeleton in neuronal infections.Herpes simplex virus type 1 primary envelopment: UL34 protein modification and the US3-UL34 catalytic relationshipGrowth and cytopathogenicity of herpes simplex virus in a macrophage cell line, RAW264: A good indicator of intraperitoneal pathogenicity.Role of pseudorabies virus Us3 protein kinase during neuronal infection.Herpes simplex virus 1 glycoprotein B and US3 collaborate to inhibit CD1d antigen presentation and NKT cell function.Expression of herpes simplex virus type 2 US3 affects the Cdc42/Rac pathway and attenuates c-Jun N-terminal kinase activation.Immunohistochemical studies on the transneuronal spread of virulent herpes simplex virus type 2 and its US3 protein kinase-deficient mutant after ocular inoculation.
P2860

The pathogenicity of a US3 protein kinase-deficient mutant of herpes simplex virus type 2 in mice.

Item
http://www.wikidata.org/entity/Q45777347
description
1993 nî lūn-bûn
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1993年の論文
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1993年学术文章
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1993年学术文章
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1993年学术文章
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1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
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1993年學術文章
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1993年學術文章
@zh-hant
name
The pathogenicity of a US3 pro ...... simplex virus type 2 in mice.
@en
The pathogenicity of a US3 pro ...... simplex virus type 2 in mice.
@nl
type
label
The pathogenicity of a US3 pro ...... simplex virus type 2 in mice.
@en
The pathogenicity of a US3 pro ...... simplex virus type 2 in mice.
@nl
prefLabel
The pathogenicity of a US3 pro ...... simplex virus type 2 in mice.
@en
The pathogenicity of a US3 pro ...... simplex virus type 2 in mice.
@nl
P1433
P1476
P2093
P2888
P304
P31
P356
P433
P478
P577
P6179
P698
P356
P1433
P1476
The pathogenicity of a US3 pro ...... simplex virus type 2 in mice.
@en
P2093
P2888
P304
P31
P356
P433
P478
P577
P6179
P698