A suboptimal src 3' splice site is necessary for efficient replication of Rous sarcoma virus.
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Avian retroviral RNA element promotes unspliced RNA accumulation in the cytoplasmSelection and characterization of replication-competent revertants of a Rous sarcoma virus src gene oversplicing mutant.Inhibition of RNA splicing at the Rous sarcoma virus src 3' splice site is mediated by an interaction between a negative cis element and a chicken embryo fibroblast nuclear factor.Efficient polyadenylation of Rous sarcoma virus RNA requires the negative regulator of splicing elementFunction of a bovine papillomavirus type 1 exonic splicing suppressor requires a suboptimal upstream 3' splice site.U1 small nuclear ribonucleoprotein and splicing inhibition by the rous sarcoma virus negative regulator of splicing element.The exon splicing silencer in human immunodeficiency virus type 1 Tat exon 3 is bipartite and acts early in spliceosome assembly.Optimization of a weak 3' splice site counteracts the function of a bovine papillomavirus type 1 exonic splicing suppressor in vitro and in vivoRepair of a Rev-minus human immunodeficiency virus type 1 mutant by activation of a cryptic splice site.Retroviral splicing suppressor requires three nonconsensus uridines in a 5' splice site-like sequence.Selective inhibition of splicing at the avian sarcoma virus src 3' splice site by direct-repeat posttranscriptional cis elementsAn exonic splicing silencer downstream of the 3' splice site A2 is required for efficient human immunodeficiency virus type 1 replication.The role of overlapping U1 and U11 5' splice site sequences in a negative regulator of splicingThe sequence complementarity between HIV-1 5' splice site SD4 and U1 snRNA determines the steady-state level of an unstable env pre-mRNA.Splicing efficiency of human immunodeficiency virus type 1 tat RNA is determined by both a suboptimal 3' splice site and a 10 nucleotide exon splicing silencer element located within tat exon 2.The retrovirus RNA trafficking granule: from birth to maturity.Structural and functional analysis of the Rous Sarcoma virus negative regulator of splicing and demonstration of its activation by the 9G8 SR proteinJuxtaposition of two distant, serine-arginine-rich protein-binding elements is required for optimal polyadenylation in Rous sarcoma virus.Branchpoint and polypyrimidine tract mutations mediating the loss and partial recovery of the Moloney murine sarcoma virus MuSVts110 thermosensitive splicing phenotype.SR protein splicing factors interact with the Rous sarcoma virus negative regulator of splicing elementSelection of the bovine papillomavirus type 1 nucleotide 3225 3' splice site is regulated through an exonic splicing enhancer and its juxtaposed exonic splicing suppressorThe Moloney murine sarcoma virus ts110 5' splice site signal contributes to the regulation of splicing efficiency and thermosensitivity.Comparative Transcriptome Analysis of Bombyx mori (Lepidoptera) Larval Midgut Response to BmNPV in Susceptible and Near-Isogenic Resistant Strains.Serine/arginine-rich proteins contribute to negative regulator of splicing element-stimulated polyadenylation in rous sarcoma virus.Presence of exon splicing silencers within human immunodeficiency virus type 1 tat exon 2 and tat-rev exon 3: evidence for inhibition mediated by cellular factors.RNA processing control in avian retrovirusesPosttranscriptional regulation of retroviral gene expression: primary RNA transcripts play three roles as pre-mRNA, mRNA, and genomic RNA.Evidence that a threshold of serine/arginine-rich (SR) proteins recruits CFIm to promote rous sarcoma virus mRNA 3' end formationR region sequences in the long terminal repeat of a murine retrovirus specifically increase expression of unspliced RNAs.An RNA splicing enhancer-like sequence is a component of a splicing inhibitor element from Rous sarcoma virus.Human T-cell leukemia virus type 2 Rex inhibits pre-mRNA splicing in vitro at an early stage of spliceosome formation.A naturally arising mutation of a potential silencer of exon splicing in human immunodeficiency virus type 1 induces dominant aberrant splicing and arrests virus production.Rous sarcoma virus direct repeat cis elements exert effects at several points in the virus life cycle.A premature termination codon mutation at the C terminus of foamy virus Gag downregulates the levels of spliced pol mRNA.Murine leukemia virus regulates alternative splicing through sequences upstream of the 5' splice site.
P2860
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P2860
A suboptimal src 3' splice site is necessary for efficient replication of Rous sarcoma virus.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年学术文章
@wuu
1995年学术文章
@zh-cn
1995年学术文章
@zh-hans
1995年学术文章
@zh-my
1995年学术文章
@zh-sg
1995年學術文章
@yue
1995年學術文章
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1995年學術文章
@zh-hant
name
A suboptimal src 3' splice sit ...... ication of Rous sarcoma virus.
@en
A suboptimal src 3' splice sit ...... ication of Rous sarcoma virus.
@nl
type
label
A suboptimal src 3' splice sit ...... ication of Rous sarcoma virus.
@en
A suboptimal src 3' splice sit ...... ication of Rous sarcoma virus.
@nl
prefLabel
A suboptimal src 3' splice sit ...... ication of Rous sarcoma virus.
@en
A suboptimal src 3' splice sit ...... ication of Rous sarcoma virus.
@nl
P356
P1433
P1476
A suboptimal src 3' splice sit ...... ication of Rous sarcoma virus.
@en
P2093
P304
P356
10.1006/VIRO.1995.1033
P407
P577
1995-02-01T00:00:00Z