The cellular 107K protein that binds to adenovirus E1A also associates with the large T antigens of SV40 and JC virus.
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pRb inactivation in mammary cells reveals common mechanisms for tumor initiation and progression in divergent epitheliaMutagenesis of the pRB pocket reveals that cell cycle arrest functions are separable from binding to viral oncoproteinsThe regions of the retinoblastoma protein needed for binding to adenovirus E1A or SV40 large T antigen are common sites for mutationsTwo distinct and frequently mutated regions of retinoblastoma protein are required for binding to SV40 T antigenHomologous sequences in adenovirus E1A and human papillomavirus E7 proteins mediate interaction with the same set of cellular proteinsAssociation of p107 with Sp1: genetically separable regions of p107 are involved in regulation of E2F- and Sp1-dependent transcriptionDifferential roles of two tandem E2F sites in repression of the human p107 promoter by retinoblastoma and p107 proteinsStructure of the retinoblastoma tumour-suppressor pocket domain bound to a peptide from HPV E7Evaluation of the cancer chemopreventive efficacy of rice bran in genetic mouse models of breast, prostate and intestinal carcinogenesisp300 family members associate with the carboxyl terminus of simian virus 40 large tumor antigenDetection of human neurotropic JC virus DNA sequence and expression of the viral oncogenic protein in pediatric medulloblastomas.Maternal consumption of canola oil suppressed mammary gland tumorigenesis in C3(1) TAg mice offspring.Binding of p110 retinoblastoma protein inhibits nuclear import of simian virus SV40 large tumor antigen.Complex formation of human papillomavirus E7 proteins with the retinoblastoma tumor suppressor gene product.Aberrant Proliferation of Differentiating Alveolar Cells Induces Hyperplasia in Resting Mammary Glands of SV40-TAg Transgenic MiceSimian virus 40 small tumor antigen and an amino-terminal domain of large tumor antigen share a common transforming functionp107 in the public eye: an Rb understudy and more.WAP-TAg transgenic mice and the study of dysregulated cell survival, proliferation, and mutation during breast carcinogenesis.Mullerian inhibiting substance suppresses tumor growth in the C3(1)T antigen transgenic mouse mammary carcinoma modelTargeted degradation of the retinoblastoma protein by human papillomavirus E7-E6 fusion proteins.A kinase activity associated with simian virus 40 large T antigen phosphorylates upstream binding factor (UBF) and promotes formation of a stable initiation complex between UBF and SL1Species-specific elements in the large T-antigen J domain are required for cellular transformation and DNA replication by simian virus 40Myc oncogenes: the enigmatic family.Oncogenic transformation by the human polyomaviruses.The Promise of Proteomics in the Study of Oncogenic Viruses.Human tumor-associated viruses and new insights into the molecular mechanisms of cancer.Development of PIN and prostate adenocarcinoma cell lines: a model system for multistage tumor progressionCarcinoma initiation via RB tumor suppressor inactivation: a versatile approach to epithelial subtype-dependent cancer initiation in diverse tissues.Genetic models in applied physiology: invited review: effect of oxygen deprivation on cell cycle activity: a profile of delay and arrest.PPARgamma induces cell cycle withdrawal: inhibition of E2F/DP DNA-binding activity via down-regulation of PP2A.Cross-talk between T-Ag presence and pRb family and p53/p73 signaling in mouse and human medulloblastoma.Oncogenicity of human papillomavirus- or adenovirus-transformed cells correlates with resistance to lysis by natural killer cells.The block of adipocyte differentiation by a C-terminally truncated, but not by full-length, simian virus 40 large tumor antigen is dependent on an intact retinoblastoma susceptibility protein family binding domain.Simian virus 40 large T antigen alters the phosphorylation state of the RB-related proteins p130 and p107.The human cytomegalovirus IE1-72 protein interacts with the cellular p107 protein and relieves p107-mediated transcriptional repression of an E2F-responsive promoter.Retinoblastoma-repression of E2F-dependent transcription depends on the ability of the retinoblastoma protein to interact with E2F and is abrogated by the adenovirus E1A oncoprotein.Oncogene-linked in situ immunotherapy of pre-B lymphoma arising in E mu/ret transgenic miceInactivating all three rb family pocket proteins is insufficient to initiate cervical cancer.JC virus DNA is present in the mucosa of the human colon and in colorectal cancers.Interaction between simian virus 40 large T antigen and insulin receptor substrate 1 is disrupted by the K1 mutation, resulting in the loss of large T antigen-mediated phosphorylation of Akt.
P2860
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P2860
The cellular 107K protein that binds to adenovirus E1A also associates with the large T antigens of SV40 and JC virus.
description
1989 nî lūn-bûn
@nan
1989年の論文
@ja
1989年学术文章
@wuu
1989年学术文章
@zh
1989年学术文章
@zh-cn
1989年学术文章
@zh-hans
1989年学术文章
@zh-my
1989年学术文章
@zh-sg
1989年學術文章
@yue
1989年學術文章
@zh-hant
name
The cellular 107K protein that ...... antigens of SV40 and JC virus.
@en
The cellular 107K protein that ...... antigens of SV40 and JC virus.
@nl
type
label
The cellular 107K protein that ...... antigens of SV40 and JC virus.
@en
The cellular 107K protein that ...... antigens of SV40 and JC virus.
@nl
prefLabel
The cellular 107K protein that ...... antigens of SV40 and JC virus.
@en
The cellular 107K protein that ...... antigens of SV40 and JC virus.
@nl
P2093
P1433
P1476
The cellular 107K protein that ...... antigens of SV40 and JC virus.
@en
P2093
P304
P356
10.1016/0092-8674(89)90839-8
P407
P577
1989-07-01T00:00:00Z