Laminin alpha1 chain reduces muscular dystrophy in laminin alpha2 chain deficient mice.
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Skeletal muscle laminin and MDC1A: pathogenesis and treatment strategies.Developmental and pathogenic mechanisms of basement membrane assemblyDistinct roles for laminin globular domains in laminin alpha1 chain mediated rescue of murine laminin alpha2 chain deficiencyBortezomib Does Not Reduce Muscular Dystrophy in the dy2J/dy2J Mouse Model of Laminin α2 Chain-Deficient Muscular DystrophyCib2 binds integrin alpha7Bbeta1D and is reduced in laminin alpha2 chain-deficient muscular dystrophyLaminin alpha1 globular domains 4-5 induce fetal development but are not vital for embryonic basement membrane assemblyNAD+ biosynthesis ameliorates a zebrafish model of muscular dystrophy.Laminin alpha1 chain mediated reduction of laminin alpha2 chain deficient muscular dystrophy involves integrin alpha7beta1 and dystroglycan.laminin alpha 1 gene is essential for normal lens development in zebrafish.Amelioration of laminin-alpha2-deficient congenital muscular dystrophy by somatic gene transfer of miniagrin.Increased neointimal thickening in dystrophin-deficient mdx mice.Porous protein-based scaffolds prepared through freezing as potential scaffolds for tissue engineering.Transgenic expression of Laminin α1 chain does not prevent muscle disease in the mdx mouse model for Duchenne muscular dystrophy.Muscle-specific expression of insulin-like growth factor 1 improves outcome in Lama2Dy-w mice, a model for congenital muscular dystrophy type 1APeripheral nerve pathology, including aberrant Schwann cell differentiation, is ameliorated by doxycycline in a laminin-α2-deficient mouse model of congenital muscular dystrophy.Quantitative proteomic analysis reveals metabolic alterations, calcium dysregulation, and increased expression of extracellular matrix proteins in laminin α2 chain-deficient muscle.Forced expression of laminin beta1 in podocytes prevents nephrotic syndrome in mice lacking laminin beta2, a model for Pierson syndromeLaminin-α1 LG4-5 domain binding to dystroglycan mediates muscle cell survival, growth, and the AP-1 and NF-κB transcription factors but also has adverse effects.Overexpression of the cytotoxic T cell (CT) carbohydrate inhibits muscular dystrophy in the dyW mouse model of congenital muscular dystrophy 1A.Laminin-111 protein therapy reduces muscle pathology and improves viability of a mouse model of merosin-deficient congenital muscular dystrophyLinker molecules between laminins and dystroglycan ameliorate laminin-alpha2-deficient muscular dystrophy at all disease stagesRole of extracellular matrix proteins and their receptors in the development of the vertebrate neuromuscular junctionLaminin-211 in skeletal muscle function.Laminin-111 restores regenerative capacity in a mouse model for alpha7 integrin congenital myopathyScaffold-forming and Adhesive Contributions of Synthetic Laminin-binding Proteins to Basement Membrane AssemblyThe synaptic CT carbohydrate modulates binding and expression of extracellular matrix proteins in skeletal muscle: Partial dependence on utrophin.Congenital muscular dystrophy. Part II: a review of pathogenesis and therapeutic perspectives.Vascular delivery of rAAVrh74.MCK.GALGT2 to the gastrocnemius muscle of the rhesus macaque stimulates the expression of dystrophin and laminin α2 surrogates.Cell-matrix interactions in muscle disease.The dystrophin-glycoprotein complex in the prevention of muscle damage.Absence of microRNA-21 does not reduce muscular dystrophy in mouse models of LAMA2-CMDLaminin: loss-of-function studies.Impaired fetal muscle development and JAK-STAT activation mark disease onset and progression in a mouse model for merosin-deficient congenital muscular dystrophy.Influence of exercise and aging on extracellular matrix composition in the skeletal muscle stem cell niche.Chimeric protein repair of laminin polymerization ameliorates muscular dystrophy phenotype.Angiotensin II type 1 receptor antagonists alleviate muscle pathology in the mouse model for laminin-α2-deficient congenital muscular dystrophy (MDC1A).Laminin alpha1 chain improves laminin alpha2 chain deficient peripheral neuropathy.Laminin {alpha}1 chain corrects male infertility caused by absence of laminin {alpha}2 chainLaminin-111 improves muscle repair in a mouse model of merosin-deficient congenital muscular dystrophy.Dystroglycan is selectively cleaved at the parenchymal basement membrane at sites of leukocyte extravasation in experimental autoimmune encephalomyelitis.
P2860
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P2860
Laminin alpha1 chain reduces muscular dystrophy in laminin alpha2 chain deficient mice.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh-hant
name
Laminin alpha1 chain reduces muscular dystrophy in laminin alpha2 chain deficient mice.
@en
Laminin alpha1 chain reduces muscular dystrophy in laminin alpha2 chain deficient mice.
@nl
type
label
Laminin alpha1 chain reduces muscular dystrophy in laminin alpha2 chain deficient mice.
@en
Laminin alpha1 chain reduces muscular dystrophy in laminin alpha2 chain deficient mice.
@nl
prefLabel
Laminin alpha1 chain reduces muscular dystrophy in laminin alpha2 chain deficient mice.
@en
Laminin alpha1 chain reduces muscular dystrophy in laminin alpha2 chain deficient mice.
@nl
P2093
P356
P1476
Laminin alpha1 chain reduces muscular dystrophy in laminin alpha2 chain deficient mice.
@en
P2093
Kinga Gawlik
Madeleine Durbeej
Peter Ekblom
Shin'ichi Takeda
Yuko Miyagoe-Suzuki
P304
P356
10.1093/HMG/DDH190
P577
2004-06-22T00:00:00Z