Exacerbations of Bronchitis: bronchial eosinophilia and gene expression for interleukin-4, interleukin-5, and eosinophil chemoattractants. - Wikidata - awgldk - TriplyDB

Exacerbations of Bronchitis: bronchial eosinophilia and gene expression for interleukin-4, interleukin-5, and eosinophil chemoattractants.

Exacerbations of Bronchitis: bronchial eosinophilia and gene expression for interleukin-4, interleukin-5, and eosinophil chemoattractants.

http://www.wikidata.org/entity/Q45878304

about

MMP-9, TIMP-1 and inflammatory cells in sputum from COPD patients during exacerbation Pathophysiology of viral-induced exacerbations of COPD Cytokine inhibition in the treatment of COPD.Significance of the microbiome in chronic obstructive pulmonary disease.Early phase resolution of mucosal eosinophilic inflammation in allergic rhinitis Toll-like receptor 3 is induced by and mediates antiviral activity against rhinovirus infection of human bronchial epithelial cells Overlap Between Asthma and COPD: Where the Two Diseases Converge.Remodeling in response to infection and injury. Airway inflammation and hypersecretion of mucus in smoking subjects with chronic obstructive pulmonary disease.Chronic obstructive pulmonary disease, pollution, pulmonary vascular disease, transplantation, pleural disease, and lung cancer in AJRCCM 2001.Novel insights into the aetiology and pathophysiology of increased airway inflammation during COPD exacerbations.Markers of exacerbation severity in chronic obstructive pulmonary disease Chronic obstructive pulmonary disease . 6: The aetiology of exacerbations of chronic obstructive pulmonary disease Airway inflammation in children with difficult asthma: relationships with airflow limitation and persistent symptoms.Methods for the assessment of endobronchial biopsies in clinical research: application to studies of pathogenesis and the effects of treatment.Inverse association of plasma IL-13 and inflammatory chemokines with lung function impairment in stable COPD: a cross-sectional cohort study.Inflammatory cells in the airways in COPD.Bronchial mucosal inflammation and upregulation of CXC chemoattractants and receptors in severe exacerbations of asthma.Profiling serum biomarkers in patients with COPD: associations with clinical parameters Role of infection and antimicrobial therapy in acute exacerbations of chronic obstructive pulmonary disease.COPD exacerbations . 2: aetiology Pulmonary biomarkers in chronic obstructive pulmonary disease.Determinants of bacteriological outcomes in exacerbations of chronic obstructive pulmonary disease.The role of airway secretions in COPD--clinical applications.Cellular and biochemical bases of chronic obstructive pulmonary disease.Is airway inflammation in chronic obstructive pulmonary disease (COPD) a risk factor for cardiovascular events?Aspects on pathophysiological mechanisms in COPD.The cytokine network in asthma and chronic obstructive pulmonary disease Pathogen-directed therapy in acute exacerbations of chronic obstructive pulmonary disease.Pulmonary epithelium, cigarette smoke, and chronic obstructive pulmonary disease.Identifying viral infections in vaccinated Chronic Obstructive Pulmonary Disease (COPD) patients using clinical features and inflammatory markers.New concepts in the pathobiology of chronic obstructive pulmonary disease Airways inflammation and treatment during acute exacerbations of COPD.Role of macrolide therapy in chronic obstructive pulmonary disease.The cytokine network in chronic obstructive pulmonary disease.Divergent functions for airway epithelial matrix metalloproteinase 7 and retinoic acid in experimental asthma.Antibiotics in acute exacerbations of chronic bronchitis.Can β2-adrenoceptor agonists, anticholinergic drugs, and theophylline contribute to the control of pulmonary inflammation and emphysema in COPD?Molecular and clinical rationale for therapeutic targeting of interleukin-5 and its receptor.β-Adrenoceptor modulation in chronic obstructive pulmonary disease: present and future perspectives.Pulmonary biomarkers in COPD exacerbations: a systematic review.

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Exacerbations of Bronchitis: bronchial eosinophilia and gene expression for interleukin-4, interleukin-5, and eosinophil chemoattractants.

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American Journal of Respiratory and Critical Care Medicine

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Exacerbations of Bronchitis: b ...... nd eosinophil chemoattractants

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Barnes N

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Fabbri LM

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Gamble E

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P2860

The response of human bone marrow to chronic cigarette smoking.

Relation of sputum inflammatory markers to symptoms and lung function changes in COPD exacerbations.

Cellular characteristics of sputum from patients with asthma and chronic bronchitis

Cytotoxic T cells clear virus but augment lung pathology in mice infected with respiratory syncytial virus

CC chemokine receptor (CCR)3/eotaxin is followed by CCR4/monocyte-derived chemokine in mediating pulmonary T helper lymphocyte type 2 recruitment after serial antigen challenge in vivo.

Bronchial inflammation in chronic bronchitis assessed by measurement of cell products in bronchial lavage fluid.

CD8 positive T cells influence antigen-specific immune responses through the expression of chemokines.

Alkaline phosphatase and peroxidase for double immunoenzymatic labelling of cellular constituents.

Sputum eosinophilia predicts benefit from prednisone in smokers with chronic obstructive bronchitis.

Respiratory syncytical virus-induced chemokine expression in the lower airways: eosinophil recruitment and degranulation.

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109-116

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1

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Graziella Turato

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