REDD1 deletion prevents dexamethasone-induced skeletal muscle atrophy.
about
Discovery of Compound A--a selective activator of the glucocorticoid receptor with anti-inflammatory and anti-cancer activityIs REDD1 a Metabolic Éminence Grise?mRNA-seq reveals skeletal muscle atrophy in response to handling stress in a marine teleost, the red cusk-eel (Genypterus chilensis)Dexamethasone and BCAA Failed to Modulate Muscle Mass and mTOR Signaling in GH-Deficient RatsTime course of the response to ACTH in pig: biological and transcriptomic study.Global deletion of BCATm increases expression of skeletal muscle genes associated with protein turnoverDisruption of REDD1 gene ameliorates sepsis-induced decrease in mTORC1 signaling but has divergent effects on proteolytic signaling in skeletal muscle.Acute Alcohol-Induced Decrease in Muscle Protein Synthesis in Female Mice Is REDD-1 and mTOR-Independent.Emerging role for regulated in development and DNA damage 1 (REDD1) in the regulation of skeletal muscle metabolism.Heat Stress Modulates Both Anabolic and Catabolic Signaling Pathways Preventing Dexamethasone-Induced Muscle Atrophy In VitroValidation of a commercially available anti-REDD1 antibody using RNA interference and REDD1-/- mouse embryonic fibroblastsEpigenomic and transcriptional control of insulin resistance.Loss of REDD1 augments the rate of the overload-induced increase in muscle mass.Implication of REDD1 in the activation of inflammatory pathways.Selective Activator of the Glucocorticoid Receptor Compound A Dissociates Therapeutic and Atrophogenic Effects of Glucocorticoid Receptor Signaling in Skin.Corticosterone alters materno-fetal glucose partitioning and insulin signalling in pregnant mice.Rev-erb-α regulates atrophy-related genes to control skeletal muscle mass.REDD1 induction regulates the skeletal muscle gene expression signature following acute aerobic exercise.Endurance training prevents negative effects of the hypoxia mimetic dimethyloxalylglycine on cardiac and skeletal muscle function.Fenofibrate prevents skeletal muscle loss in mice with lung cancer.The eIF2-alpha kinase HRI is a novel therapeutic target in multiple myeloma.Glucocorticoid-dependent REDD1 expression reduces muscle metabolism to enable adaptation under energetic stress.High-intensity eccentric training ameliorates muscle wasting in colon 26 tumor-bearing mice.Hypoxia impairs adaptation of skeletal muscle protein turnover- and AMPK signaling during fasting-induced muscle atrophyProtein Supplementation Prevents Dexamethasone-Induced Muscle Atrophy in C57BL/6 Mice
P2860
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P2860
REDD1 deletion prevents dexamethasone-induced skeletal muscle atrophy.
description
2014 nî lūn-bûn
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2014年の論文
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2014年学术文章
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2014年学术文章
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2014年学术文章
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2014年学术文章
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2014年学术文章
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name
REDD1 deletion prevents dexamethasone-induced skeletal muscle atrophy.
@en
REDD1 deletion prevents dexamethasone-induced skeletal muscle atrophy.
@nl
type
label
REDD1 deletion prevents dexamethasone-induced skeletal muscle atrophy.
@en
REDD1 deletion prevents dexamethasone-induced skeletal muscle atrophy.
@nl
prefLabel
REDD1 deletion prevents dexamethasone-induced skeletal muscle atrophy.
@en
REDD1 deletion prevents dexamethasone-induced skeletal muscle atrophy.
@nl
P2093
P2860
P1476
REDD1 deletion prevents dexamethasone-induced skeletal muscle atrophy
@en
P2093
Anne Bonnieu
Arnaud Ferry
Aurélie Docquier
Barbara Vernus
Bernadette Rossano
Chamroeun Sar
François B Favier
Gwenaelle Begue
Vincent Ollendorff
P2860
P304
P356
10.1152/AJPENDO.00234.2014
P577
2014-10-14T00:00:00Z