Increased serum IL-17 and IL-23 in the patient with ankylosing spondylitis. - Wikidata - awgldk - TriplyDB

Increased serum IL-17 and IL-23 in the patient with ankylosing spondylitis.

Increased serum IL-17 and IL-23 in the patient with ankylosing spondylitis.

http://www.wikidata.org/entity/Q45905113

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Old and new treatment targets in axial spondyloarthritis Paget's disease of bone: an osteoimmunological disorder?Impact of inflammation on the osteoblast in rheumatic diseases Review: The interleukin-23/interleukin-17 axis in spondyloarthritis pathogenesis: Th17 and beyond Roles of interleukin-17 in uveitis Increased frequencies of Th22 cells as well as Th17 cells in the peripheral blood of patients with ankylosing spondylitis and rheumatoid arthritis Phosphorylation of STAT-1 serine 727 is prolonged in HLA-B27-expressing human monocytic cells.Consumption of Lactobacillus casei fermented milk prevents Salmonella reactive arthritis by modulating IL-23/IL-17 expression Interleukin-37 is increased in ankylosing spondylitis patients and associated with disease activity.Role of IL-38 and its related cytokines in inflammation Bone Loss Triggered by the Cytokine Network in Inflammatory Autoimmune Diseases.IL-38 binds to the IL-36 receptor and has biological effects on immune cells similar to IL-36 receptor antagonist Biomarkers for diagnosis, monitoring of progression, and treatment responses in ankylosing spondylitis and axial spondyloarthritis.Effect of Tumor Necrosis Factor Inhibitor Therapy on Osteoclasts Precursors in Ankylosing Spondylitis Elevated serum levels of IL-6 and IL-17 may associate with the development of ankylosing spondylitis.A Single Nucleotide Polymorphism in the Il17ra Promoter Is Associated with Functional Severity of Ankylosing Spondylitis.The Role of IL-17 and Th17 Lymphocytes in Autoimmune Diseases Elevated serum interleukin-23 levels in ankylosing spondylitis patients and the relationship with disease activity.IL-23/Th17 axis is not influenced by TNF-blocking agents in ankylosing spondylitis patients Ankylosing spondylitis: from cells to genes Clinical improvement and reduction in serum calprotectin levels after an intensive exercise programme for patients with ankylosing spondylitis and non-radiographic axial spondyloarthritis.Evidence that autophagy, but not the unfolded protein response, regulates the expression of IL-23 in the gut of patients with ankylosing spondylitis and subclinical gut inflammation Ankylosing Spondylitis Patients Have Impaired Osteoclast Gene Expression in Circulating Osteoclast Precursors.TNF-α Induced the Enhanced Apoptosis of Mesenchymal Stem Cells in Ankylosing Spondylitis by Overexpressing TRAIL-R2.How to translate basic knowledge into clinical application of biologic therapy in spondyloarthritis.Therapeutic potential of interleukin-17 in inflammation and autoimmune diseases.Epigenetic and gene expression analysis of ankylosing spondylitis-associated loci implicate immune cells and the gut in the disease pathogenesis.Ustekinumab for the treatment of patients with active ankylosing spondylitis: results of a 28-week, prospective, open-label, proof-of-concept study (TOPAS).Role of IL-17 in the pathogenesis of psoriatic arthritis and axial spondyloarthritis.Mediators of inflammation and bone remodeling in rheumatic disease.The ongoing quest for biomarkers in Ankylosing Spondylitis.Pathogenesis of ankylosing spondylitis - recent advances and future directions.IL-23/IL-17 axis in spondyloarthritis-bench to bedside.Targeting the interleukin-23/17 axis in axial spondyloarthritis.Secukinumab: A Review in Ankylosing Spondylitis.Targeting IL-17 in autoimmunity and inflammation.Osteoimmunology: memorandum for rheumatologists.Differential Effects of Inflammation on Bone and Response to Biologics in Rheumatoid Arthritis and Spondyloarthritis.Ankylosing spondylitis M-CSF-derived macrophages are undergoing unfolded protein response (UPR) and express higher levels of interleukin-23.Cytokine profiles in axial spondyloarthritis.

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Increased serum IL-17 and IL-23 in the patient with ankylosing spondylitis.

http://www.wikidata.org/entity/Q45905113

description

2010 nî lūn-bûn

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2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年學術文章

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2010年學術文章

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name

Increased serum IL-17 and IL-23 in the patient with ankylosing spondylitis.

@en

Increased serum IL-17 and IL-23 in the patient with ankylosing spondylitis.

@nl

type

label

Increased serum IL-17 and IL-23 in the patient with ankylosing spondylitis.

@en

Increased serum IL-17 and IL-23 in the patient with ankylosing spondylitis.

@nl

prefLabel

Increased serum IL-17 and IL-23 in the patient with ankylosing spondylitis.

@en

Increased serum IL-17 and IL-23 in the patient with ankylosing spondylitis.

@nl

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P1433

Clinical Rheumatology

P1476

Increased serum IL-17 and IL-23 in the patient with ankylosing spondylitis.

@en

P2093

Dongqing Ye

http://www.w3.org/2001/XMLSchema#string

Faming Pan

http://www.w3.org/2001/XMLSchema#string

Fangfang Liao

http://www.w3.org/2001/XMLSchema#string

Guo Xia

http://www.w3.org/2001/XMLSchema#string

Jianhua Xu

http://www.w3.org/2001/XMLSchema#string

Jing Gao

http://www.w3.org/2001/XMLSchema#string

Li Zhang

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Rui Ge

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Sheng Wang

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Shengqian Xu

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P2860

Early rheumatoid arthritis is characterized by a distinct and transient synovial fluid cytokine profile of T cell and stromal cell origin.

Frequency and phenotype of peripheral blood Th17 cells in ankylosing spondylitis and rheumatoid arthritis

IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages

Divergent pro- and antiinflammatory roles for IL-23 and IL-12 in joint autoimmune inflammation

Evolving concepts of rheumatoid arthritis

More stories on Th17 cells.

Evaluation of diagnostic criteria for ankylosing spondylitis. A proposal for modification of the New York criteria.

Discovery and biology of IL-23 and IL-27: related but functionally distinct regulators of inflammation.

T(H)-17 cells in the circle of immunity and autoimmunity.

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10.1007/S10067-010-1647-4

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2

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30

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2010-12-17T00:00:00Z

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21161669

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