CORM-3-derived CO modulates polymorphonuclear leukocyte migration across the vascular endothelium by reducing levels of cell surface-bound elastase.
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Therapeutic potential of carbon monoxide in multiple sclerosisThe carbon monoxide releasing molecule CORM-2 attenuates Pseudomonas aeruginosa biofilm formationCarbon monoxide: present and future indications for a medical gasSubcellular Energetics and Metabolism: Potential Therapeutic Applications.Heme oxygenase in the regulation of vascular biology: from molecular mechanisms to therapeutic opportunities.Suppressive effect of CORM-2 on LPS-induced platelet activation by glycoprotein mediated HS1 phosphorylation interferenceTherapeutic potential of heme oxygenase-1/carbon monoxide in lung disease.Suppressive effect of exogenous carbon monoxide on endotoxin-stimulated platelet over-activation via the glycoprotein-mediated PI3K-Akt-GSK3β pathwayCarbon monoxide in exhaled breath testing and therapeutics.Targeting heme oxygenase-1 and carbon monoxide for therapeutic modulation of inflammation.Reduced caveolin-1 promotes hyperinflammation due to abnormal heme oxygenase-1 localization in lipopolysaccharide-challenged macrophages with dysfunctional cystic fibrosis transmembrane conductance regulator.Alterations of tumor microenvironment by carbon monoxide impedes lung cancer growth.Regulatory effect and mechanisms of carbon monoxide-releasing molecule II on hepatic energy metabolism in septic mice.Carbon monoxide: an unusual drug.Emerging concepts on the anti-inflammatory actions of carbon monoxide-releasing molecules (CO-RMs).CO and CO-releasing molecules (CO-RMs) in acute gastrointestinal inflammation.Therapeutic roles of carbon monoxide in intestinal ischemia-reperfusion injury.Pretreatment of human cerebrovascular endothelial cells with CO-releasing molecule-3 interferes with JNK/AP-1 signaling and suppresses LPS-induced proadhesive phenotype.Anti-inflammatory effects of tobramycin and a copper-tobramycin complex with superoxide dismutase-like activity.Heme Oxygenase-1 and Carbon Monoxide Promote Burkholderia pseudomallei Infection.Prevention of clinical and histological signs of proteolipid protein (PLP)-induced experimental allergic encephalomyelitis (EAE) in mice by the water-soluble carbon monoxide-releasing molecule (CORM)-A1.Carbon monoxide-releasing molecule, CORM-3, modulates alveolar macrophage M1/M2 phenotype in vitro.
P2860
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P2860
CORM-3-derived CO modulates polymorphonuclear leukocyte migration across the vascular endothelium by reducing levels of cell surface-bound elastase.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
CORM-3-derived CO modulates po ...... f cell surface-bound elastase.
@en
CORM-3-derived CO modulates po ...... f cell surface-bound elastase.
@nl
type
label
CORM-3-derived CO modulates po ...... f cell surface-bound elastase.
@en
CORM-3-derived CO modulates po ...... f cell surface-bound elastase.
@nl
prefLabel
CORM-3-derived CO modulates po ...... f cell surface-bound elastase.
@en
CORM-3-derived CO modulates po ...... f cell surface-bound elastase.
@nl
P2093
P2860
P1476
CORM-3-derived CO modulates po ...... f cell surface-bound elastase.
@en
P2093
Alfredo Capretta
Gediminas Cepinskas
Jancy Stephen
Nevena Markovic
Relka Bihari
Richard F Potter
Shigefumi Suehiro
Shinjiro Mizuguchi
P2860
P304
P356
10.1152/AJPHEART.00305.2009
P577
2009-06-26T00:00:00Z